about
The human subject: an integrative animal model for 21(st) century heart failure researchSex-based differences in cardiac ischaemic injury and protection: therapeutic implicationsEstrogen attenuates chronic volume overload induced structural and functional remodeling in male rat hearts.Endothelial dysfunction of resistance vessels in female apolipoprotein E-deficient mice.The effects of endogenous sex hormones and acute hypoxia on vasoconstriction in isolated rat pulmonary artery rings.Spontaneous heart disease in the adult chimpanzee (Pan troglodytes).Modulation of sarcoplasmic reticulum Ca(2+) cycling in systolic and diastolic heart failure associated with aging.17 Beta-estradiol differentially affects left ventricular and cardiomyocyte hypertrophy following myocardial infarction and pressure overloadAlpha-1-adrenergic receptors: targets for agonist drugs to treat heart failure.Direct vasoactive effects of the chromogranin A (CHGA) peptide catestatin in humans in vivo.Prior exercise training does not prevent acute cardiac alterations after myocardial infarction in female rats.Exercise training attenuates acute hyperalgesia in streptozotocin-induced diabetic female rats.Estrogen attenuates left ventricular and cardiomyocyte hypertrophy by an estrogen receptor-dependent pathway that increases calcineurin degradation.A decade of discoveries in cardiac biology.Revisiting Mars and Venus: understanding gender differences in critical illnessSex-defined T-cell responses to cardiac self determine differential outcomes of murine dilated cardiomyopathyTherapeutic implications of the gender-specific aspects of cardiovascular disease.Effects of injectable anesthetic combinations on left ventricular function and cardiac morphology in Sprague-Dawley ratsSystolic and diastolic heart failure: different phenotypes of the same disease?The Importance of Biological Sex and Estrogen in Rodent Models of Cardiovascular Health and DiseasePromoter methylation of Egr-1 site contributes to fetal hypoxia-mediated PKCε gene repression in the developing heartSex-specific pathways in early cardiac response to pressure overload in mice.Myocardial AKT: the omnipresent nexus.The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.Acute and Subchronic Toxic Effects of the Fruits of Physalis peruviana L.Gender differences in cardiac ischemic injury and protection--experimental aspects.Coronary artery disease in women: an unsolved dilemma.Glucocorticoid receptor alters isovolumetric contraction and restrains cardiac fibrosis.Pharmacogenomics, pharmacokinetics and pharmacodynamics: interaction with biological differences between men and women.Role of animal models in HCM research.Gender aspects in cardiovascular pharmacology.Cardiac ischaemic stress: cardiomyocyte Ca²⁺, sex and sex steroids.Ablation of cyclase-associated protein 2 (CAP2) leads to cardiomyopathy.Lifelong exposure to bisphenol a alters cardiac structure/function, protein expression, and DNA methylation in adult mice.Sex differences in cardiovascular function.Sex-specific differences in natriuretic peptide and nitric oxide synthase expression in ANP gene-disrupted mice.Sex-dependent impairment of cardiac action potential conduction in type 1 diabetic rats.Dietary isoflavones during pregnancy and lactation provide cardioprotection to offspring rats in adulthood.The intrinsic resistance of female hearts to an ischemic insult is abrogated in primary cardiac hypertrophy.Cardiac mass and cardiomyocyte size are governed by different genetic loci on either autosomes or chromosome Y in recombinant inbred mice.
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Sex is a potent modifier of the cardiovascular system
@ast
Sex is a potent modifier of the cardiovascular system
@en
type
label
Sex is a potent modifier of the cardiovascular system
@ast
Sex is a potent modifier of the cardiovascular system
@en
prefLabel
Sex is a potent modifier of the cardiovascular system
@ast
Sex is a potent modifier of the cardiovascular system
@en
P2860
P356
P1476
Sex is a potent modifier of the cardiovascular system
@en
P2860
P304
P356
10.1172/JCI200319429
P407
P577
2003-08-01T00:00:00Z