Loss of inverse relationship between pulsatile insulin and glucagon secretion in patients with type 2 diabetes - Wikidata - awgldk - TriplyDB

Loss of inverse relationship between pulsatile insulin and glucagon secretion in patients with type 2 diabetes

Loss of inverse relationship between pulsatile insulin and glucagon secretion in patients with type 2 diabetes

http://www.wikidata.org/entity/Q35123524

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Incretin therapies: highlighting common features and differences in the modes of action of glucagon-like peptide-1 receptor agonists and dipeptidyl peptidase-4 inhibitors Pancreatic α-Cell Dysfunction in Type 2 Diabetes: Old Kids on the Block Phosphofructo-2-kinase/fructose-2,6-bisphosphatase modulates oscillations of pancreatic islet metabolism Design Principles of Pancreatic Islets: Glucose-Dependent Coordination of Hormone Pulses Connexin-36 gap junctions regulate in vivo first- and second-phase insulin secretion dynamics and glucose tolerance in the conscious mouse Direct measurements of oscillatory glycolysis in pancreatic islet β-cells using novel fluorescence resonance energy transfer (FRET) biosensors for pyruvate kinase M2 activity A Local Counter-Regulatory Motif Modulates the Global Phase of Hormonal Oscillations.Acute glucagon induces postprandial peripheral insulin resistance.Minireview: Glucagon in the pathogenesis of hypoglycemia and hyperglycemia in diabetes.Efficacy and Acceptability of Glycemic Control of Glucagon-Like Peptide-1 Receptor Agonists among Type 2 Diabetes: A Systematic Review and Network Meta-Analysis Pulsatile insulin secretion, impaired glucose tolerance and type 2 diabetes.Ca2+ Effects on ATP Production and Consumption Have Regulatory Roles on Oscillatory Islet Activity.Neuronostatin acts via GPR107 to increase cAMP-independent PKA phosphorylation and proglucagon mRNA accumulation in pancreatic α-cells Paracrine regulation of glucagon secretion: the β/α/δ model.Postprandial metabolite profiles reveal differential nutrient handling after bariatric surgery compared with matched caloric restriction Connexin 36 mediates blood cell flow in mouse pancreatic islets New insights into the role of connexins in pancreatic islet function and diabetes.The alpha-cell as target for type 2 diabetes therapy.Disordered insulin secretion in the development of insulin resistance and Type 2 diabetes.α-cell role in β-cell generation and regeneration.Glucose-induced inhibition of insulin secretion.Cellular communication and heterogeneity in pancreatic islet insulin secretion dynamics.The role of glucagon on type 2 diabetes at a glance.Inhibiting or antagonizing glucagon: making progress in diabetes care.Dimensionality and size scaling of coordinated Ca(2+) dynamics in MIN6 β-cell clusters.Fluorescence recovery after photobleaching reveals regulation and distribution of connexin36 gap junction coupling within mouse islets of Langerhans.Regulation of hepatic glucose metabolism in health and disease.Delta cell death in the islet of Langerhans and the progression from normal glucose tolerance to type 2 diabetes in non-human primates (baboon, Papio hamadryas).Role of reduced β-cell mass versus impaired β-cell function in the pathogenesis of type 2 diabetes.Stable and flexible system for glucose homeostasis.Endoscopic duodenal-jejunal bypass liner rapidly improves type 2 diabetes.A mathematical model of the impact of insulin secretion dynamics on selective hepatic insulin resistance.Correlated disorder in the Kuramoto model: Effects on phase coherence, finite-size scaling, and dynamic fluctuations.MafB is critical for glucagon production and secretion in mouse pancreatic α-cells in vivo.Impaired incretin effect and fasting hyperglucagonaemia characterizing type 2 diabetic subjects are early signs of dysmetabolism in obesity.Beta-cell hubs maintain Ca oscillations in human and mouse islet simulations

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Loss of inverse relationship between pulsatile insulin and glucagon secretion in patients with type 2 diabetes

http://www.wikidata.org/entity/Q35123524

description

2011 nî lūn-bûn

@nan

2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Loss of inverse relationship b ...... patients with type 2 diabetes

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Loss of inverse relationship b ...... patients with type 2 diabetes

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type

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Loss of inverse relationship b ...... patients with type 2 diabetes

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Loss of inverse relationship b ...... patients with type 2 diabetes

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Loss of inverse relationship b ...... patients with type 2 diabetes

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Loss of inverse relationship b ...... patients with type 2 diabetes

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Loss of inverse relationship b ...... patients with type 2 diabetes

@en

P2093

Björn A Menge

http://www.w3.org/2001/XMLSchema#string

Juris J Meier

http://www.w3.org/2001/XMLSchema#string

Lena Grüber

http://www.w3.org/2001/XMLSchema#string

Signe M Jørgensen

http://www.w3.org/2001/XMLSchema#string

Wolfgang E Schmidt

http://www.w3.org/2001/XMLSchema#string

P2860

Approximate entropy as a measure of system complexity

Beta-cell deficit and increased beta-cell apoptosis in humans with type 2 diabetes

Deconvolution analysis of hormone data.

Paracrinology of islets and the paracrinopathy of diabetes.

Effect of insulin-glucose infusions on plasma glucagon levels in fasting diabetics and nondiabetics

Pulsatile insulin secretion: detection, regulation, and role in diabetes.

Studies of pancreatic alpha cell function in normal and diabetic subjects.

Diminished glucagon suppression after β-cell reduction is due to impaired α-cell function rather than an expansion of α-cell mass.

Lilly lecture 1987. The triumvirate: beta-cell, muscle, liver. A collusion responsible for NIDDM.

Glucagon physiology and pathophysiology in the light of new advances.

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2160-2168

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scholarly article

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10.2337/DB11-0251

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8

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60

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Jens Juul Holst

Carolyn F. Deacon

Johannes D Veldhuis

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2011-06-15T00:00:00Z

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51223038

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21677283

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type-2 diabetes

P932

3142077

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