Pathogenic role of P-selectin in experimental cerebral malaria: importance of the endothelial compartment
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Endothelial-Leukocyte Interaction in Severe Malaria: Beyond the BrainExperimental cerebral malaria pathogenesis--hemodynamics at the blood brain barrierP-selectin is a host receptor for Plasmodium MSP7 ligandsA contrast agent recognizing activated platelets reveals murine cerebral malaria pathology undetectable by conventional MRIThe role of platelets in the pathogenesis of cerebral malaria.Plasmodium falciparum adhesion on human brain microvascular endothelial cells involves transmigration-like cup formation and induces opening of intercellular junctions.Sequestration and histopathology in Plasmodium chabaudi malaria are influenced by the immune response in an organ-specific mannerInflammatory responses associated with the induction of cerebral malaria: lessons from experimental murine modelsReview Article: blood-brain barrier in falciparum malaria.An evolutionary history of the selectin gene cluster in humans.Host biomarkers and biological pathways that are associated with the expression of experimental cerebral malaria in mice.Angiopoietin-2 is associated with decreased endothelial nitric oxide and poor clinical outcome in severe falciparum malaria.Potential efficacy of citicoline as adjunct therapy in treatment of cerebral malaria.Host resistance to malaria: using mouse models to explore the host response.Microvesicles and exosomes as vehicles between protozoan and host cell communication.Platelets in inflammation and immunity.The immunological balance between host and parasite in malaria.Characterisation of Plasmodium falciparum populations selected on the human endothelial receptors P-selectin, E-selectin, CD9 and CD151.Experimental cerebral malaria progresses independently of the Nlrp3 inflammasome.Sphingosine-1-phosphate receptor 3 promotes leukocyte rolling by mobilizing endothelial P-selectin.CXCR3 determines strain susceptibility to murine cerebral malaria by mediating T lymphocyte migration toward IFN-gamma-induced chemokines.Low nitric oxide bioavailability contributes to the genesis of experimental cerebral malaria.von Willebrand factor propeptide in malaria: evidence of acute endothelial cell activation.
P2860
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P2860
Pathogenic role of P-selectin in experimental cerebral malaria: importance of the endothelial compartment
description
2004 nî lūn-bûn
@nan
2004 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մարտին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Pathogenic role of P-selectin ...... of the endothelial compartment
@ast
Pathogenic role of P-selectin ...... of the endothelial compartment
@en
type
label
Pathogenic role of P-selectin ...... of the endothelial compartment
@ast
Pathogenic role of P-selectin ...... of the endothelial compartment
@en
prefLabel
Pathogenic role of P-selectin ...... of the endothelial compartment
@ast
Pathogenic role of P-selectin ...... of the endothelial compartment
@en
P2093
P2860
P921
P1476
Pathogenic role of P-selectin ...... of the endothelial compartment
@en
P2093
Alexander R Rosenkranz
Dietmar Vestweber
Georges E Grau
Giampaolo Pizzolato
Mireille Redard
Tanya N Mayadas
Valéry Combes
P2860
P304
P356
10.1016/S0002-9440(10)63166-5
P407
P577
2004-03-01T00:00:00Z