about
Mechanisms of disease reversal in focal and segmental glomerulosclerosisIntravital imaging of podocyte calcium in glomerular injury and disease.TGF-β-activated kinase 1 is crucial in podocyte differentiation and glomerular capillary formation.The Role of SIRT1 in Diabetic Kidney Disease.Obesity in kidney disease: A heavyweight opponent.Mosaicism of podocyte involvement is related to podocyte injury in females with Fabry diseaseProteomic analysis of formalin-fixed paraffin-embedded glomeruli suggests depletion of glomerular filtration barrier proteins in two-kidney, one-clip hypertensive ratsThe role of retinoic acid receptors in the signal pathway of all-trans retinoic acid-induced differentiation in adriamycin-induced podocyte injury.Podocyte injury-driven intracapillary plasminogen activator inhibitor type 1 accelerates podocyte loss via uPAR-mediated β1-integrin endocytosis.Endothelin and the podocyteTriptolide Attenuates Podocyte Injury by Regulating Expression of miRNA-344b-3p and miRNA-30b-3p in Rats with Adriamycin-Induced NephropathyAnti-TGF-β Antibody, 1D11, Ameliorates Glomerular Fibrosis in Mouse Models after the Onset of Proteinuria.Following specific podocyte injury captopril protects against progressive long term renal damage.The phenotypes of podocytes and parietal epithelial cells may overlap in diabetic nephropathy.Parietal epithelial cell activation marker in early recurrence of FSGS in the transplant.Recent progress in the pathophysiology and treatment of FSGS recurrence.The Level of Serum and Urinary Nephrin in Normal Pregnancy and Pregnancy with Subsequent PreeclampsiaKeap1 inhibition attenuates glomerulosclerosis.Mechanistic Insight and Management of Diabetic Nephropathy: Recent Progress and Future Perspective.Alport syndrome--insights from basic and clinical research.Human kidney cell reprogramming: applications for disease modeling and personalized medicineThe pathogenesis of focal segmental glomerulosclerosis.Causes and pathogenesis of focal segmental glomerulosclerosis.The kidney in obesity.Minimal change disease and idiopathic FSGS: manifestations of the same disease.Podocyturia: Potential applications and current limitations.SLIT2/ROBO2 signaling pathway inhibits nonmuscle myosin IIA activity and destabilizes kidney podocyte adhesion.Glomerular homeostasis requires a match between podocyte mass and metabolic load.Podocyte injury: the role of proteinuria, urinary plasminogen, and oxidative stress.Obesity-related glomerulopathy: pathogenesis, pathologic, clinical characteristics and treatment.Urinary podocyte and TGF-β1 mRNA as markers for disease activity and progression in anti-glomerular basement membrane nephritis.Rac1 in podocytes promotes glomerular repair and limits the formation of sclerosis.Mice are unable to endogenously regenerate podocytes during the repair of immunotoxin-induced glomerular injury.Similar Biophysical Abnormalities in Glomeruli and Podocytes from Two Distinct Models.Podocytopenia, parietal epithelial cells and glomerulosclerosis
P2860
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P2860
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Podocyte injury damages other podocytes.
@ast
Podocyte injury damages other podocytes.
@en
type
label
Podocyte injury damages other podocytes.
@ast
Podocyte injury damages other podocytes.
@en
prefLabel
Podocyte injury damages other podocytes.
@ast
Podocyte injury damages other podocytes.
@en
P2093
P2860
P356
P1476
Podocyte injury damages other podocytes.
@en
P2093
Agnes B Fogo
Ayumi Shintani
Eric Sandgren
Iekuni Ichikawa
Ira Pastan
Taiji Matsusaka
Valentina Kon
P2860
P304
P356
10.1681/ASN.2010090963
P577
2011-06-30T00:00:00Z