Severely impaired insulin signaling in chronic wounds of diabetic ob/ob mice: a potential role of tumor necrosis factor-alpha - Wikidata - awgldk - TriplyDB

Severely impaired insulin signaling in chronic wounds of diabetic ob/ob mice: a potential role of tumor necrosis factor-alpha

Severely impaired insulin signaling in chronic wounds of diabetic ob/ob mice: a potential role of tumor necrosis factor-alpha

http://www.wikidata.org/entity/Q35088035

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Promising new treatments for psoriasis Exercise, Obesity, and Cutaneous Wound Healing: Evidence from Rodent and Human Studies Topical insulin accelerates wound healing in diabetes by enhancing the AKT and ERK pathways: a double-blind placebo-controlled clinical trial Anti-Inflammatory Effects of Rosiglitazone in Obesity-Impaired Wound Healing Depend on Adipocyte Differentiation Gsdma3 is a new factor needed for TNF-α-mediated apoptosis signal pathway in mouse skin keratinocytes Cell and molecular mechanisms of keratinocyte function stimulated by insulin during wound healing.Skin wound healing in diabetic β6 integrin-deficient mice.Wound healing in mice with high-fat diet- or ob gene-induced diabetes-obesity syndromes: a comparative study.p38 MAPK inhibition reduces diabetes-induced impairment of wound healing Cellular and molecular basis of wound healing in diabetes.Diabetic complications and dysregulated innate immunity.Transcription factor binding site analysis identifies FOXO transcription factors as regulators of the cutaneous wound healing process.Implant healing in experimental animal models of diabetes.Dysfunctional γδ T cells contribute to impaired keratinocyte homeostasis in mouse models of obesity Biphasic regulation of HMG-CoA reductase expression and activity during wound healing and its functional role in the control of keratinocyte angiogenic and proliferative responses.Development of low blood glucose readings in nine non-diabetic patients treated with tumor necrosis factor-alpha inhibitors: a case series.Antihyperglycemic effect of Annona squamosa hexane extract in type 2 diabetes animal model: PTP1B inhibition, a possible mechanism of action?Leptin, resistin and visfatin: the missing link between endocrine metabolic disorders and immunity.Models for diabetic wound healing and healing into percutaneous devices.Impaired glucose tolerance in pediatric burn patients at discharge from the acute hospital stay.Role of neuropeptides in skin inflammation and its involvement in diabetic wound healing.Skin wound healing modulation by macrophages.Blocking interleukin-1β in acute and chronic autoinflammatory diseases.Diabetic lower extremity wounds: the rationale for growth factors-based infiltration treatment.Linagliptin: from bench to bedside.Non-healing foot ulcers in diabetic patients: general and local interfering conditions and management options with advanced wound dressings.Effects of insulin on the skin: possible healing benefits for diabetic foot ulcers.Role of biomarkers in predicting diabetes complications with special reference to diabetic foot ulcers.A transgenic mouse model of inducible macrophage depletion: effects of diphtheria toxin-driven lysozyme M-specific cell lineage ablation on wound inflammatory, angiogenic, and contractive processes.TLR2 expression and signaling-dependent inflammation impair wound healing in diabetic mice.The effect of the glycolipoprotein extract (G-90) from earthworm Eisenia foetida on the wound healing process in alloxan-induced diabetic rats.Evolution of the Macrophage CD163 Phenotype and Cytokine Profiles in a Human Model of Resolving Inflammation.Vitamin D deficiency is associated with inflammatory cytokine concentrations in patients with diabetic foot infection.Bioburden after Staphylococcus aureus inoculation in type 1 diabetic rats undergoing internal fixation.Role of inflammation and insulin resistance in endothelial progenitor cell dysfunction.Selective blockade of the OGF-OGFr pathway by naltrexone accelerates fibroblast proliferation and wound healing.Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.Full-thickness splinted skin wound healing models in db/db and heterozygous mice: implications for wound healing impairment.Topical insulin application improves healing by regulating the wound inflammatory response.Association of the gene expression variation of tumor necrosis factor-α and expressions changes of dopamine receptor genes in progression of diabetic severe foot ulcers.

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P2860

Severely impaired insulin signaling in chronic wounds of diabetic ob/ob mice: a potential role of tumor necrosis factor-alpha

http://www.wikidata.org/entity/Q35088035

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Severely impaired insulin sign ...... of tumor necrosis factor-alpha

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Elke Müller

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Itamar Goren

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Josef Pfeilschifter

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Stefan Frank

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P2860

Positional cloning of the mouse obese gene and its human homologue

Leptin enhances wound re-epithelialization and constitutes a direct function of leptin in skin repair

GSK-3: tricks of the trade for a multi-tasking kinase

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance

Global and societal implications of the diabetes epidemic

Effects of the obese gene product on body weight regulation in ob/ob mice

C-reactive protein, interleukin 6, and risk of developing type 2 diabetes mellitus

The regulation of skin proliferation and differentiation in the IR null mouse: implications for skin complications of diabetes.

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