Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR-gamma coactivator 1alpha. - Wikidata - awgldk - TriplyDB

Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR-gamma coactivator 1alpha.

Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR-gamma coactivator 1alpha.

http://www.wikidata.org/entity/Q34772551

about

Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injury Thioredoxin and ventricular remodeling Mitochondrial biogenesis and dynamics in the developing and diseased heart Maternal cardiac metabolism in pregnancy Using exercise to measure and modify cardiac function New insights into PGC-1 coactivators: redefining their role in the regulation of mitochondrial function and beyond PGC-1 proteins and heart failure Functional O-GlcNAc modifications: implications in molecular regulation and pathophysiology Pathogenesis of target organ damage in hypertension: role of mitochondrial oxidative stress PGC-1/Spargel Counteracts High-Fat-Diet-Induced Obesity and Cardiac Lipotoxicity Downstream of TOR and Brummer ATGL Lipase.Cardiac-specific hexokinase 2 overexpression attenuates hypertrophy by increasing pentose phosphate pathway flux PGC-1 alpha serine 570 phosphorylation and GCN5-mediated acetylation by angiotensin II drive catalase down-regulation and vascular hypertrophy Cardiac lipin 1 expression is regulated by the peroxisome proliferator activated receptor γ coactivator 1α/estrogen related receptor axis.Cancer therapy-induced cardiotoxicity: basic mechanisms and potential cardioprotective therapies.Alpha1A-adrenergic receptor-directed autoimmunity induces left ventricular damage and diastolic dysfunction in rats.Low levels of physical activity increase metabolic responsiveness to cold in a rat (Rattus fuscipes).A role for peroxisome proliferator-activated receptor γ coactivator-1 in the control of mitochondrial dynamics during postnatal cardiac growth Disruption of sarcolemmal ATP-sensitive potassium channel activity impairs the cardiac response to systolic overload.Defective DNA replication impairs mitochondrial biogenesis in human failing hearts Maintaining PGC-1α expression following pressure overload-induced cardiac hypertrophy preserves angiogenesis but not contractile or mitochondrial function.Effects of aging on angiogenesis.Tom70 serves as a molecular switch to determine pathological cardiac hypertrophy Induction of PGC-1α expression can be detected in blood samples of patients with ST-segment elevation acute myocardial infarction Telomere dysfunction induces metabolic and mitochondrial compromise.O-linked β-N-acetylglucosamine transferase is indispensable in the failing heart.Pathological role of serum- and glucocorticoid-regulated kinase 1 in adverse ventricular remodeling.Identification of the molecular basis of doxorubicin-induced cardiotoxicity.Promoting PGC-1α-driven mitochondrial biogenesis is detrimental in pressure-overloaded mouse hearts.PGC1α drives NAD biosynthesis linking oxidative metabolism to renal protection.Divergent mitochondrial biogenesis responses in human cardiomyopathy The PGC-1 cascade as a therapeutic target for heart failure QiShenYiQi Pills, a Compound Chinese Medicine, Ameliorates Doxorubicin-Induced Myocardial Structure Damage and Cardiac Dysfunction in Rats Analysis of skeletal muscle gene expression patterns and the impact of functional capacity in patients with systolic heart failure.Transcriptional coactivators PGC-1alpha and PGC-lbeta control overlapping programs required for perinatal maturation of the heart Glucose metabolism and cardiac hypertrophy microRNA-22 promotes heart failure through coordinate suppression of PPAR/ERR-nuclear hormone receptor transcription.Mitochondrial adaptations to physiological vs. pathological cardiac hypertrophy.Enhanced cardiac Akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes.High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failure.Toll-like receptor-mediated inflammatory signaling reprograms cardiac energy metabolism by repressing peroxisome proliferator-activated receptor γ coactivator-1 signaling.

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P2860

Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR-gamma coactivator 1alpha.

http://www.wikidata.org/entity/Q34772551

description

2006 nî lūn-bûn

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2006 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2006 թվականի հունիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Transverse aortic constriction ...... PPAR-gamma coactivator 1alpha.

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Transverse aortic constriction ...... PPAR-gamma coactivator 1alpha.

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Transverse aortic constriction ...... PPAR-gamma coactivator 1alpha.

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Transverse aortic constriction ...... PPAR-gamma coactivator 1alpha.

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Transverse aortic constriction ...... PPAR-gamma coactivator 1alpha.

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Transverse aortic constriction ...... PPAR-gamma coactivator 1alpha.

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Transverse aortic constriction ...... PPAR-gamma coactivator 1alpha.

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Proceedings of the National Academy of Sciences of the United States of America

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Transverse aortic constriction ...... PPAR-gamma coactivator 1alpha.

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Anthony Rosenzweig

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Mikhail Novikov

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Sherry Chin

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Yanhong Ma

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Zoltan Arany

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P2860

A cold-inducible coactivator of nuclear receptors linked to adaptive thermogenesis

Control of hepatic gluconeogenesis through the transcriptional coactivator PGC-1

Peroxisome proliferator-activated receptor coactivator-1alpha (PGC-1alpha) coactivates the cardiac-enriched nuclear receptors estrogen-related receptor-alpha and -gamma. Identification of novel leucine-rich interaction motif within PGC-1alpha

Transcriptional co-activator PGC-1 alpha drives the formation of slow-twitch muscle fibres

CREB regulates hepatic gluconeogenesis through the coactivator PGC-1

Metabolic control through the PGC-1 family of transcription coactivators

Regional absence of mitochondria causing energy depletion in the myocardium of muscle LIM protein knockout mice.

Transcriptional activation of energy metabolic switches in the developing and hypertrophied heart.

Energy metabolism in the hypertrophied heart.

Regulation of peroxisome proliferator-activated receptor gamma coactivator 1 alpha (PGC-1 alpha ) and mitochondrial function by MEF2 and HDAC5

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10.1073/PNAS.0603615103

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26

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103

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Bruce Spiegelman

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2006-06-14T00:00:00Z

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7008772

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