Transcriptional activation by EBV nuclear antigen 1 is essential for the expression of EBV's transforming genes. - Wikidata - awgldk - TriplyDB

Transcriptional activation by EBV nuclear antigen 1 is essential for the expression of EBV's transforming genes.

Transcriptional activation by EBV nuclear antigen 1 is essential for the expression of EBV's transforming genes.

http://www.wikidata.org/entity/Q35080523

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The viral etiology of AIDS-associated malignancies The Role of Gammaherpesviruses in Cancer Pathogenesis Similarities between the Epstein-Barr Virus (EBV) Nuclear Protein EBNA1 and the Pioneer Transcription Factor FoxA: Is EBNA1 a "Bookmarking" Oncoprotein that Alters the Host Cell Epigenotype?Potential cellular functions of Epstein-Barr Nuclear Antigen 1 (EBNA1) of Epstein-Barr Virus Epstein-Barr virus and host cell methylation: regulation of latency, replication and virus reactivation Keeping it quiet: chromatin control of gammaherpesvirus latency Contributions of Epstein-Barr nuclear antigen 1 (EBNA1) to cell immortalization and survival Discovery of selective inhibitors against EBNA1 via high throughput in silico virtual screening The Epstein-Barr virus-encoded microRNA MiR-BART9 promotes tumor metastasis by targeting E-cadherin in nasopharyngeal carcinoma Human lymphoblastoid B-cell lines reprogrammed to EBV-free induced pluripotent stem cells Options for change in the Australian energy profile Interaction between basic residues of Epstein-Barr virus EBNA1 protein and cellular chromatin mediates viral plasmid maintenance.Reliable generation of induced pluripotent stem cells from human lymphoblastoid cell lines.Restricted expression of Epstein-Barr virus latent genes in murine B cells derived from embryonic stem cells Zinc coordination is required for and regulates transcription activation by Epstein-Barr nuclear antigen 1 CTCF prevents the epigenetic drift of EBV latency promoter Qp Development of a high-throughput screen for inhibitors of Epstein-Barr virus EBNA1.Epigenetic regulation of EBV persistence and oncogenesis.Chromatin organization of gammaherpesvirus latent genomes.Hsp90 inhibitors block outgrowth of EBV-infected malignant cells in vitro and in vivo through an EBNA1-dependent mechanism.Regulation of Epstein-Barr virus OriP replication by poly(ADP-ribose) polymerase 1.Minicircle-oriP-IFNγ: a novel targeted gene therapeutic system for EBV positive human nasopharyngeal carcinoma.EBV latency types adopt alternative chromatin conformations.BZLF1 governs CpG-methylated chromatin of Epstein-Barr Virus reversing epigenetic repression.The effect of Epstein-Barr virus Latent Membrane Protein 2 expression on the kinetics of early B cell infection The importance of epigenetic alterations in the development of epstein-barr virus-related lymphomas.Lymphomas differ in their dependence on Epstein-Barr virus The Epstein-Barr virus BamHI C promoter is not essential for B cell immortalization in vitro, but it greatly enhances B cell growth transformation.The ZIIR element of the Epstein-Barr virus BZLF1 promoter plays a central role in establishment and maintenance of viral latency Reprogramming of EBV-immortalized B-lymphocyte cell lines into induced pluripotent stem cells.Live-cell imaging reveals multiple interactions between Epstein-Barr virus nuclear antigen 1 and cellular chromatin during interphase and mitosis Epstein-Barr virus exploits BSAP/Pax5 to achieve the B-cell specificity of its growth-transforming program Epstein-Barr virus latent membrane protein 2A is a B-cell receptor mimic and essential for B-cell survival Serological profiling of the EBV immune response in Chronic Fatigue Syndrome using a peptide microarray.Interaction between HMGA1a and the origin recognition complex creates site-specific replication origins Epstein-Barr virus interactions with the Bcl-2 protein family and apoptosis in human tumor cells.The affinity of EBNA1 for its origin of DNA synthesis is a determinant of the origin's replicative efficiency.The plasmid replicon of Epstein-Barr virus: mechanistic insights into efficient, licensed, extrachromosomal replication in human cells.Chromatin organization and virus gene expression.Epstein-Barr Nuclear Antigen 1 modulates replication of oriP-plasmids by impeding replication and transcription fork migration through the family of repeats.

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Transcriptional activation by EBV nuclear antigen 1 is essential for the expression of EBV's transforming genes.

http://www.wikidata.org/entity/Q35080523

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2006 nî lūn-bûn

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Transcriptional activation by ...... n of EBV's transforming genes.

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Bill Sugden

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Dagmar Pich

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Jindong Wang

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Markus Altmann

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Romana Ruiss

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Wolfgang Hammerschmidt

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P2860

EBNA-1, a bifunctional transcriptional activator

The EBV nuclear antigen 1 (EBNA1) enhances B cell immortalization several thousandfold

Epstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.

EBNA-1: a protein pivotal to latent infection by Epstein-Barr virus.

Latent Marek's disease virus can be activated from its chromosomally integrated state in herpesvirus-transformed lymphoma cells.

Maintenance of Epstein-Barr virus (EBV) oriP-based episomes requires EBV-encoded nuclear antigen-1 chromosome-binding domains, which can be replaced by high-mobility group-I or histone H1.

Mutually exclusive use of viral promoters in Epstein-Barr virus latently infected lymphocytes

Glycoprotein gp110 of Epstein-Barr virus determines viral tropism and efficiency of infection

Construction of large DNA segments in Escherichia coli.

Epstein-Barr virus sustains Burkitt's lymphomas and Hodgkin's disease.

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38

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103

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16966603

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