Low-level ozone exposure induces airways inflammation and modifies cell surface phenotypes in healthy humans.
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Climate change. A global threat to cardiopulmonary healthAir pollution exposure: a novel environmental risk factor for interstitial lung disease?Meteorological conditions, climate change, new emerging factors, and asthma and related allergic disorders. A statement of the World Allergy OrganizationInflammatory Cytokines and White Blood Cell Counts Response to Environmental Levels of Diesel Exhaust and Ozone Inhalation ExposuresAir toxics and epigenetic effects: ozone altered microRNAs in the sputum of human subjects.Air pollution toxicology--a brief review of the role of the science in shaping the current understanding of air pollution health risks.Lung function and inflammatory responses in healthy young adults exposed to 0.06 ppm ozone for 6.6 hoursAmbient PM2.5 exposure up-regulates the expression of costimulatory receptors on circulating monocytes in diabetic individualsGSTM1 modulation of IL-8 expression in human bronchial epithelial cells exposed to ozoneSRC-mediated EGF receptor activation regulates ozone-induced interleukin 8 expression in human bronchial epithelial cells.Interaction with epithelial cells modifies airway macrophage response to ozoneIdentification of oxidized phospholipids in bronchoalveolar lavage exposed to low ozone levels using multivariate analysis.Sex differences in the expression of lung inflammatory mediators in response to ozone.The glutathione-S-transferase mu 1 (GSTM1) null genotype and increased neutrophil response to low-level ozone (0.06 ppm).Individuals with increased inflammatory response to ozone demonstrate muted signaling of immune cell trafficking pathways.Baseline Chromatin Modification Levels May Predict Interindividual Variability in Ozone-Induced Gene Expression.Ambient ozone and pulmonary innate immunity.The role of oxidative stress and innate immunity in O(3) and endotoxin-induced human allergic airway disease.Challenge models to assess new therapies in chronic obstructive pulmonary disease.Ozone-derived Oxysterols Affect Liver X Receptor (LXR) Signaling: A POTENTIAL ROLE FOR LIPID-PROTEIN ADDUCTS.L-arginine promotes DNA repair in cultured bronchial epithelial cells exposed to ozone: involvement of the ATM pathway.Ozone primes alveolar macrophage-derived innate immunity in healthy human subjects.Ozone-induced Metabolic Effects in Humans. Ieiunium, Conviviorum, aut Timor? (Fasting, Feasting, or Fear?).
P2860
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P2860
Low-level ozone exposure induces airways inflammation and modifies cell surface phenotypes in healthy humans.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Low-level ozone exposure induc ...... phenotypes in healthy humans.
@ast
Low-level ozone exposure induc ...... phenotypes in healthy humans.
@en
type
label
Low-level ozone exposure induc ...... phenotypes in healthy humans.
@ast
Low-level ozone exposure induc ...... phenotypes in healthy humans.
@en
prefLabel
Low-level ozone exposure induc ...... phenotypes in healthy humans.
@ast
Low-level ozone exposure induc ...... phenotypes in healthy humans.
@en
P2093
P2860
P1476
Low-level ozone exposure induc ...... phenotypes in healthy humans.
@en
P2093
Bradford Harris
David B Peden
David Diaz-Sanchez
Howard Kehrl
John C Lay
Michelle L Hernandez
Milan Hazucha
Neil E Alexis
Philip A Bromberg
P2860
P304
P356
10.3109/08958371003596587
P577
2010-06-01T00:00:00Z