Abnormal cell responses and role of TNF-α in impaired diabetic wound healing. - Wikidata - awgldk - TriplyDB

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

http://www.wikidata.org/entity/Q43240676

about

Ceramide kinase is required for a normal eicosanoid response and the subsequent orderly migration of fibroblasts Gr-1+CD11b+ myeloid cells efficiently home to site of injury after intravenous administration and enhance diabetic wound healing by neoangiogenesis.FOXO1, TGF-β regulation and wound healing Association of TNF-α with impaired migration capacity of mesenchymal stem cells in patients with systemic lupus erythematosus.Dehydroabietic acid reverses TNF-α-induced the activation of FOXO1 and suppression of TGF-β1/Smad signaling in human adult dermal fibroblasts.Diabetes reduces mesenchymal stem cells in fracture healing through a TNFα-mediated mechanism Biphasic presence of fibrocytes in a porcine hypertrophic scar model.FOXO1 differentially regulates both normal and diabetic wound healing.Receptor-Interacting Protein Kinase 3 Deficiency Delays Cutaneous Wound Healing Effect of the herbal mixture composed of Aloe Vera, Henna, Adiantum capillus-veneris, and Myrrha on wound healing in streptozotocin-induced diabetic rats.Macrophage peroxisome proliferator-activated receptor γ deficiency delays skin wound healing through impairing apoptotic cell clearance in mice.Identification of Biomarkers for Footpad Dermatitis Development and Wound Healing.Wound management of chronic diabetic foot ulcers: from the basics to regenerative medicine.Diabetic ulcer regeneration: stem cells, biomaterials, growth factors.Microbiota of Chronic Diabetic Wounds: Ecology, Impact, and Potential for Innovative Treatment Strategies.PKCζ as a promising therapeutic target for TNFα-induced inflammatory disorders in chronic cutaneous wounds Impact of Trace Minerals on Wound Healing of Footpad Dermatitis in Broilers.Network Analysis of MPO and Other Relevant Proteins Involved in Diabetic Foot Ulcer and Other Diabetic Complications.Sheng-ji Hua-yu formula promotes diabetic wound healing of re-epithelization via Activin/Follistatin regulation.Mesenchymal stem cells in the treatment of Cesarean section skin scars: study protocol for a randomized, controlled trial.Hyperbaric oxygen treatment reduces neutrophil-endothelial adhesion in chronic wound conditions through S-nitrosation.miR-5591-5p regulates the effect of ADSCs in repairing diabetic wound via targeting AGEs/AGER/JNK signaling axis.Hesperidin, a plant flavonoid accelerated the cutaneous wound healing in streptozotocin-induced diabetic rats: Role of TGF-ß/Smads and Ang-1/Tie-2 signaling pathways.Evaluation of the Epithelial Barrier Function and Ileal Microbiome in an Established Necrotic Enteritis Challenge Model in Broiler Chickens

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P2860

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

http://www.wikidata.org/entity/Q43240676

description

2013 nî lūn-bûn

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2013年の論文

@ja

2013年学术文章

@wuu

2013年学术文章

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2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

@zh-hant

name

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

@en

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

@nl

type

label

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

@en

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

@nl

prefLabel

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

@en

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

@nl

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BioMed Research International

P1476

Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.

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Chenying Zhang

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Dana T Graves

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Creative Commons Attribution 3.0 Unported

P2860

A mechanism of suppression of TGF-beta/SMAD signaling by NF-kappa B/RelA

Delayed and deficient dermal maturation in mice lacking the CXCR3 ELR-negative CXC chemokine receptor

High glucose-mediated oxidative stress impairs cell migration.

Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance

Impaired gastric ulcer healing in diabetic rats: role of heat shock protein, growth factors, prostaglandins and proinflammatory cytokines

Lifetime risk for diabetes mellitus in the United States

The pattern of inflammatory/anti-inflammatory cytokines and chemokines in type 1 diabetic patients over time

Chemokine receptor CX3CR1 mediates skin wound healing by promoting macrophage and fibroblast accumulation and function

Suppression of PLCbeta2 by endotoxin plays a role in the adenosine A(2A) receptor-mediated switch of macrophages from an inflammatory to an angiogenic phenotype

Regulation of wound healing by growth factors and cytokines

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10.1155/2013/754802

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Tumor necrosis factor

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