Multiple recombination pathways for sister chromatid exchange in Saccharomyces cerevisiae: role of RAD1 and the RAD52 epistasis group genes. - Wikidata - awgldk - TriplyDB

Multiple recombination pathways for sister chromatid exchange in Saccharomyces cerevisiae: role of RAD1 and the RAD52 epistasis group genes.

Multiple recombination pathways for sister chromatid exchange in Saccharomyces cerevisiae: role of RAD1 and the RAD52 epistasis group genes.

http://www.wikidata.org/entity/Q35017836

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Inverted repeat-stimulated sister-chromatid exchange events are RAD1-independent but reduced in a msh2 mutant.Competing roles of DNA end resection and non-homologous end joining functions in the repair of replication-born double-strand breaks by sister-chromatid recombination.RAD51 mutants cause replication defects and chromosomal instability Effects of mutations in SGS1 and in genes functionally related to SGS1 on inverted repeat-stimulated spontaneous unequal sister-chromatid exchange in yeast Elevated dNTP levels suppress hyper-recombination in Saccharomyces cerevisiae S-phase checkpoint mutants Is homologous recombination really an error-free process?Replication and recombination factors contributing to recombination-dependent bypass of DNA lesions by template switch Both RAD5-dependent and independent pathways are involved in DNA damage-associated sister chromatid exchange in budding yeast.Alkylation base damage is converted into repairable double-strand breaks and complex intermediates in G2 cells lacking AP endonuclease.Optimization of ordered plasmid assembly by gap repair in Saccharomyces cerevisiae.Aflatoxin B(1)-Associated DNA Adducts Stall S Phase and Stimulate Rad51 foci in Saccharomyces cerevisiae Role of mismatch repair in the fidelity of RAD51- and RAD59-dependent recombination in Saccharomyces cerevisiae.Defining a genotoxic profile with mouse embryonic stem cells Trex2 enables spontaneous sister chromatid exchanges without facilitating DNA double-strand break repair.Meiotic Recombination in Neurospora crassa Proceeds by Two Pathways with Extensive Holliday Junction Migration A mutant allele of the transcription factor IIH helicase gene, RAD3, promotes loss of heterozygosity in response to a DNA replication defect in Saccharomyces cerevisiae.Role of the Saccharomyces cerevisiae Rad51 paralogs in sister chromatid recombination.Activation of aflatoxin B1 by expression of human CYP1A2 polymorphisms in Saccharomyces cerevisiae.Stimulation of sister chromatid exchanges and mutation by aflatoxin B1-DNA adducts in Saccharomyces cerevisiae requires MEC1 (ATR), RAD53, and DUN1 UV but not X rays stimulate homologous recombination between sister chromatids and homologs in a Saccharomyces cerevisiae mec1 (ATR) hypomorphic mutant.The Shu complex promotes error-free tolerance of alkylation-induced base excision repair products.Rad10 exhibits lesion-dependent genetic requirements for recruitment to DNA double-strand breaks in Saccharomyces cerevisiae.Transcriptional response of yeast to aflatoxin B1: recombinational repair involving RAD51 and RAD1 RSC facilitates Rad59-dependent homologous recombination between sister chromatids by promoting cohesin loading at DNA double-strand breaks.Distinct cellular phenotype linked to defective DNA interstrand crosslink repair and homologous recombination CYP1A1 I462V polymorphism is associated with reduced genotoxicity in yeast despite positive association with increased cancer risk.Corrupting the DNA damage response: a critical role for Rad52 in tumor cell survival Saccharomyces cerevisiae Srs2 DNA helicase selectively blocks expansions of trinucleotide repeats.Both CAG repeats and inverted DNA repeats stimulate spontaneous unequal sister-chromatid exchange in Saccharomyces cerevisiae.Activation of the DNA damage checkpoint in yeast lacking the histone chaperone anti-silencing function 1.Different genetic requirements for repair of replication-born double-strand breaks by sister-chromatid recombination and break-induced replication Rad10-YFP focus induction in response to UV depends on RAD14 in yeast.

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Multiple recombination pathways for sister chromatid exchange in Saccharomyces cerevisiae: role of RAD1 and the RAD52 epistasis group genes.

http://www.wikidata.org/entity/Q35017836

description

2003 nî lūn-bûn

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2003 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2003 թվականի մայիսին հրատարակված գիտական հոդված

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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Multiple recombination pathway ...... e RAD52 epistasis group genes.

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Multiple recombination pathway ...... e RAD52 epistasis group genes.

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Multiple recombination pathway ...... e RAD52 epistasis group genes.

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Multiple recombination pathway ...... e RAD52 epistasis group genes.

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Nucleic Acids Research

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Multiple recombination pathway ...... e RAD52 epistasis group genes.

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Michael Fasullo

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Zheng Dong

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P2860

The structure-specific endonuclease Ercc1-Xpf is required for targeted gene replacement in embryonic stem cells

Sister chromatids are preferred over homologs as substrates for recombinational repair in Saccharomyces cerevisiae

Cell cycle and genetic requirements of two pathways of nonhomologous end-joining repair of double-strand breaks in Saccharomyces cerevisiae

Genetic requirements for the single-strand annealing pathway of double-strand break repair in Saccharomyces cerevisiae

A yeast homologue of the human phosphotyrosyl phosphatase activator PTPA is implicated in protection against oxidative DNA damage induced by the model carcinogen 4-nitroquinoline 1-oxide.

RAD1, an excision repair gene of Saccharomyces cerevisiae, is also involved in recombination

Preferential binding of yeast Rad4.Rad23 complex to damaged DNA.

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Removal of nonhomologous DNA ends in double-strand break recombination: the role of the yeast ultraviolet repair gene RAD1.

RAD10, an excision repair gene of Saccharomyces cerevisiae, is involved in the RAD1 pathway of mitotic recombination.

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2576-2585

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scholarly article

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10.1093/NAR/GKG352

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10

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31

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2003-05-01T00:00:00Z

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12736307

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156034

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