Expression of Smad7 in mouse eyes accelerates healing of corneal tissue after exposure to alkali - Wikidata - awgldk - TriplyDB

Expression of Smad7 in mouse eyes accelerates healing of corneal tissue after exposure to alkali

Expression of Smad7 in mouse eyes accelerates healing of corneal tissue after exposure to alkali

http://www.wikidata.org/entity/Q35087770

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Therapeutic effect of topical administration of SN50, an inhibitor of nuclear factor-kappaB, in treatment of corneal alkali burns in mice Activation of JNK signaling mediates connective tissue growth factor expression and scar formation in corneal wound healing The wound healing effects of vitamin A eye drops after a corneal alkali burn in rats Gene therapy in the cornea: 2005--present.Inhibition of autoregulated TGFbeta signaling simultaneously enhances proliferation and differentiation of kidney epithelium and promotes repair following renal ischemia.Induction of FGF-2 synthesis by IL-1beta in aqueous humor through P13-kinase and p38 in rabbit corneal endothelium.Pretranscriptional regulation of Tgf-beta1 by PI polyamide prevents scarring and accelerates wound healing of the cornea after exposure to alkali.Tetrandrine suppresses activation of human subconjunctival fibroblasts in vitro.Corneal transduction by intra-stromal injection of AAV vectors in vivo in the mouse and ex vivo in human explants.Therapeutic potential of trichostatin A to control inflammatory and fibrogenic disorders of the ocular surface.TRPV1 involvement in inflammatory tissue fibrosis in mice Loss of tumor necrosis factor alpha potentiates transforming growth factor beta-mediated pathogenic tissue response during wound healing.The role of Smad7 in oral mucositis.Temporal smad7 transgene induction in mouse epidermis accelerates skin wound healing Unraveling the biological functions of Smad7 with mouse models.Inhibition of development of laser-induced choroidal neovascularization with suppression of infiltration of macrophages in Smad3-null mice.Suppression of In Vivo Neovascularization by the Loss of TRPV1 in Mouse Cornea.Corrective GUSB transfer to the canine mucopolysaccharidosis VII cornea using a helper-dependent canine adenovirus vector.Diospyros kaki Extract Inhibits Alkali Burn-Induced Corneal Neovascularization.Sma- and Mad-related protein 7 (Smad7) is required for embryonic eye development in the mouse.Molecular insights on the effect of TGF-β1/-β3 in human corneal fibroblasts Future therapeutic treatment of COPD: struggle between oxidants and cytokines WOUND-HEALING PROPERTIES OF TRANSFORMING GROWTH FACTOR β (TGF-β) INDUCIBLE EARLY GENE 1 (TIEG1) KNOCKOUT MICE.Inhibition of NUCKS Facilitates Corneal Recovery Following Alkali Burn Gene therapy for diseases of the cornea - a review.Corneal morphogenesis during development and wound healing.Regulation of TGF-β Family Signaling by Inhibitory Smads.Sonic hedgehog: its expression in a healing cornea and its role in neovascularization.HSV-1 infection suppresses TGF-beta1 and SMAD3 expression in human corneal epithelial cells.Inhibition of p38MAP kinase suppresses fibrotic reaction of retinal pigment epithelial cells.An ophthalmic solution of a peroxisome proliferator-activated receptor gamma agonist prevents corneal inflammation in a rat alkali burn model Impaired Healing of a Cutaneous Wound in an Inducible Nitric Oxide Synthase-Knockout Mouse.Distribution of TGF-beta isoforms and signaling intermediates in corneal fibrotic wound repair.Inhibitory effect of blocking TGF-beta/Smad signal on injury-induced fibrosis of corneal endothelium.Loss of tenascin X gene function impairs injury-induced stromal angiogenesis in mouse corneas.Adenoviral gene transfer of BMP-7, Id2, or Id3 suppresses injury-induced epithelial-to-mesenchymal transition of lens epithelium in mice.CHO proteome alterations induced by reverse transformation.TRPA1 is required for TGF-β signaling and its loss blocks inflammatory fibrosis in mouse corneal stroma.Smad7 enhances ATM activity by facilitating the interaction between ATM and Mre11-Rad50-Nbs1 complex in DNA double-strand break repair.Impaired cornea wound healing in a tenascin C-deficient mouse model.

P2860

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P2860

Expression of Smad7 in mouse eyes accelerates healing of corneal tissue after exposure to alkali

http://www.wikidata.org/entity/Q35087770

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2005 nî lūn-bûn

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2005年の論文

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Expression of Smad7 in mouse e ...... issue after exposure to alkali

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Expression of Smad7 in mouse e ...... issue after exposure to alkali

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Expression of Smad7 in mouse e ...... issue after exposure to alkali

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Expression of Smad7 in mouse e ...... issue after exposure to alkali

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Expression of Smad7 in mouse e ...... issue after exposure to alkali

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Expression of Smad7 in mouse e ...... issue after exposure to alkali

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The American Journal of Pathology

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Akira Ooshima

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Kathleen C Flanders

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Kazuo Ikeda

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Misako Sato

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Osamu Yamanaka

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Shizuya Saika

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Winston W-Y Kao

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Yasuteru Muragaki

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Yoshitaka Ohnishi

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P2860

Migration of human monocytes in response to vascular endothelial growth factor (VEGF) is mediated via the VEGF receptor flt-1

A mechanism of suppression of TGF-beta/SMAD signaling by NF-kappa B/RelA

Therapeutic potential of inhibition of the NF-kappaB pathway in the treatment of inflammation and cancer

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

Myofibroblasts and mechano-regulation of connective tissue remodelling

The MAD-related protein Smad7 associates with the TGFbeta receptor and functions as an antagonist of TGFbeta signaling

Identification of Smad7, a TGFbeta-inducible antagonist of TGF-beta signalling

Wound healing in MIP-1alpha(-/-) and MCP-1(-/-) mice

Therapeutic effect of topical administration of SN50, an inhibitor of nuclear factor-kappaB, in treatment of corneal alkali burns in mice

TGFbeta2 in corneal morphogenesis during mouse embryonic development

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