Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition. - Wikidata - awgldk - TriplyDB

Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition.

Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition.

http://www.wikidata.org/entity/Q35085117

about

Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosis Oxidative stress and glutathione in TGF-beta-mediated fibrogenesis Snail activation disrupts tissue homeostasis and induces fibrosis in the adult kidney Calcium/NFAT signalling promotes early nephrogenesis Predictive usefulness of urinary biomarkers for the identification of cyclosporine A-induced nephrotoxicity in a rat model Evaluation of molecular profiles in calcineurin inhibitor toxicity post-kidney transplant: input to chronic allograft dysfunction.Direct transcriptional reprogramming of adult cells to embryonic nephron progenitors.Cyclosporine a mediates pathogenesis of aggressive cutaneous squamous cell carcinoma by augmenting epithelial-mesenchymal transition: role of TGFβ signaling pathway.Late kidney allograft loss: what we know about it, and what we can do about it Tacrolimus increases Nox4 expression in human renal fibroblasts and induces fibrosis-related genes by aberrant TGF-beta receptor signalling Nox2 is a mediator of chronic CsA nephrotoxicity.Relevance of the stroma and epithelial-mesenchymal transition (EMT) for the rheumatic diseases.The NRF2-heme oxygenase-1 system modulates cyclosporin A-induced epithelial-mesenchymal transition and renal fibrosis.Epithelial-to-mesenchymal transition in early transplant tubulointerstitial damage Immunosuppressive therapy and malignancy in organ transplant recipients: a systematic review.Immunohistochemical Localization of Epithelial Mesenchymal Transition Markers in Cyclosporine A Induced Gingival Overgrowth Stem cell options for kidney disease.Deciphering calcineurin inhibitor nephrotoxicity: a pharmacological approach.Epithelial-mesenchymal transition (EMT) in kidney fibrosis: fact or fantasy?Pharmacogenetics and drug-induced nephrotoxicity in renal transplant recipients.The safety of calcineurin inhibitors for kidney-transplant patients.Tacrolimus Modulates TGF-β Signaling to Induce Epithelial-Mesenchymal Transition in Human Renal Proximal Tubule Epithelial Cells c-Jun-N-Terminal Kinase Signaling Is Involved in Cyclosporine-Induced Epithelial Phenotypic Changes MicroRNAs and drug-induced kidney injury.BMP6 reverses TGF-beta1-induced changes in HK-2 cells: implications for the treatment of renal fibrosis.Osteopontin mediating cyclosporine A induced epithelial-to-mesenchymal transition on rat renal tubular epithelial cells.TDAG51 mediates epithelial-to-mesenchymal transition in human proximal tubular epithelium.Cyclosporine A--induced oxidative stress in human renal mesangial cells: a role for ERK 1/2 MAPK signaling.TGF-β1 mediates sirolimus and cyclosporine A-induced alteration of barrier function in renal epithelial cells via a noncanonical ERK1/2 signaling pathway.A proteomic approach to immune-mediated epithelial-mesenchymal transition.Proteinase-activated receptor 4 stimulation-induced epithelial-mesenchymal transition in alveolar epithelial cells.Prostaglandin D2 inhibits TGF-beta1-induced epithelial-to-mesenchymal transition in MDCK cells.Overexpression of E2A proteins induces epithelial-mesenchymal transition in human renal proximal tubular epithelial cells suggesting a potential role in renal fibrosis.Sirolimus enhances cyclosporine a-induced cytotoxicity in human renal glomerular mesangial cells Protein kinase B/Akt activity is involved in renal TGF-beta1-driven epithelial-mesenchymal transition in vitro and in vivo.Fascin-1 is released from proximal tubular cells in response to calcineurin inhibitors (CNIs) and correlates with isometric vacuolization in kidney transplanted patients.Brain dead donor kidneys are immunologically active: is intervention justified?Defining a microRNA-mRNA interaction map for calcineurin inhibitor induced nephrotoxicity.Induction of PPAR gamma mRNA and protein expression by rosiglitazone in chronic cyclosporine nephropathy in the rat.Aldosterone induces collagen synthesis via activation of extracellular signal-regulated kinase 1 and 2 in renal proximal tubules.

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Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition.

http://www.wikidata.org/entity/Q35085117

description

2005 nî lūn-bûn

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2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2005 թվականի օգոստոսին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition.

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Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition.

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Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition.

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Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition.

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Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition.

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Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition.

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The American Journal of Pathology

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Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition

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Craig Slattery

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Michael P Ryan

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P2860

Interactions between heterologous helix-loop-helix proteins generate complexes that bind specifically to a common DNA sequence

Helix-loop-helix proteins: regulators of transcription in eucaryotic organisms

B-cell-specific DNA binding by an E47 homodimer

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE)

The helix-loop-helix gene E2A is required for B cell formation

The transcription factor snail controls epithelial-mesenchymal transitions by repressing E-cadherin expression

Identification of calcineurin as a key signalling enzyme in T-lymphocyte activation

Smooth muscle alpha-actin gene requires two E-boxes for proper expression in vivo and is a target of class I basic helix-loop-helix proteins.

Factors produced by activated leukocytes alter renal epithelial cell differentiation.

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Eric L Campbell

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