Lung function and inflammatory responses in healthy young adults exposed to 0.06 ppm ozone for 6.6 hours - Wikidata - awgldk - TriplyDB

Lung function and inflammatory responses in healthy young adults exposed to 0.06 ppm ozone for 6.6 hours

Lung function and inflammatory responses in healthy young adults exposed to 0.06 ppm ozone for 6.6 hours

http://www.wikidata.org/entity/Q35042499

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Air pollution and epigenetics: effects on SP-A and innate host defence in the lung Genes of innate immunity and the biological response to inhaled ozone Promotion of lung health: NHLBI Workshop on the Primary Prevention of Chronic Lung Diseases Physicochemical Characterization of Simulated Welding Fume from a Spark Discharge System Inflammatory Cytokines and White Blood Cell Counts Response to Environmental Levels of Diesel Exhaust and Ozone Inhalation Exposures Climate change and our environment: the effect on respiratory and allergic disease Rethinking Meta-Analysis: Applications for Air Pollution Data and Beyond.Experimental human exposure to air pollutants is essential to understand adverse health effects.Air toxics and epigenetic effects: ozone altered microRNAs in the sputum of human subjects.Effect of acute ozone induced airway inflammation on human sympathetic nerve traffic: a randomized, placebo controlled, crossover study Evaluating potential response-modifying factors for associations between ozone and health outcomes: a weight-of-evidence approach.Hourly differences in air pollution and risk of respiratory disease in the elderly: a time-stratified case-crossover study Effects of air pollutants on innate immunity: the role of Toll-like receptors and nucleotide-binding oligomerization domain-like receptors.Diesel exhaust modulates ozone-induced lung function decrements in healthy human volunteers.Interaction with epithelial cells modifies airway macrophage response to ozone Interaction effects of temperature and ozone on lung function and markers of systemic inflammation, coagulation, and fibrinolysis: a crossover study of healthy young volunteers.Inflammatory and repair pathways induced in human bronchoalveolar lavage cells with ozone inhalation Exposure to medium and high ambient levels of ozone causes adverse systemic inflammatory and cardiac autonomic effects Effect of Obesity on Acute Ozone-Induced Changes in Airway Function, Reactivity, and Inflammation in Adult Females Role of GSTM1 in resistance to lung inflammation.The glutathione-S-transferase mu 1 (GSTM1) null genotype and increased neutrophil response to low-level ozone (0.06 ppm).Hyaluronan activation of the Nlrp3 inflammasome contributes to the development of airway hyperresponsiveness.Asthmatic airway neutrophilia after allergen challenge is associated with the glutathione S-transferase M1 genotype Individuals with increased inflammatory response to ozone demonstrate muted signaling of immune cell trafficking pathways.Gonadal hormones and oxidative stress interaction differentially affects survival of male and female mice after lung Klebsiella pneumoniae infection.A Time-Stratified Case-Crossover Study of Ambient Ozone Exposure and Emergency Department Visits for Specific Respiratory Diagnoses in California (2005-2008).CD36 mediates endothelial dysfunction downstream of circulating factors induced by O3 exposure.Update in environmental and occupational medicine 2011.Evaluation of adverse human lung function effects in controlled ozone exposure studies.Evaluation of the causal framework used for setting national ambient air quality standards.Linking physiological parameters to perturbations in the human exposome: Environmental exposures modify blood pressure and lung function via inflammatory cytokine pathway.Effects of clinical and environmental factors on bronchoalveolar antibody responses to Pneumocystis jirovecii: A prospective cohort study of HIV+ patients.Ozone-derived Oxysterols Affect Liver X Receptor (LXR) Signaling: A POTENTIAL ROLE FOR LIPID-PROTEIN ADDUCTS.Prediction of lung function response for populations exposed to a wide range of ozone conditions.Weekly Personal Ozone Exposure and Respiratory Health in a Panel of Greek Schoolchildren.Is daily exposure to ozone associated with respiratory morbidity and lung function in a representative sample of schoolchildren? Results from a panel study in Greece.Increased antioxidant activity after exposure of ozone in murine asthma model.Regulation of ozone-induced lung inflammation by the epidermal growth factor receptor in mice.Ozone air pollution: how low can you go?Baseline airway inflammation may be a determinant of the response to ozone exposure in asthmatic patients.

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Lung function and inflammatory responses in healthy young adults exposed to 0.06 ppm ozone for 6.6 hours

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2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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Lung function and inflammatory ...... o 0.06 ppm ozone for 6.6 hours

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RCCM.201011-1813OC

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American Journal of Respiratory and Critical Care Medicine

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Lung function and inflammatory ...... o 0.06 ppm ozone for 6.6 hours

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Ana G Rappold

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Chong S Kim

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David B Peden

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David Diaz-Sanchez

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Howard Kehrl

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Martin Case

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Mike T Schmitt

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Milan J Hazucha

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Neil E Alexis

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P2860

Standardisation of spirometry

Nuclear factor-kappaB: a pivotal transcription factor in chronic inflammatory diseases

Effects of exposure to 0.06 ppm ozone on FEV1 in humans: a secondary analysis of existing data.

Short-term associations between ambient air pollutants and pediatric asthma emergency department visits.

The glutathione-S-transferase Mu 1 null genotype modulates ozone-induced airway inflammation in human subjects.

Low-level ozone exposure induces airways inflammation and modifies cell surface phenotypes in healthy humans.

Genetic polymorphism of GSTM1 and antioxidant supplementation influence lung function in relation to ozone exposure in asthmatic children in Mexico City

Effects of antioxidant enzyme polymorphisms on ozone-induced lung function changes.

Genetic susceptibility to the respiratory effects of air pollution.

Time-dependent changes of inflammatory mediators in the lungs of humans exposed to 0.4 ppm ozone for 2 hr: a comparison of mediators found in bronchoalveolar lavage fluid 1 and 18 hr after exposure.

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1215-1221

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10.1164/RCCM.201011-1813OC

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9

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183

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2011-01-07T00:00:00Z

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3114053

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