The effect of glatiramer acetate therapy on functional properties of B cells from patients with relapsing-remitting multiple sclerosis. - Wikidata - awgldk - TriplyDB

The effect of glatiramer acetate therapy on functional properties of B cells from patients with relapsing-remitting multiple sclerosis.

The effect of glatiramer acetate therapy on functional properties of B cells from patients with relapsing-remitting multiple sclerosis.

http://www.wikidata.org/entity/Q35105313

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B Cells Are Multifunctional Players in Multiple Sclerosis Pathogenesis: Insights from Therapeutic Interventions Cytokine-Defined B Cell Responses as Therapeutic Targets in Multiple Sclerosis Circulating immune cells in multiple sclerosis.Seeking balance: Potentiation and inhibition of multiple sclerosis autoimmune responses by IL-6 and IL-10.Myelin Basic Protein-Induced Production of Tumor Necrosis Factor-α and Interleukin-6, and Presentation of the Immunodominant Peptide MBP85-99 by B Cells from Patients with Relapsing-Remitting Multiple Sclerosis.The brain antigen-specific B cell response correlates with glatiramer acetate responsiveness in relapsing-remitting multiple sclerosis patients Regulatory T Cell Dysfunction Acquiesces to BTLA+ Regulatory B Cells Subsequent to Oral Intervention in Experimental Autoimmune Encephalomyelitis B Lymphocytes in Multiple Sclerosis: Bregs and BTLA/CD272 Expressing-CD19+ Lymphocytes Modulate Disease Severity.CD40-Mediated NF-κB Activation in B Cells Is Increased in Multiple Sclerosis and Modulated by Therapeutics.Glatiramer acetate: long-term safety and efficacy in relapsing-remitting multiple sclerosis.B cells and their cytokine activities implications in human diseases.B Cell-Directed Therapeutics in Multiple Sclerosis: Rationale and Clinical Evidence.Memory B Cells are Major Targets for Effective Immunotherapy in Relapsing Multiple Sclerosis.Deciphering the Role of B Cells in Multiple Sclerosis-Towards Specific Targeting of Pathogenic Function.Role of Immunological Memory Cells as a Therapeutic Target in Multiple Sclerosis.Effects of rituximab and dexamethasone on regulatory and proinflammatory B-cell subsets in patients with primary immune thrombocytopenia.Efficacy of glatiramer acetate in neuromyelitis optica spectrum disorder: a multicenter retrospective study.Impact of Glatiramer Acetate on B Cell-Mediated Pathogenesis of Multiple Sclerosis Regulatory B and T lymphocytes in multiple sclerosis: friends or foes?

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The effect of glatiramer acetate therapy on functional properties of B cells from patients with relapsing-remitting multiple sclerosis.

http://www.wikidata.org/entity/Q35105313

description

2014 nî lūn-bûn

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2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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The effect of glatiramer aceta ...... -remitting multiple sclerosis.

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The effect of glatiramer aceta ...... -remitting multiple sclerosis.

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The effect of glatiramer aceta ...... -remitting multiple sclerosis.

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The effect of glatiramer aceta ...... -remitting multiple sclerosis.

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JAMANEUROL.2014.1472

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The effect of glatiramer aceta ...... g-remitting multiple sclerosis

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Alyssa A Guzman

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Angela Flores

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Benjamin Greenberg

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Dina E O'Brien

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Donna Graves

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Elliot M Frohman

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Gina Remington

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Laurie S Davis

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Nancy L Monson

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Samuel Hughes

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P2860

Activation of B cells by non-canonical helper signals

Correlation of circulating CD27high plasma cells and disease activity in systemic lupus erythematosus

Glatiramer acetate (Copaxone) induces degenerate, Th2-polarized immune responses in patients with multiple sclerosis.

Induction of CD4+CD25+ regulatory T cells by copolymer-I through activation of transcription factor Foxp3

CD19(+)CD24(hi)CD38(hi) B cells exhibit regulatory capacity in healthy individuals but are functionally impaired in systemic Lupus Erythematosus patients.

Characterization of a rare IL-10-competent B-cell subset in humans that parallels mouse regulatory B10 cells.

Aberrant CD40-induced NF-κB activation in human lupus B lymphocytes

Memory B cells from a subset of treatment-naïve relapsing-remitting multiple sclerosis patients elicit CD4(+) T-cell proliferation and IFN-γ production in response to myelin basic protein and myelin oligodendrocyte glycoprotein.

Glatiramer acetate (Copaxone) therapy induces CD8(+) T cell responses in patients with multiple sclerosis

Helminth infections associated with multiple sclerosis induce regulatory B cells.

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10.1001/JAMANEUROL.2014.1472

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multiple sclerosis

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