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B cells from relapsing remitting multiple sclerosis patients support neuro-antigen-specific Th17 responsesA genome-wide screen to identify transcription factors expressed in pelvic Ganglia of the lower urinary tractThe effect of glatiramer acetate therapy on functional properties of B cells from patients with relapsing-remitting multiple sclerosis.Single dose of glycoengineered anti-CD19 antibody (MEDI551) disrupts experimental autoimmune encephalomyelitis by inhibiting pathogenic adaptive immune responses in the bone marrow and spinal cord while preserving peripheral regulatory mechanisms.Seeking balance: Potentiation and inhibition of multiple sclerosis autoimmune responses by IL-6 and IL-10.Changes in JC virus-specific T cell responses during natalizumab treatment and in natalizumab-associated progressive multifocal leukoencephalopathy.Elevated CNS inflammation in patients with preclinical Alzheimer's disease.CD40-Mediated NF-κB Activation in B Cells Is Increased in Multiple Sclerosis and Modulated by Therapeutics.Peripheral VH4+ plasmablasts demonstrate autoreactive B cell expansion toward brain antigens in early multiple sclerosis patients.Antibody-independent B cell effector functions in relapsing remitting multiple sclerosis: clues to increased inflammatory and reduced regulatory B cell capacity.Adaptive lymphocyte profiles correlate to brain Aβ burden in patients with mild cognitive impairment.Migration pathways of sacral neural crest during development of lower urogenital tract innervation.Repetitive hypoxic preconditioning induces an immunosuppressed B cell phenotype during endogenous protection from stroke.Autoreactive CD19+CD20- Plasma Cells Contribute to Disease Severity of Experimental Autoimmune Encephalomyelitis.
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P50
description
hulumtuese
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onderzoeker
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researcher
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հետազոտող
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Sara J Ireland
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Sara J Ireland
@en
Sara J Ireland
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Sara J Ireland
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type
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Sara J Ireland
@ast
Sara J Ireland
@en
Sara J Ireland
@es
Sara J Ireland
@nl
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Sara J Ireland
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Sara J Ireland
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Sara J Ireland
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Sara J Ireland
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0000-0002-7811-197X