Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+ channel subunit contrasts with that observed in SeSAME/EAST syndrome.
about
Development and validation of fluorescence-based and automated patch clamp-based functional assays for the inward rectifier potassium channel Kir4.1.Hypomagnesemia: a clinical perspective.KCNJ10 determines the expression of the apical Na-Cl cotransporter (NCC) in the early distal convoluted tubule (DCT1).Regulation of potassium channel trafficking in the distal nephron.Molecular aspects of structure, gating, and physiology of pH-sensitive background K2P and Kir K+-transport channels.Genetic defects in the hotspot of inwardly rectifying K(+) (Kir) channels and their metabolic consequences: a reviewKCNJ10 (Kir4.1) is expressed in the basolateral membrane of the cortical thick ascending limb.Basolateral membrane K+ channels in renal epithelial cellsCaveolin-1 Deficiency Inhibits the Basolateral K+ Channels in the Distal Convoluted Tubule and Impairs Renal K+ and Mg2+ TransportRegulation of transport in the connecting tubule and cortical collecting ductNovel KCNJ10 Gene Variations Compromise Function of Inwardly Rectifying Potassium Channel 4.1.The expression, regulation, and function of Kir4.1 (Kcnj10) in the mammalian kidney.Src family protein tyrosine kinase regulates the basolateral K channel in the distal convoluted tubule (DCT) by phosphorylation of KCNJ10 protein.Disruption of KCNJ10 (Kir4.1) stimulates the expression of ENaC in the collecting ductNovel diuretic targetsMolecular bases of K(+) secretory cells in the inner ear: shared and distinct features between birds and mammalsDirect inhibition of basolateral Kir4.1/5.1 and Kir4.1 channels in the cortical collecting duct by dopamineExpression of Kir4.1 and Kir5.1 inwardly rectifying potassium channels in oligodendrocytes, the myelinating cells of the CNS.Potassium channels: structures, diseases, and modulators.Inward-rectifying potassium channelopathies: new insights into disorders of sodium and potassium homeostasis.Loss of transcriptional activation of the potassium channel Kir5.1 by HNF1β drives autosomal dominant tubulointerstitial kidney disease.Role and mechanisms of regulation of the basolateral Kir 4.1/Kir 5.1K+ channels in the distal tubules.Basolateral Kir4.1 activity in the distal convoluted tubule regulates K secretion by determining NaCl cotransporter activityEssential role of Kir5.1 channels in renal salt handling and blood pressure control.HNF1B controls epithelial organization and cell polarity during ureteric bud branching and collecting duct morphogenesis.Potassium Sensing by Renal Distal Tubules Requires Kir4.1.Congenital heart defects in patients with deletions upstream of SOX9.Extracellular K+ rapidly controls NaCl cotransporter phosphorylation in the native distal convoluted tubule by Cl- -dependent and independent mechanisms.
P2860
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P2860
Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+ channel subunit contrasts with that observed in SeSAME/EAST syndrome.
description
2011 nî lūn-bûn
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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
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2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
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2011年论文
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name
Renal phenotype in mice lackin ...... erved in SeSAME/EAST syndrome.
@ast
Renal phenotype in mice lackin ...... erved in SeSAME/EAST syndrome.
@en
type
label
Renal phenotype in mice lackin ...... erved in SeSAME/EAST syndrome.
@ast
Renal phenotype in mice lackin ...... erved in SeSAME/EAST syndrome.
@en
prefLabel
Renal phenotype in mice lackin ...... erved in SeSAME/EAST syndrome.
@ast
Renal phenotype in mice lackin ...... erved in SeSAME/EAST syndrome.
@en
P2093
P2860
P50
P356
P1476
Renal phenotype in mice lackin ...... served in SeSAME/EAST syndrome
@en
P2093
Fabien Sohet
May Bloch-Faure
Pascal Houillier
Suresh Krishna Ramakrishnan
Thibaut Jacques
P2860
P304
10361-10366
P356
10.1073/PNAS.1101400108
P407
P50
P577
2011-06-01T00:00:00Z