CASZ1, a candidate tumor-suppressor gene, suppresses neuroblastoma tumor growth through reprogramming gene expression. - Wikidata - awgldk - TriplyDB

CASZ1, a candidate tumor-suppressor gene, suppresses neuroblastoma tumor growth through reprogramming gene expression.

CASZ1, a candidate tumor-suppressor gene, suppresses neuroblastoma tumor growth through reprogramming gene expression.

http://www.wikidata.org/entity/Q35092733

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Lithocholic bile acid selectively kills neuroblastoma cells, while sparing normal neuronal cells Gene-age interactions in blood pressure regulation: a large-scale investigation with the CHARGE, Global BPgen, and ICBP Consortia Congenital heart disease protein 5 associates with CASZ1 to maintain myocardial tissue integrity.The CASZ1/Egfl7 transcriptional pathway is required for RhoA expression in vascular endothelial cells.CASZ1b, the short isoform of CASZ1 gene, coexpresses with CASZ1a during neurogenesis and suppresses neuroblastoma cell growth Genistein suppresses prostate cancer growth through inhibition of oncogenic microRNA-151.Essential role of the zinc finger transcription factor Casz1 for mammalian cardiac morphogenesis and development Deep clonal profiling of formalin fixed paraffin embedded clinical samples Therapeutic targets for neuroblastomas.Characterization of critical domains within the tumor suppressor CASZ1 required for transcriptional regulation and growth suppression Reversal of Chemoresistance in Ovarian Cancer by Co-Delivery of a P-Glycoprotein Inhibitor and Paclitaxel in a Liposomal Platform.Loss of fragile histidine triad and amplification of 1p36.22 and 11p15.5 in primary gastric adenocarcinomas.Characterization of long noncoding RNA and messenger RNA signatures in melanoma tumorigenesis and metastasis.EZH2 Mediates epigenetic silencing of neuroblastoma suppressor genes CASZ1, CLU, RUNX3, and NGFR Zinc finger transcription factor CASZ1 interacts with histones, DNA repair proteins and recruits NuRD complex to regulate gene transcription UBE4B levels are correlated with clinical outcomes in neuroblastoma patients and with altered neuroblastoma cell proliferation and sensitivity to epidermal growth factor receptor inhibitors Fine map of the Gct1 spontaneous ovarian granulosa cell tumor locus.Castor is required for Hedgehog-dependent cell-fate specification and follicle stem cell maintenance in Drosophila oogenesis.The Cardiac TBX5 Interactome Reveals a Chromatin Remodeling Network Essential for Cardiac Septation CASZ1 is a novel promoter of metastasis in ovarian cancer.CASZ1 inhibits cell cycle progression in neuroblastoma by restoring pRb activity Long non-coding RNA containing ultraconserved genomic region 8 promotes bladder cancer tumorigenesis.Identification of CASZ1 NES reveals potential mechanisms for loss of CASZ1 tumor suppressor activity in neuroblastoma.Genetically engineered murine models--contribution to our understanding of the genetics, molecular pathology and therapeutic targeting of neuroblastoma.The role of genetic and epigenetic alterations in neuroblastoma disease pathogenesis.Neuroblastoma patient outcomes, tumor differentiation, and ERK activation are correlated with expression levels of the ubiquitin ligase UBE4B.Cancer driver gene discovery through an integrative genomics approach in a non-parametric Bayesian framework.Differential regulation of CASZ1 protein expression during cardiac and skeletal muscle development.The calcium-sensing receptor is silenced by genetic and epigenetic mechanisms in unfavorable neuroblastomas and its reactivation induces ERK1/2-dependent apoptosis.Zinc finger transcription factor Casz1 expression is regulated by homeodomain transcription factor Prrxl1 in embryonic spinal dorsal horn late-born excitatory interneurons.Identification of Casz1 as a Regulatory Protein Controlling T Helper Cell Differentiation, Inflammation, and Immunity.Temporal requirements for ISL1 in sympathetic neuron proliferation, differentiation, and diversification.Downregulation of castor zinc finger 1 predicts poor prognosis and facilitates hepatocellular carcinoma progression via MAPK/ERK signaling.Loss of gene function as a consequence of human papillomavirus DNA integration.

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CASZ1, a candidate tumor-suppressor gene, suppresses neuroblastoma tumor growth through reprogramming gene expression.

http://www.wikidata.org/entity/Q35092733

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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CASZ1, a candidate tumor-suppr ...... reprogramming gene expression.

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CASZ1, a candidate tumor-suppr ...... reprogramming gene expression.

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CASZ1, a candidate tumor-suppr ...... reprogramming gene expression.

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CASZ1, a candidate tumor-suppr ...... reprogramming gene expression.

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Cell Death & Differentiation

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CASZ1, a candidate tumor-suppr ...... reprogramming gene expression.

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B Mock

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TRPM7 regulates cell adhesion by controlling the calcium-dependent protease calpain

ONCOMINE: a cancer microarray database and integrated data-mining platform

CHD5 is a tumor suppressor at human 1p36

ICAM-2 expression mediates a membrane-actin link, confers a nonmetastatic phenotype and reflects favorable tumor stage or histology in neuroblastoma.

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The kinesin KIF1Bbeta acts downstream from EglN3 to induce apoptosis and is a potential 1p36 tumor suppressor

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10.1038/CDD.2010.187

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7

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