High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failure. - Wikidata - awgldk - TriplyDB

High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failure.

High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failure.

http://www.wikidata.org/entity/Q35075534

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An Overview of Murine High Fat Diet as a Model for Type 2 Diabetes Mellitus A New Method to Stabilize C-Kit Expression in Reparative Cardiac Mesenchymal Cells.Left ventricular mechanical dysfunction in diet-induced obese mice is exacerbated during inotropic stress: a cine DENSE cardiovascular magnetic resonance study Saturated high-fat diet-induced obesity increases adenylate cyclase of myocardial β-adrenergic system and does not compromise cardiac function.High-Fat and Fat-Enriched Diets Impair the Benefits of Moderate Physical Training in the Aorta and the Heart in Rats.Metabolic and Biochemical Stressors in Diabetic Cardiomyopathy.Acute consumption of a high-fat diet prior to ischemia-reperfusion results in cardioprotection through NF-κB-dependent regulation of autophagic pathways.MicroRNA-539 is up-regulated in failing heart, and suppresses O-GlcNAcase expression.Obesity superimposed on aging magnifies inflammation and delays the resolving response after myocardial infarction Oxidative Stress-Dependent Coronary Endothelial Dysfunction in Obese Mice Cardiac Dysfunction Induced by Obesity Is Not Related to β-Adrenergic System Impairment at the Receptor-Signalling Pathway.Successful metabolic adaptations leading to the prevention of high fat diet-induced murine cardiac remodeling Excess Linoleic Acid Increases Collagen I/III Ratio and "Stiffens" the Heart Muscle Following High Fat Diets.Induction of activating transcription factor 3 limits survival following infarct-induced heart failure in mice.Long-term consumption of an obesogenic high fat diet prior to ischemia-reperfusion mediates cardioprotection via Epac1-dependent signaling.Estrogen Receptor α Participates to the Beneficial Effect of Red Wine Polyphenols in a Mouse Model of Obesity-Related Disorders Good and bad consequences of altered fatty acid metabolism in heart failure: evidence from mouse models.Short-term high-fat diet compromises myocardial function: a radial strain rate imaging study.Partial deletion of ROCK2 protects mice from high-fat diet-induced cardiac insulin resistance and contractile dysfunction.Loss of the melanocortin-4 receptor in mice causes dilated cardiomyopathy.Obesity and Cardiometabolic Defects in Heart Failure Pathology.Murine models provide insight to the development of non-alcoholic fatty liver disease.Circulating miR-19b and miR-181b are potential biomarkers for diabetic cardiomyopathy.Murine diet-induced obesity remodels cardiac and liver mitochondrial phospholipid acyl chains with differential effects on respiratory enzyme activity.Frequency of mononuclear diploid cardiomyocytes underlies natural variation in heart regeneration.Cardiomyocyte-specific deletion of GSK-3β leads to cardiac dysfunction in a diet induced obesity model.The effects of fatty acid composition on cardiac hypertrophy and function in mouse models of diet-induced obesity.Diabetic db/db mice do not develop heart failure upon pressure overload: a longitudinal in vivo PET, MRI, and MRS study on cardiac metabolic, structural, and functional adaptations.ATF3 expression in cardiomyocytes preserves homeostasis in the heart and controls peripheral glucose tolerance.Type-2 diabetic aldehyde dehydrogenase 2 mutant mice (ALDH 2*2) exhibiting heart failure with preserved ejection fraction phenotype can be determined by exercise stress echocardiography.Cardiac-specific overexpression of aldehyde dehydrogenase 2 exacerbates cardiac remodeling in response to pressure overload.

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P2860

High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failure.

http://www.wikidata.org/entity/Q35075534

description

2013 nî lūn-bûn

@nan

2013 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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High fat feeding in mice is in ...... not exacerbate heart failure.

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High fat feeding in mice is in ...... not exacerbate heart failure.

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High fat feeding in mice is in ...... not exacerbate heart failure.

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High fat feeding in mice is in ...... not exacerbate heart failure.

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Adjoa Agyemang Boakye

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Angelica M Demartino

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Aruni Bhatnagar

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Deqing Zhang

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Joseph David Hoetker

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Kenneth R Brittian

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Lewis J Watson

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Robert E Brainard

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Shahid Pervez Baba

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P2860

Adverse effects of high glucose and free fatty acid on cardiomyocytes are mediated by connective tissue growth factor

Non-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transition

Acute exposure to a high cholesterol diet attenuates myocardial ischemia-reperfusion injury in cholesteryl ester transfer protein mice.

O-linked β-N-acetylglucosamine transferase is indispensable in the failing heart.

Improved mitochondrial function with diet-induced increase in either docosahexaenoic acid or arachidonic acid in membrane phospholipids.

Cardioprotective actions of acute HMG-CoA reductase inhibition in the setting of myocardial infarction.

Myocardial insulin resistance induced by high fat feeding in heart failure is associated with preserved contractile function.

Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR-gamma coactivator 1alpha.

Modulating fatty acid oxidation in heart failure.

Endothelial nitric oxide synthase overexpression attenuates congestive heart failure in mice.

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