PPARγ: a circadian transcription factor in adipogenesis and osteogenesis. - Wikidata - awgldk - TriplyDB

PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

http://www.wikidata.org/entity/Q35093192

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Interdependence of nutrient metabolism and the circadian clock system: Importance for metabolic health Childhood obesity, bone development, and cardiometabolic risk factors Evolving roles of circadian rhythms in liver homeostasis and pathology The alliance of mesenchymal stem cells, bone, and diabetes Nocturnin: at the crossroads of clocks and metabolism Increased sensitivity of the circadian system to temporal changes in the feeding regime of spontaneously hypertensive rats - a potential role for Bmal2 in the liver Demographically-Based Evaluation of Genomic Regions under Selection in Domestic Dogs Transcriptome-Wide Analysis Reveals the Role of PPARγ Controlling the Lipid Metabolism in Goat Mammary Epithelial Cells Acoustic-frequency vibratory stimulation regulates the balance between osteogenesis and adipogenesis of human bone marrow-derived mesenchymal stem cells PPARD is an Inhibitor of Cartilage Growth in External Ears Central adiponectin administration reveals new regulatory mechanisms of bone metabolism in mice PPARγ silencing enhances osteogenic differentiation of human adipose-derived mesenchymal stem cells.A novel PPARγ2 modulator sLZIP controls the balance between adipogenesis and osteogenesis during mesenchymal stem cell differentiation Effects of pioglitazone mediated activation of PPAR-γ on CIDEC and obesity related changes in mice.Increased marrow adiposity in premenopausal women with idiopathic osteoporosis Is graphene a promising nano-material for promoting surface modification of implants or scaffold materials in bone tissue engineering?Function of SREBP1 in the milk fat synthesis of dairy cow mammary epithelial cells Retinaldehyde dehydrogenase 1 deficiency inhibits PPARγ-mediated bone loss and marrow adiposity.Early chronotype and tissue-specific alterations of circadian clock function in spontaneously hypertensive rats.Melatonin, noncoding RNAs, messenger RNA stability and epigenetics--evidence, hints, gaps and perspectives.Activation of Akt by advanced glycation end products (AGEs): involvement of IGF-1 receptor and caveolin-1 Novel insulin sensitizer modulates nutrient sensing pathways and maintains β-cell phenotype in human islets.Loss of JNK2 increases intestinal tumor susceptibility in Apc1638+/- mice with dietary modulation.Minireview: nuclear receptor regulation of osteoclast and bone remodeling.The Fragile X proteins Fmrp and Fxr2p cooperate to regulate glucose metabolism in mice.S-nitrosoglutathione reductase-dependent PPARγ denitrosylation participates in MSC-derived adipogenesis and osteogenesis A high-fat diet induces bone loss in mice lacking the Alox5 gene Long-Term Endurance Exercise in Humans Stimulates Cell Fusion of Myoblasts along with Fusogenic Endogenous Retroviral Genes In Vivo Fibroblast growth factor 21 promotes bone loss by potentiating the effects of peroxisome proliferator-activated receptor γ.Oxidative stress and heme oxygenase-1 regulated human mesenchymal stem cells differentiation Nutraceuticals as Ligands of PPARγ Thiazolidinediones induce osteocyte apoptosis by a G protein-coupled receptor 40-dependent mechanism.Histone demethylases KDM4B and KDM6B promotes osteogenic differentiation of human MSCs.Reduced osteoblast activity in the mice lacking TR4 nuclear receptor leads to osteoporosis.Gene Expression and Correlation of Pten and Fabp4 in Liver, Muscle, and Adipose Tissues of Type 2 Diabetes Rats.Regulation of the osteogenic and adipogenic differentiation of bone marrow-derived stromal cells by extracellular uridine triphosphate: The role of P2Y2 receptor and ERK1/2 signaling.Epigenetic Mechanisms Regulating Mesenchymal Stem Cell Differentiation.Redundant miR-3077-5p and miR-705 mediate the shift of mesenchymal stem cell lineage commitment to adipocyte in osteoporosis bone marrow Kiss your tail goodbye: the role of PARN, Nocturnin, and Angel deadenylases in mRNA biology.PPAR γ Regulates Genes Involved in Triacylglycerol Synthesis and Secretion in Mammary Gland Epithelial Cells of Dairy Goats

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PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

http://www.wikidata.org/entity/Q35093192

description

2010 nî lūn-bûn

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2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

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PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

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type

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PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

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PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

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prefLabel

PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

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PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

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NRENDO.2010.155

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Nature Reviews Endocrinology

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PPARγ: a circadian transcription factor in adipogenesis and osteogenesis.

@en

P2093

Clifford J Rosen

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Masanobu Kawai

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P2860

BMAL1 and CLOCK, two essential components of the circadian clock, are involved in glucose homeostasis

Identification of four families of yCCR4- and Mg2+-dependent endonuclease-related proteins in higher eukaryotes, and characterization of orthologs of yCCR4 with a conserved leucine-rich repeat essential for hCAF1/hPOP2 binding

Molecular recognition of fatty acids by peroxisome proliferator-activated receptors

Endocrine regulation of energy metabolism by the skeleton

Obesity and metabolic syndrome in circadian Clock mutant mice

Insulin receptor signaling in osteoblasts regulates postnatal bone acquisition and body composition

Rhythmic expression of Nocturnin mRNA in multiple tissues of the mouse.

The transcription factor associated Ccr4 and Caf1 proteins are components of the major cytoplasmic mRNA deadenylase in Saccharomyces cerevisiae.

The Krüppel-like factor KLF2 inhibits peroxisome proliferator-activated receptor-gamma expression and adipogenesis

The orphan nuclear receptor REV-ERBalpha controls circadian transcription within the positive limb of the mammalian circadian oscillator

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nrendo.2010.155

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629-636

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scholarly article

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10.1038/NRENDO.2010.155

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11

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6

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2010-09-07T00:00:00Z

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46158616

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20820194

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circadian rhythm

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3132113

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