Pre- and neonatal exposure to lipopolysaccharide or the enteric metabolite, propionic acid, alters development and behavior in adolescent rats in a sexually dimorphic manner.
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Identification and Treatment of Pathophysiological Comorbidities of Autism Spectrum Disorder to Achieve Optimal Outcomes.Neuroinflammation in Autism: Plausible Role of Maternal Inflammation, Dietary Omega 3, and MicrobiotaEnteric bacterial metabolites propionic and butyric acid modulate gene expression, including CREB-dependent catecholaminergic neurotransmission, in PC12 cells--possible relevance to autism spectrum disordersModulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines.Enteric short-chain fatty acids: microbial messengers of metabolism, mitochondria, and mind: implications in autism spectrum disorders.Approaches to studying and manipulating the enteric microbiome to improve autism symptomsGastrointestinal dysfunction in autism spectrum disorder: the role of the mitochondria and the enteric microbiome.Inflammation early in life is a vulnerability factor for emotional behavior at adolescence and for lipopolysaccharide-induced spatial memory and neurogenesis alteration at adulthood.Modeling environmental risk factors of autism in mice induces IBD-related gut microbial dysbiosis and hyperserotonemia.Activation of mGluR2/3 underlies the effects of N-acetylcystein on amygdala-associated autism-like phenotypes in a valproate-induced rat model of autism.Relative abundance of short chain and polyunsaturated fatty acids in propionic acid-induced autistic features in rat pups as potential markers in autismLow-dose aspartame consumption differentially affects gut microbiota-host metabolic interactions in the diet-induced obese rat.The Central Nervous System and the Gut Microbiome.Sex differences in the gut microbiome-brain axis across the lifespan.The microbiome in early life: self-completion and microbiota protection as health priorities.Gut Dysbiosis in Animals Due to Environmental Chemical Exposures.Modulation of Immunological Pathways in Autistic and Neurotypical Lymphoblastoid Cell Lines by the Enteric Microbiome Metabolite Propionic Acid.Harnessing Gut Microbes for Mental Health: Getting From Here to There.Butyrate enhances mitochondrial function during oxidative stress in cell lines from boys with autism.Pathophysiological and neurobehavioral characteristics of a propionic acid-mediated autism-like rat model.The Role of the Microbiome in the Developmental Origins of Health and Disease.GUT in FOCUS Symposium NOBEL FORUM, Karolinska Institutet, February 2nd 2015Intravenous immunoglobulin for the treatment of autoimmune encephalopathy in children with autism
P2860
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P2860
Pre- and neonatal exposure to lipopolysaccharide or the enteric metabolite, propionic acid, alters development and behavior in adolescent rats in a sexually dimorphic manner.
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2014 nî lūn-bûn
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2014 թուականի Յունուարին հրատարակուած գիտական յօդուած
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2014 թվականի հունվարին հրատարակված գիտական հոդված
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2014年の論文
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name
Pre- and neonatal exposure to ...... n a sexually dimorphic manner.
@ast
Pre- and neonatal exposure to ...... n a sexually dimorphic manner.
@en
type
label
Pre- and neonatal exposure to ...... n a sexually dimorphic manner.
@ast
Pre- and neonatal exposure to ...... n a sexually dimorphic manner.
@en
prefLabel
Pre- and neonatal exposure to ...... n a sexually dimorphic manner.
@ast
Pre- and neonatal exposure to ...... n a sexually dimorphic manner.
@en
P2093
P2860
P1433
P1476
Pre- and neonatal exposure to ...... n a sexually dimorphic manner.
@en
P2093
Derrick F Macfabe
Kelly A Foley
Klaus-Peter Ossenkopp
Martin Kavaliers
P2860
P304
P356
10.1371/JOURNAL.PONE.0087072
P407
P577
2014-01-22T00:00:00Z