Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells.
about
Collision of Trapped Topoisomerase 2 with Transcription and Replication: Generation and Repair of DNA Double-Strand Breaks with 5' AdductsDNA damage tolerance by recombination: Molecular pathways and DNA structuresInhibition of Topoisomerase (DNA) I (TOP1): DNA Damage Repair and Anticancer TherapyOrigin-Dependent Inverted-Repeat Amplification: Tests of a Model for Inverted DNA AmplificationRPA and Rad51 constitute a cell intrinsic mechanism to protect the cytosol from self DNARad53-Mediated Regulation of Rrm3 and Pif1 DNA Helicases Contributes to Prevention of Aberrant Fork Transitions under Replication Stress.Replication stress: getting back on trackReplication fork instability and the consequences of fork collisions from rereplicationThe Causes and Consequences of Topological Stress during DNA ReplicationPreventing replication fork collapse to maintain genome integrityReplication stress in Mammalian cells and its consequences for mitosisA Dominant Mutation in Human RAD51 Reveals Its Function in DNA Interstrand Crosslink Repair Independent of Homologous RecombinationNUCKS1 is a novel RAD51AP1 paralog important for homologous recombination and genome stabilityUbiquitinated Fancd2 recruits Fan1 to stalled replication forks to prevent genome instabilityWerner syndrome: Clinical features, pathogenesis and potential therapeutic interventionsDifferential requirement of Srs2 helicase and Rad51 displacement activities in replication of hairpin-forming CAG/CTG repeats.Non-catalytic Roles for XPG with BRCA1 and BRCA2 in Homologous Recombination and Genome StabilityPoly(ADP-ribosyl) glycohydrolase prevents the accumulation of unusual replication structures during unperturbed S phaseDNA2 drives processing and restart of reversed replication forks in human cells.Regulation of recombination at yeast nuclear pores controls repair and triplet repeat stability.Pyrimidine Pool Disequilibrium Induced by a Cytidine Deaminase Deficiency Inhibits PARP-1 Activity, Leading to the Under Replication of DNA.HLTF's Ancient HIRAN Domain Binds 3' DNA Ends to Drive Replication Fork Reversal.Catalytic strand separation by RECQ1 is required for RPA-mediated response to replication stress.TRAIP is a PCNA-binding ubiquitin ligase that protects genome stability after replication stresshMSH5 Facilitates the Repair of Camptothecin-induced Double-strand Breaks through an Interaction with FANCJ.Chromosomal Integrity after UV Irradiation Requires FANCD2-Mediated Repair of Double Strand Breaks.Replication Restart after Replication-Transcription Conflicts Requires RecA in Bacillus subtilis.A short G1 phase imposes constitutive replication stress and fork remodelling in mouse embryonic stem cells.Triplex structures induce DNA double strand breaks via replication fork collapse in NER deficient cells.Folate levels modulate oncogene-induced replication stress and tumorigenicityMERIT40 cooperates with BRCA2 to resolve DNA interstrand cross-links.The MMS22L-TONSL heterodimer directly promotes RAD51-dependent recombination upon replication stress.NEK8 regulates DNA damage-induced RAD51 foci formation and replication fork protectionRad51 recombinase prevents Mre11 nuclease-dependent degradation and excessive PrimPol-mediated elongation of nascent DNA after UV irradiation.Mammalian RAD51 paralogs protect nascent DNA at stalled forks and mediate replication restart.A subset of cancer cell lines is acutely sensitive to the Chk1 inhibitor MK-8776 as monotherapy due to CDK2 activation in S phase.RECQL: a new breast cancer susceptibility gene.TRAIP regulates replication fork recovery and progression via PCNAKinase-dead ATM protein is highly oncogenic and can be preferentially targeted by Topo-isomerase I inhibitorsFANCJ is essential to maintain microsatellite structure genome-wide during replication stress.
P2860
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P2860
Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells.
description
2015 nî lūn-bûn
@nan
2015 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի մարտին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Rad51-mediated replication for ...... xic treatments in human cells.
@ast
Rad51-mediated replication for ...... xic treatments in human cells.
@en
type
label
Rad51-mediated replication for ...... xic treatments in human cells.
@ast
Rad51-mediated replication for ...... xic treatments in human cells.
@en
prefLabel
Rad51-mediated replication for ...... xic treatments in human cells.
@ast
Rad51-mediated replication for ...... xic treatments in human cells.
@en
P2093
P2860
P356
P1476
Rad51-mediated replication for ...... xic treatments in human cells.
@en
P2093
Alessandro Vindigni
Damian Dalcher
Jonas A Schmid
Karun Mutreja
Massimo Lopes
Matteo Berti
Ralph Zellweger
Raquel Herrador
P2860
P304
P356
10.1083/JCB.201406099
P407
P577
2015-03-01T00:00:00Z