Superoxide production by macrophages and T cells is critical for the induction of autoreactivity and type 1 diabetes - Wikidata - awgldk - TriplyDB

Superoxide production by macrophages and T cells is critical for the induction of autoreactivity and type 1 diabetes

Superoxide production by macrophages and T cells is critical for the induction of autoreactivity and type 1 diabetes

http://www.wikidata.org/entity/Q35123480

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Multiple sclerosis: molecular mechanisms and therapeutic opportunities Generation of improved humanized mouse models for human infectious diseases.Is HOT a cool treatment for type 1 diabetes?Loss of NADPH oxidase-derived superoxide skews macrophage phenotypes to delay type 1 diabetes How the location of superoxide generation influences the β-cell response to nitric oxide.Hydrogen-bonded multilayers of tannic acid as mediators of T-cell immunity.Use of Humanized Mice to Study the Pathogenesis of Autoimmune and Inflammatory Diseases Peripheral blood monocyte gene expression profile clinically stratifies patients with recent-onset type 1 diabetes.Calcium-independent phospholipases A2 and their roles in biological processes and diseases.Regulation of B lymphocyte responses to Toll-like receptor ligand binding during diabetes prevention in non-obese diabetic (NOD) mice Redox Regulation of Pro-IL-1β Processing May Contribute to the Increased Severity of Serum-Induced Arthritis in NOX2-Deficient Mice.Loss of NOX-Derived Superoxide Exacerbates Diabetogenic CD4 T-Cell Effector Responses in Type 1 Diabetes Cytotoxic mechanisms employed by mouse T cells to destroy pancreatic β-cells.Decreased STAT5 phosphorylation and GATA-3 expression in NOX2-deficient T cells: role in T helper development Dysregulated TLR3-dependent signaling and innate immune activation in superoxide-deficient macrophages from nonobese diabetic mice Comparative genetics: synergizing human and NOD mouse studies for identifying genetic causation of type 1 diabetes Do β-cells generate peroxynitrite in response to cytokine treatment?A parasite-derived 68-mer peptide ameliorates autoimmune disease in murine models of Type 1 diabetes and multiple sclerosis.Rpl13a small nucleolar RNAs regulate systemic glucose metabolism.Oxidative stress induced by HIV-1 reverse transcriptase modulates the enzyme's performance in gene immunization.Type 1 diabetes mellitus in 2011: Heterogeneity of T1DM raises questions for therapy.The role of reactive oxygen species and proinflammatory cytokines in type 1 diabetes pathogenesis.Mitochondria as oxidative signaling organelles in T-cell activation: physiological role and pathological implications.Are reactive oxygen species still the basis for diabetic complications?Mechanism of BDE209-induced impaired glucose homeostasis based on gene microarray analysis of adult rat liver.Reactive oxygen homeostasis - the balance for preventing autoimmunity.NADPH Oxidase-Derived Superoxide Provides a Third Signal for CD4 T Cell Effector Responses.Radical innate regulation of autoimmune diabetes.T cells display mitochondria hyperpolarization in human type 1 diabetes.Redox-Sensitive Innate Immune Pathways During Macrophage Activation in Type 1 Diabetes.T Cell-Mediated Beta Cell Destruction: Autoimmunity and Alloimmunity in the Context of Type 1 Diabetes.Superoxide Production by NADPH Oxidase Intensifies Macrophage Antiviral Responses during Diabetogenic Coxsackievirus Infection.The Type 1 Diabetes-Resistance Locus Idd22 Controls Trafficking of Autoreactive CTLs into the Pancreatic Islets of NOD Mice.Mitochondrial Reactive Oxygen Species and Type 1 Diabetes.Redox-Dependent Inflammation in Islet Transplantation Rejection.Protective Role of Myeloid Cells Expressing a G-CSF Receptor Polymorphism in an Induced Model of Lupus.

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Superoxide production by macrophages and T cells is critical for the induction of autoreactivity and type 1 diabetes

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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Superoxide production by macro ...... reactivity and type 1 diabetes

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Superoxide production by macro ...... reactivity and type 1 diabetes

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Superoxide production by macro ...... reactivity and type 1 diabetes

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Chao Liu

http://www.w3.org/2001/XMLSchema#string

Clayton E Mathews

http://www.w3.org/2001/XMLSchema#string

Hubert M Tse

http://www.w3.org/2001/XMLSchema#string

Jing Chen

http://www.w3.org/2001/XMLSchema#string

Jon D Piganelli

http://www.w3.org/2001/XMLSchema#string

Lindsey E Padgett

http://www.w3.org/2001/XMLSchema#string

Lyle L Moldawer

http://www.w3.org/2001/XMLSchema#string

Mani Annamali

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P2860

Positional identification of Ncf1 as a gene that regulates arthritis severity in rats

T cells express a phagocyte-type NADPH oxidase that is activated after T cell receptor stimulation

Positional cloning of Ncf1--a piece in the puzzle of arthritis genetics.

Definition of arthritis candidate risk genes by combining rat linkage-mapping results with human case-control association data.

The nonphagocytic NADPH oxidase Duox1 mediates a positive feedback loop during T cell receptor signaling.

Reactive oxygen species as intracellular messengers during cell growth and differentiation.

The nonobese diabetic mouse as a model of autoimmune diabetes: immune dysregulation gets the NOD.

Biological defense mechanisms. The production by leukocytes of superoxide, a potential bactericidal agent.

Unusual resistance of ALR/Lt mouse beta cells to autoimmune destruction: role for beta cell-expressed resistance determinants

The superoxide-generating NADPH oxidase: structural aspects and activation mechanism.

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2144-2151

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scholarly article

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10.2337/DB10-1222

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P433

8

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60

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Matthew J. Delano

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2011-06-29T00:00:00Z

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51454132

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21715554

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type-1 diabetes

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3142064

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