Type I interferon production by tertiary lymphoid tissue developing in response to 2,6,10,14-tetramethyl-pentadecane (pristane) - Wikidata - awgldk - TriplyDB

Type I interferon production by tertiary lymphoid tissue developing in response to 2,6,10,14-tetramethyl-pentadecane (pristane)

Type I interferon production by tertiary lymphoid tissue developing in response to 2,6,10,14-tetramethyl-pentadecane (pristane)

http://www.wikidata.org/entity/Q35088161

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Silica, Silicosis, and Autoimmunity Ectopic lymphoid follicles: inducible centres for generating antigen-specific immune responses within tissues Role of type I interferons in the activation of autoreactive B cells Potential of Cells and Cytokines/Chemokines to Regulate Tertiary Lymphoid Structures in Human Diseases Mndal, a new interferon-inducible family member, is highly polymorphic, suppresses cell growth, and may modify plasmacytoma susceptibility Arthritis suppression by NADPH activation operates through an interferon-beta pathway High prevalence of autoantibodies to RNA helicase A in Mexican patients with systemic lupus erythematosus.Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps.Serum autoantibodies in pristane induced lupus are regulated by neutrophil gelatinase associated lipocalin Deficiency of the type I interferon receptor protects mice from experimental lupus.Distinct granuloma responses in C57BL/6J and BALB/cByJ mice in response to pristane.CD11b+Ly6C++Ly6G- cells show distinct function in mice with chronic inflammation or tumor burden.Toll-like receptor 2 is required for autoantibody production and development of renal disease in pristane-induced lupus.Requirements for innate immune pathways in environmentally induced autoimmunity.RORγt modulates macrophage recruitment during a hydrocarbon oil-induced inflammation Pathogenic role of B cells in the development of diffuse alveolar hemorrhage induced by pristane Differential development of oil granulomas induced by pristane injection in galectin-3 deficient mice Myeloid dendritic cells from B6.NZM Sle1/Sle2/Sle3 lupus-prone mice express an IFN signature that precedes disease onset B cell proliferation, somatic hypermutation, class switch recombination, and autoantibody production in ectopic lymphoid tissue in murine lupus.Immune-Mediated Nephropathy and Systemic Autoimmunity in Mice Does Not Require Receptor Interacting Protein Kinase 3 (RIPK3).A fully human monoclonal antibody with novel binding epitope and excellent neutralizing activity to multiple human IFN-α subtypes: A candidate therapy for systemic lupus erythematosus.Monocytes from Irf5-/- mice have an intrinsic defect in their response to pristane-induced lupus.IRF9 and STAT1 are required for IgG autoantibody production and B cell expression of TLR7 in mice.Maintenance of anti-Sm/RNP autoantibody production by plasma cells residing in ectopic lymphoid tissue and bone marrow memory B cells.Nuclear factor erythroid 2-related factor 2 is a critical target for the treatment of glucocorticoid-resistant lupus nephritis.TLR7-dependent and FcgammaR-independent production of type I interferon in experimental mouse lupus.Lymphocyte-specific TRAF3 transgenic mice have enhanced humoral responses and develop plasmacytosis, autoimmunity, inflammation, and cancer Type I interferon receptor controls B-cell expression of nucleic acid-sensing Toll-like receptors and autoantibody production in a murine model of lupus.Induction of autoimmunity by pristane and other naturally occurring hydrocarbons.Effects of acute and chronic inflammation on B-cell development and differentiation.Colocalization of antigen-specific B and T cells within ectopic lymphoid tissue following immunization with exogenous antigen.Type I interferon modulates monocyte recruitment and maturation in chronic inflammation.MicroRNA 155-deficiency leads to decreased autoantibody levels and reduced severity of nephritis and pneumonitis in pristane-induced lupus.Stimulation with the Aureobasidium pullulans-produced β-glucan effectively induces interferon stimulated genes in macrophage-like cell lines.Lymphotoxin signalling in tertiary lymphoid structures and immunotherapy.IL-1α modulates neutrophil recruitment in chronic inflammation induced by hydrocarbon oil A novel monoclonal antibody against human CD80 and its immune protection in a mouse lupus-like disease.Early IL-1 Signaling Promotes iBALT Induction after Influenza Virus Infection.Essential Requirement for IFN Regulatory Factor 7 in Autoantibody Production but Not Development of Nephritis in Murine Lupus.Basophils contribute to pristane-induced Lupus-like nephritis model.

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Type I interferon production by tertiary lymphoid tissue developing in response to 2,6,10,14-tetramethyl-pentadecane (pristane)

http://www.wikidata.org/entity/Q35088161

description

2006 nî lūn-bûn

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2006 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2006 թվականի ապրիլին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Type I interferon production b ...... amethyl-pentadecane (pristane)

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Type I interferon production b ...... amethyl-pentadecane (pristane)

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Type I interferon production b ...... amethyl-pentadecane (pristane)

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The American Journal of Pathology

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Type I interferon production b ...... amethyl-pentadecane (pristane)

@en

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Dina C Nacionales

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Eric Sobel

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Haoyang Zhuang

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Jason S Weinstein

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Jun Akaogi

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Kindra M Kelly

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Minoru Satoh

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Pui Y Lee

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Westley H Reeves

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Yi Li

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P2860

Plasmacytoid dendritic cells induce plasma cell differentiation through type I interferon and interleukin 6

In vivo microbial stimulation induces rapid CD40 ligand-independent production of interleukin 12 by dendritic cells and their redistribution to T cell areas

The induction of tolerance by dendritic cells that have captured apoptotic cells

A chemokine expressed in lymphoid high endothelial venules promotes the adhesion and chemotaxis of naive T lymphocytes

Dendritic cells and the control of immunity

Induction of lupus autoantibodies by adjuvants

Interferon-inducible gene expression signature in peripheral blood cells of patients with severe lupus

DCs induce CD40-independent immunoglobulin class switching through BLyS and APRIL

Exogenous lipoid pneumonia

Interferon and granulopoiesis signatures in systemic lupus erythematosus blood

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