Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence. - Wikidata - awgldk - TriplyDB

Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

http://www.wikidata.org/entity/Q35007125

about

Nuclear receptor binding protein 1 regulates intestinal progenitor cell homeostasis and tumour formation Multiple TSC22D4 iso-/phospho-glycoforms display idiosyncratic subcellular localizations and interacting protein partners A Phosphoproteomic Comparison of B-RAFV600E and MKK1/2 Inhibitors in Melanoma Cells.A prognostic signature of G(2) checkpoint function in melanoma cell lines.Hepatic transforming growth factor-β 1 stimulated clone-22 D1 controls systemic cholesterol metabolism.Narrowing the knowledge gaps for melanoma Phosphoproteome dynamics in onset and maintenance of oncogene-induced senescence.Near-genomewide RNAi screening for regulators of BRAF(V600E) -induced senescence identifies RASEF, a gene epigenetically silenced in melanoma.Hepatitis B virus (HBV) DNA integration in patients with occult HBV infection and hepatocellular carcinoma.TSC22D4 is a molecular output of hepatic wasting metabolism.Transforming growth factor-β-stimulated clone-22 is a negative-feedback regulator of Ras / Raf signaling: Implications for tumorigenesis.High NRBP1 expression in prostate cancer is linked with poor clinical outcomes and increased cancer cell growth.Human ribosomal protein L9 is a Bax suppressor that promotes cell survival in yeast.Specificity of phosphorylation responses to MAP kinase pathway inhibitors in melanoma cells.

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P2860

Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

http://www.wikidata.org/entity/Q35007125

description

2011 nî lūn-bûn

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2011 թուականի Մարտին հրատարակուած գիտական յօդուած

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2011 թվականի մարտին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

@ast

Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

@en

type

label

Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

@ast

Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

@en

prefLabel

Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

@ast

Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

@en

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The EMBO Journal

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Antagonistic TSC22D1 variants control BRAF(E600)-induced senescence.

@en

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Celia Vogel

http://www.w3.org/2001/XMLSchema#string

Cornelia Hömig-Hölzel

http://www.w3.org/2001/XMLSchema#string

Daniel S Peeper

http://www.w3.org/2001/XMLSchema#string

Els Verdegaal

http://www.w3.org/2001/XMLSchema#string

Marco G Cecchini

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Markus Germann

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Remco van Doorn

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P2860

Transforming growth factor-beta-stimulated clone-22 is a member of a family of leucine zipper proteins that can homo- and heterodimerize and has transcriptional repressor activity

A novel role for high-mobility group a proteins in cellular senescence and heterochromatin formation

Molecular cloning and characterization of a transcription factor for the C-type natriuretic peptide gene promoter

Autophagy mediates the mitotic senescence transition

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

Remodeling of chromatin structure in senescent cells and its potential impact on tumor suppression and aging

Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis

A synthetic lethal interaction between K-Ras oncogenes and Cdk4 unveils a therapeutic strategy for non-small cell lung carcinoma.

A system for stable expression of short interfering RNAs in mammalian cells

Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network.

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1753-1765

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scholarly article

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10.1038/EMBOJ.2011.95

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9

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30

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50907680

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21448135

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3102000

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