Lithocholic acid feeding induces segmental bile duct obstruction and destructive cholangitis in mice. - Wikidata - awgldk - TriplyDB

Lithocholic acid feeding induces segmental bile duct obstruction and destructive cholangitis in mice.

Lithocholic acid feeding induces segmental bile duct obstruction and destructive cholangitis in mice.

http://www.wikidata.org/entity/Q35087943

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Bile salts of vertebrates: structural variation and possible evolutionary significance The genetics of complex cholestatic disorders Pathogenesis of primary sclerosing cholangitis and advances in diagnosis and management A new, major C27 biliary bile acid in the red-winged tinamou (Rhynchotus rufescens):25R-1beta, 3alpha,7alpha-trihydroxy-5beta-cholestan-27-oic acid A new xenobiotic-induced mouse model of sclerosing cholangitis and biliary fibrosis Immunoprecipitation and MALDI-MS identification of lithocholic acid-tagged proteins in liver of bile duct-ligated rats.Investigation of triamterene as an inhibitor of the TGR5 receptor: identification in cells and animals.Intestinal detoxification limits the activation of hepatic pregnane X receptor by lithocholic acid.Mouse organic solute transporter alpha deficiency enhances renal excretion of bile acids and attenuates cholestasis Lithocholic acid feeding results in direct hepato-toxicity independent of neutrophil function in mice Urinary metabolomics in Fxr-null mice reveals activated adaptive metabolic pathways upon bile acid challenge Bile acid changes after high-dose ursodeoxycholic acid treatment in primary sclerosing cholangitis: Relation to disease progression.Vitamin D modulates biliary fibrosis in ABCB4-deficient mice.Role of cholangiocytes in primary biliary cirrhosis.Participation of nuclear factor (erythroid 2-related), factor 2 in ameliorating lithocholic acid-induced cholestatic liver injury in mice.Fibrosis in autoimmune and cholestatic liver disease An updated review on drug-induced cholestasis: mechanisms and investigation of physicochemical properties and pharmacokinetic parameters.The anti-fibrotic effects of epigallocatechin-3-gallate in bile duct-ligated cholestatic rats and human hepatic stellate LX-2 cells are mediated by the PI3K/Akt/Smad pathway.Characterization of animal models for primary sclerosing cholangitis (PSC).Molecular mechanisms of ursodeoxycholic acid toxicity & side effects: ursodeoxycholic acid freezes regeneration & induces hibernation mode.Novel insight into mechanisms of cholestatic liver injury.A comparative study of the sulfation of bile acids and a bile alcohol by the Zebra danio (Danio rerio) and human cytosolic sulfotransferases (SULTs).Critical Factors in the Assessment of Cholestatic Liver Injury In Vitro.High-dose ursodeoxycholic acid increases risk of adverse outcomes in patients with early stage primary sclerosing cholangitis.Constitutive androstane receptor-mediated changes in bile acid composition contributes to hepatoprotection from lithocholic acid-induced liver injury in mice Roles of the hemostatic system and neutrophils in liver injury from co-exposure to amiodarone and lipopolysaccharide.Bile Acid sulfation: a pathway of bile acid elimination and detoxification.Inflammatory pseudo-tumor of the liver: a rare pathological entity Ursodeoxycholic acid in cholestasis: linking action mechanisms to therapeutic applications.Pathogenesis of primary sclerosing cholangitis.Mechanisms of tissue injury in autoimmune liver diseases.Animal models in primary biliary cirrhosis and primary sclerosing cholangitis.Modulation of the gut microbiota impacts nonalcoholic fatty liver disease: a potential role for bile acids.Development of betulinic acid as an agonist of TGR5 receptor using a new in vitro assay Efficacy and safety of tauroursodeoxycholic acid in the treatment of liver cirrhosis: a double-blind randomized controlled trial.Kinetic analysis of bile acid sulfation by stably expressed human sulfotransferase 2A1 (SULT2A1).Switch from Mnt-Max to Myc-Max induces p53 and cyclin D1 expression and apoptosis during cholestasis in mouse and human hepatocytes.Gut barrier failure biomarkers are associated with poor disease outcome in patients with primary sclerosing cholangitis.The receptor TGR5 protects the liver from bile acid overload during liver regeneration in mice.Role of pregnane X-receptor in regulating bacterial translocation in chronic liver diseases.

P2860

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P2860

Lithocholic acid feeding induces segmental bile duct obstruction and destructive cholangitis in mice.

http://www.wikidata.org/entity/Q35087943

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2006 nî lūn-bûn

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2006 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2006 թվականի փետրվարին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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Lithocholic acid feeding induc ...... structive cholangitis in mice.

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Lithocholic acid feeding induc ...... structive cholangitis in mice.

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Lithocholic acid feeding induc ...... estructive cholangitis in mice

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Andrea Fuchsbichler

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Hanns-Ulrich Marschall

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Hartmut Jaeschke

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Helmut Denk

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Robert Krause

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P2860

The nuclear receptor PXR is a lithocholic acid sensor that protects against liver toxicity

The continuing importance of bile acids in liver and intestinal disease

Trafficking of the bile salt export pump from the Golgi to the canalicular membrane is regulated by the p38 MAP kinase

Production of bile duct hyperplasia and gallstones by lithocholic acid.

Bile salt transporters: molecular characterization, function, and regulation.

An essential role for nuclear receptors SXR/PXR in detoxification of cholestatic bile acids.

Apoptosis versus oncotic necrosis in hepatic ischemia/reperfusion injury.

Cytokeratins as targets for bile acid-induced toxicity.

The cholangiopathies: disorders of biliary epithelia.

Detoxification of lithocholic acid, a toxic bile acid: relevance to drug hepatotoxicity.

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