Monocytes from Irf5-/- mice have an intrinsic defect in their response to pristane-induced lupus.
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Linking susceptibility genes and pathogenesis mechanisms using mouse models of systemic lupus erythematosusAn AAAG-Rich Oligodeoxynucleotide Rescues Mice from Bacterial Septic Peritonitis by Interfering Interferon Regulatory Factor 5.Serum autoantibodies in pristane induced lupus are regulated by neutrophil gelatinase associated lipocalinAn essential role of caspase 1 in the induction of murine lupus and its associated vascular damageRequirements for innate immune pathways in environmentally induced autoimmunity.The IRF5-TNPO3 association with systemic lupus erythematosus has two components that other autoimmune disorders variably shareC1q-mediated repression of human monocytes is regulated by leukocyte-associated Ig-like receptor 1 (LAIR-1).IRF5 controls both acute and chronic inflammation.The interferon regulatory factors as novel potential targets in the treatment of cardiovascular diseases.Caspase-1 is required for maintenance of marginal zone B cells in pristane-induced lupusC1q Modulates the Response to TLR7 Stimulation by Pristane-Primed Macrophages: Implications for Pristane-Induced Lupus.Specific detection of interferon regulatory factor 5 (IRF5): A case of antibody inequality.Thymidine phosphorylase exerts complex effects on bone resorption and formation in myeloma.IRF5 governs liver macrophage activation that promotes hepatic fibrosis in mice and humans.In vivo silencing of the transcription factor IRF5 reprograms the macrophage phenotype and improves infarct healing.Epigenetics and the IRFs: a complex interplay in the control of immunity and autoimmunity.Blocking the VISTA pathway enhances disease progression in (NZB × NZW) F1 female mice.IRF5, IRF8, and IRF7 in human pDCs - the good, the bad, and the insignificant?Deletion of Irf5 protects hematopoietic stem cells from DNA damage-induced apoptosis and suppresses γ-irradiation-induced thymic lymphomagenesis.
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P2860
Monocytes from Irf5-/- mice have an intrinsic defect in their response to pristane-induced lupus.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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name
Monocytes from Irf5-/- mice ha ...... nse to pristane-induced lupus.
@ast
Monocytes from Irf5-/- mice ha ...... nse to pristane-induced lupus.
@en
type
label
Monocytes from Irf5-/- mice ha ...... nse to pristane-induced lupus.
@ast
Monocytes from Irf5-/- mice ha ...... nse to pristane-induced lupus.
@en
prefLabel
Monocytes from Irf5-/- mice ha ...... nse to pristane-induced lupus.
@ast
Monocytes from Irf5-/- mice ha ...... nse to pristane-induced lupus.
@en
P2093
P2860
P356
P1476
Monocytes from Irf5-/- mice ha ...... nse to pristane-induced lupus.
@en
P2093
Betsy J Barnes
Lisong Yang
Rivka C Stone
Xiaohui Bi
P2860
P304
P356
10.4049/JIMMUNOL.1201162
P407
P577
2012-08-29T00:00:00Z