Far upstream regions of class II MHC Ea are necessary for position-independent, copy-dependent expression of Ea transgene.
about
Locus control regionsSevere B cell deficiency and disrupted splenic architecture in transgenic mice expressing the E41K mutated form of Bruton's tyrosine kinase.Dividing the empire: boundary chromatin elements delimit the territory of enhancers.Regulation of major histocompatibility complex class II gene expression, genetic variation and disease.Position independent expression and developmental regulation is directed by the beta myosin heavy chain gene's 5' upstream region in transgenic mice.The 5' flanking region of the rat LAP (C/EBP beta) gene can direct high-level, position-independent, copy number-dependent expression in multiple tissues in transgenic mice.Transcription-coupled deposition of histone modifications during MHC class II gene activation.Correction of the X-linked immunodeficiency phenotype by transgenic expression of human Bruton tyrosine kinase under the control of the class II major histocompatibility complex Ea locus control regionRetinoblastoma protein expression facilitates chromatin remodeling at the HLA-DRA promoter.CFTR expression from a BAC carrying the complete human gene and associated regulatory elements.Sequencing of an upstream region of the human HLA-DRA gene containing X' and Y' boxes.Delivery of DNA into mammalian cells by receptor-mediated endocytosis and gene therapy.Analysis of a 70 kb segment of DNA containing the human zeta and alpha-globin genes linked to their regulatory element (HS-40) in transgenic mice.Dissection of the locus control function located on the chicken lysozyme gene domain in transgenic mice.Chromatin remodeling and extragenic transcription at the MHC class II locus control region.Contrasting effects of alpha and beta globin regulatory elements on chromatin structure may be related to their different chromosomal environments.Murine MHC class II locus control region drives expression of human beta-glucocerebrosidase in antigen presenting cells of transgenic mice.
P2860
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P2860
Far upstream regions of class II MHC Ea are necessary for position-independent, copy-dependent expression of Ea transgene.
description
1993 nî lūn-bûn
@nan
1993 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
Far upstream regions of class ...... nt expression of Ea transgene.
@ast
Far upstream regions of class ...... nt expression of Ea transgene.
@en
Far upstream regions of class ...... nt expression of Ea transgene.
@nl
type
label
Far upstream regions of class ...... nt expression of Ea transgene.
@ast
Far upstream regions of class ...... nt expression of Ea transgene.
@en
Far upstream regions of class ...... nt expression of Ea transgene.
@nl
prefLabel
Far upstream regions of class ...... nt expression of Ea transgene.
@ast
Far upstream regions of class ...... nt expression of Ea transgene.
@en
Far upstream regions of class ...... nt expression of Ea transgene.
@nl
P2860
P356
P1476
Far upstream regions of class ...... nt expression of Ea transgene.
@en
P2093
P2860
P304
P356
10.1093/NAR/21.9.2065
P407
P577
1993-05-01T00:00:00Z