FGF-23/Klotho signaling is not essential for the phosphaturic and anabolic functions of PTH. - Wikidata - awgldk - TriplyDB

FGF-23/Klotho signaling is not essential for the phosphaturic and anabolic functions of PTH.

FGF-23/Klotho signaling is not essential for the phosphaturic and anabolic functions of PTH.

http://www.wikidata.org/entity/Q35129687

about

Regulation of serum phosphate Deletion of PTH rescues skeletal abnormalities and high osteopontin levels in Klotho-/- mice Fibroblast growth factor 23 enhances renal klotho abundance FGF23 deficiency leads to mixed hearing loss and middle ear malformation in mice.Fibroblast growth factor 23 and Klotho: physiology and pathophysiology of an endocrine network of mineral metabolism PTH ablation ameliorates the anomalies of Fgf23-deficient mice by suppressing the elevated vitamin D and calcium levels.Parathyroid hormone 1 receptor is essential to induce FGF23 production and maintain systemic mineral ion homeostasis.Fibroblast growth factor 23 and bone mineralisation.Insights from genetic disorders of phosphate homeostasis Fibroblast Growth Factor Receptor 3 Deficiency Does Not Impair the Osteoanabolic Action of Parathyroid Hormone on Mice.DNA N6-methyladenine demethylase ALKBH1 enhances osteogenic differentiation of human MSCs.Increased osteopontin contributes to inhibition of bone mineralization in FGF23-deficient mice.Neither absence nor excess of FGF23 disturbs murine fetal-placental phosphorus homeostasis or prenatal skeletal development and mineralization.The expanding family of hypophosphatemic syndromes.FGF23 and PTH--double agents at the heart of CKD.FGF-23 and secondary hyperparathyroidism in chronic kidney disease.Review of cinacalcet hydrochloride in the management of secondary hyperparathyroidism.Effects of klotho deletion from bone during chronic kidney disease.Arteriosclerosis, bone biology, and calciotropic hormone signaling: learning the ABCs of disease in the bone-vascular axis.The Association Between Fibroblast Growth Factor 23 and Renal Transplantation Outcome Is Modified by Follow-up Duration and Glomerular Filtration Rate Assessment Method.Pseudoxanthoma Elasticum-Like in β-Thalassemia Major, a matter of α-Klotho and Parathyroid Hormone?Fructus Ligustri Lucidi in Osteoporosis: A Review of its Pharmacology, Phytochemistry, Pharmacokinetics and Safety.Guanylate Binding Protein 1 Inhibits Osteogenic Differentiation of Human Mesenchymal Stromal Cells Derived from Bone Marrow.FGF23 Is Not Required to Regulate Fetal Phosphorus Metabolism but Exerts Effects Within 12 Hours After Birth.Acute Parathyroid Hormone Injection Increases C-Terminal but Not Intact Fibroblast Growth Factor 23 Levels.Bone quality changes associated with aging and disease: a review.Mettl3-mediated mA RNA methylation regulates the fate of bone marrow mesenchymal stem cells and osteoporosis

P2860

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P2860

FGF-23/Klotho signaling is not essential for the phosphaturic and anabolic functions of PTH.

http://www.wikidata.org/entity/Q35129687

description

2011 nî lūn-bûn

@nan

2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

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2011年論文

@zh-tw

2011年论文

@wuu

name

FGF-23/Klotho signaling is not ...... and anabolic functions of PTH.

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FGF-23/Klotho signaling is not ...... and anabolic functions of PTH.

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type

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FGF-23/Klotho signaling is not ...... and anabolic functions of PTH.

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FGF-23/Klotho signaling is not ...... and anabolic functions of PTH.

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prefLabel

FGF-23/Klotho signaling is not ...... and anabolic functions of PTH.

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FGF-23/Klotho signaling is not ...... and anabolic functions of PTH.

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P1433

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P1433

Journal of Bone and Mineral Research

P1476

FGF-23/Klotho signaling is not ...... and anabolic functions of PTH.

@en

P2093

Beate Lanske

http://www.w3.org/2001/XMLSchema#string

Christiane Schüler

http://www.w3.org/2001/XMLSchema#string

Hiroaki Saito

http://www.w3.org/2001/XMLSchema#string

Michael Densmore

http://www.w3.org/2001/XMLSchema#string

Quan Yuan

http://www.w3.org/2001/XMLSchema#string

Reinhold G Erben

http://www.w3.org/2001/XMLSchema#string

Tadatoshi Sato

http://www.w3.org/2001/XMLSchema#string

P2860

Overexpression of fibroblast growth factor 23 suppresses osteoblast differentiation and matrix mineralization in vitro

Critical role of activating transcription factor 4 in the anabolic actions of parathyroid hormone in bone

Human fibroblast growth factor-23 mutants suppress Na+-dependent phosphate co-transport activity and 1alpha,25-dihydroxyvitamin D3 production

Klotho converts canonical FGF receptor into a specific receptor for FGF23

Parathyroid hormone - a bone anabolic and catabolic agent

Genetic evidence of serum phosphate-independent functions of FGF-23 on bone

Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice

Regulation of intestinal Na+-dependent phosphate co-transporters by a low-phosphate diet and 1,25-dihydroxyvitamin D3

The parathyroid is a target organ for FGF23 in rats

PTH increases FGF23 gene expression and mediates the high-FGF23 levels of experimental kidney failure: a bone parathyroid feedback loop

P304

2026-2035

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scholarly article

P356

10.1002/JBMR.433

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9

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P478

26

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P577

2011-09-01T00:00:00Z

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51144895

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21590742

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P932

4343543

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