Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth. - Wikidata - awgldk - TriplyDB

Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

http://www.wikidata.org/entity/Q35018711

about

Onto better TRAILs for cancer treatment TWEAK/Fn14 signaling in tumors.Programmed Death Ligand 1 (PD-L1)-targeted TRAIL combines PD-L1-mediated checkpoint inhibition with TRAIL-mediated apoptosis induction.TWEAK/Fn14 Axis-Targeted Therapeutics: Moving Basic Science Discoveries to the Clinic.

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Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

http://www.wikidata.org/entity/Q35018711

description

2013 nî lūn-bûn

@nan

2013 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年学术文章

@wuu

2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

name

Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

@ast

Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

@en

type

label

Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

@ast

Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

@en

prefLabel

Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

@ast

Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

@en

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JOURNAL.PONE.0077050

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Fn14•TRAIL effectively inhibits hepatocellular carcinoma growth.

@en

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Alexandra Aronin

http://www.w3.org/2001/XMLSchema#string

Jacob Rachmilewitz

http://www.w3.org/2001/XMLSchema#string

Kobi Tzdaka

http://www.w3.org/2001/XMLSchema#string

Mark L Tykocinski

http://www.w3.org/2001/XMLSchema#string

Michal Dranitzki Elhalel

http://www.w3.org/2001/XMLSchema#string

Noam Shani

http://www.w3.org/2001/XMLSchema#string

Shira Amsili

http://www.w3.org/2001/XMLSchema#string

Tatyana B Prigozhina

http://www.w3.org/2001/XMLSchema#string

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TWEAK induces apoptosis through a death-signaling complex comprising receptor-interacting protein 1 (RIP1), Fas-associated death domain (FADD), and caspase-8

The Fn14 immediate-early response gene is induced during liver regeneration and highly expressed in both human and murine hepatocellular carcinomas

The human Fn14 receptor gene is up-regulated in migrating glioma cells in vitro and overexpressed in advanced glial tumors

TWEAK, a member of the TNF superfamily, is a multifunctional cytokine that binds the TweakR/Fn14 receptor

Fibroblast growth factor-inducible 14 mediates multiple pathways of TWEAK-induced cell death

TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis

TRAIL receptors 1 (DR4) and 2 (DR5) signal FADD-dependent apoptosis and activate NF-kappaB

Increased fibroblast growth factor-inducible 14 expression levels promote glioma cell invasion via Rac1 and nuclear factor-kappaB and correlate with poor patient outcome

The tumor necrosis factor-like weak inducer of apoptosis (TWEAK)-fibroblast growth factor-inducible 14 (Fn14) signaling system regulates glioma cell survival via NFkappaB pathway activation and BCL-XL/BCL-W expression

Superior antitumoral activity of dimerized targeted single-chain TRAIL fusion proteins under retention of tumor selectivity

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e77050

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scholarly article

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10.1371/JOURNAL.PONE.0077050

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10

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