A comparison of EDHF-mediated and anandamide-induced relaxations in the rat isolated mesenteric artery.
about
Cannabinoid-induced mesenteric vasodilation through an endothelial site distinct from CB1 or CB2 receptorsReceptors for acylethanolamides-GPR55 and GPR119The endothelial component of cannabinoid-induced relaxation in rabbit mesenteric artery depends on gap junctional communicationEndothelium-dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?Characterisation of the vasorelaxant properties of the novel endocannabinoid N-arachidonoyl-dopamine (NADA)Identification of two distinct vasodilator pathways activated by ATP in the mesenteric bed of the ratMechanisms of anandamide-induced vasorelaxation in rat isolated coronary arteriesAnandamide-induced relaxation of sheep coronary arteries: the role of the vascular endothelium, arachidonic acid metabolites and potassium channelsDefinitive role for natriuretic peptide receptor-C in mediating the vasorelaxant activity of C-type natriuretic peptide and endothelium-derived hyperpolarising factorEndothelium-dependent metabolism by endocannabinoid hydrolases and cyclooxygenases limits vasorelaxation to anandamide and 2-arachidonoylglycerolRegulator of G protein signaling 2 deficiency causes endothelial dysfunction and impaired endothelium-derived hyperpolarizing factor-mediated relaxation by dysregulating Gi/o signalingCB(1) receptor antagonist SR141716A inhibits Ca(2+)-induced relaxation in CB(1) receptor-deficient mice.P2Y receptor-induced EDHF vasodilatation is of primary importance for the regulation of perfusion pressure in the peripheral circulation of the rat.Role of endothelial Ni(2+)-sensitive Ca(2+) entry pathway in regulation of EDHF in porcine coronary artery.Direct effect of carbon monoxide on relaxation induced by electrical field stimulation in rat corpus cavernosum.Regulation of endocannabinoid release by G proteins: a paracrine mechanism of G protein-coupled receptor action.GPR55 deletion in mice leads to age-related ventricular dysfunction and impaired adrenoceptor-mediated inotropic responses.Endothelium-derived hyperpolarizing factor: is there a novel chemical mediator?Potassium ions and endothelium-derived hyperpolarizing factor in guinea-pig carotid and porcine coronary arteriesInvolvement of CYP3A-derived arachidonic acid metabolite(s) in responses to endothelium-derived K+ channel opening substance in monkey lingual arteryEvidence against potassium as an endothelium-derived hyperpolarizing factor in rat mesenteric small arteries.Effects of inhibitors of small- and intermediate-conductance calcium-activated potassium channels, inwardly-rectifying potassium channels and Na(+)/K(+) ATPase on EDHF relaxations in the rat hepatic artery.Vanilloid receptors on capsaicin-sensitive sensory nerves mediate relaxation to methanandamide in the rat isolated mesenteric arterial bed and small mesenteric arteries.Endothelium-dependent relaxation and endothelial hyperpolarization by P2Y receptor agonists in rat-isolated mesenteric artery.The complexities of the cardiovascular actions of cannabinoidsHeterogeneity in the mechanisms of vasorelaxation to anandamide in resistance and conduit rat mesenteric arteries.The effects of Delta9-tetrahydrocannabinol in rat mesenteric vasculature, and its interactions with the endocannabinoid anandamide.Vasorelaxant effects of oleamide in rat small mesenteric artery indicate action at a novel cannabinoid receptor.Cannabinoid pharmacology in the cardiovascular system: potential protective mechanisms through lipid signalling.Cannabinoids as therapeutic agents in cardiovascular disease: a tale of passions and illusionsVascular pharmacology of a novel cannabinoid-like compound, 3-(5-dimethylcarbamoyl-pent-1-enyl)-N-(2-hydroxy-1-methyl-ethyl)benzamide (VSN16) in the rat.Cardiovascular pharmacology of cannabinoids.Endocannabinoids and the heart.The endocannabinoid anandamide causes endothelium-dependent vasorelaxation in human mesenteric arteries.Calycosin and Formononetin Induce Endothelium-Dependent Vasodilation by the Activation of Large-Conductance Ca2+-Activated K+ Channels (BKCa).Altered reactivity of resistance vasculature contributes to hypertension in elastin insufficiency.Endothelial atypical cannabinoid receptor: do we have enough evidence?Integrin clustering enables anandamide-induced Ca2+ signaling in endothelial cells via GPR55 by protection against CB1-receptor-triggered repressionActivation of large-conductance, Ca2+-activated K+ channels by cannabinoids.Anandamide initiates Ca(2+) signaling via CB2 receptor linked to phospholipase C in calf pulmonary endothelial cells.
P2860
Q24642509-1C688CD2-001F-468B-A355-9311FE5170FBQ24647538-34DD6688-EFB5-48B7-9A33-A319D215892EQ24651338-246F7973-A212-441B-AC04-606A0CF4490EQ24672135-54C86FA2-977E-4237-AB58-2CEB9B273CE6Q28242911-9C6404FA-9916-4D1E-8D32-02790D3ECEC5Q28343908-C969D762-D806-44AF-A55D-9985E85ECA97Q28362103-A347F20A-F0BE-4218-950F-514453C8CA3EQ28362724-88FF45DA-CDAD-4C19-93D8-3904AD9E0404Q28582616-27943265-8FEA-43DC-B936-0E87604EFCAAQ28582727-8F21D3F7-34D3-4752-88CF-7E45829E6906Q30512752-274C523C-262A-439F-97D9-C067DB11C0F4Q30813670-500817A0-768B-4969-915C-8338C09E3DBDQ30831247-4929854A-A353-44A8-A7C8-5D0AF48197CBQ31833742-B3890784-365B-4D80-AA30-C8276131ADE2Q34075996-C178DA12-2EA0-407E-8FA0-8D26720BE3FCQ34214049-552C7AAC-F7F4-49B8-AB62-81F4AF1C139EQ34282113-D6DE4E8A-E035-4614-9B49-21C197627330Q34572615-F398FD51-DCBC-413C-BFEB-CE881D64CD53Q35028761-8ABC64AA-1DF6-40ED-8DDB-BDBA28B90E02Q35041604-EB7D8399-C5B4-4E11-B795-CF9AB3CC3C61Q35041874-57CF63CA-CBF9-4DBA-B9FB-B05BFC6CCADEQ35042110-EFE7C854-E070-4F5E-A25D-7C0EEFD2159BQ35042506-4EB3BECB-1F54-433D-8053-7C2CCE00B4ADQ35045366-7162CD6D-9F37-4D35-9297-56F7258B81E4Q35047093-E72D38C5-7DC0-4973-9554-71C98E8744EDQ35047182-F0C2E9E0-5EE9-40CB-96F3-17F6FFB69B1CQ35048945-AD08A846-6A05-4E19-A0B6-C54FAB24A570Q35100641-C987B67C-8327-4CBB-8D34-FA268D22CF74Q35684044-4FE37544-CC27-4512-9C13-AE3798F0FF30Q36052772-A731A2A4-4A53-4C1C-ABF2-61866718FB58Q36358729-9757AA14-8F7C-4720-B836-13E7567C362AQ36431860-EDA4890E-05C7-43B4-BA9B-820BE4AE8F97Q37412217-3AB9E7B0-D6CE-41C9-92FB-15F81B213E44Q37420729-70216D72-B5A1-418C-AB3D-6474A217EDF0Q37479253-02704219-BEE5-4A43-8634-B87FD97AD2C5Q37629251-9B80A8E0-112A-43AD-8892-3D3B5C4936C8Q38235155-A40A422F-CFE9-4F4B-8E42-2E6C6CEEDFDBQ39987047-E8987A46-1507-4A8B-8209-E4C0D969A5BDQ40384021-E49CD6E8-91AD-450A-9AF3-DB4317E50FD1Q40612911-29655E62-E9AE-4076-8F28-E4141F6D391C
P2860
A comparison of EDHF-mediated and anandamide-induced relaxations in the rat isolated mesenteric artery.
description
1997 nî lūn-bûn
@nan
1997 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
name
A comparison of EDHF-mediated ...... at isolated mesenteric artery.
@ast
A comparison of EDHF-mediated ...... at isolated mesenteric artery.
@en
type
label
A comparison of EDHF-mediated ...... at isolated mesenteric artery.
@ast
A comparison of EDHF-mediated ...... at isolated mesenteric artery.
@en
prefLabel
A comparison of EDHF-mediated ...... at isolated mesenteric artery.
@ast
A comparison of EDHF-mediated ...... at isolated mesenteric artery.
@en
P2860
P356
P1476
A comparison of EDHF-mediated ...... rat isolated mesenteric artery
@en
P2093
P2860
P304
P356
10.1038/SJ.BJP.0701546
P407
P577
1997-12-01T00:00:00Z