The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy. - Wikidata - awgldk - TriplyDB

The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

http://www.wikidata.org/entity/Q37239549

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The cell biology of disease: cellular mechanisms of cardiomyopathy.Cardiac Troponin and Tropomyosin: Structural and Cellular Perspectives to Unveil the Hypertrophic Cardiomyopathy Phenotype Distinct cardiac transcriptional profiles defining pregnancy and exercise Abnormal heart rate regulation in murine hearts with familial hypertrophic cardiomyopathy-related cardiac troponin T mutations Akt and MAPK signaling mediate pregnancy-induced cardiac adaptation.Dual specificity phosphatase 4 mediates cardiomyopathy caused by lamin A/C (LMNA) gene mutation A Tension-Based Model Distinguishes Hypertrophic versus Dilated Cardiomyopathy.A Review of the Molecular Mechanisms Underlying the Development and Progression of Cardiac Remodeling.Ectopic expression of Cdk8 induces eccentric hypertrophy and heart failure.

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The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

http://www.wikidata.org/entity/Q37239549

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 21 February 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

@en

The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

@nl

type

label

The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

@en

The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

@nl

prefLabel

The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

@en

The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

@nl

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J.YJMCC.2009.02.010

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Journal of Molecular and Cellular Cardiology

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The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy.

@en

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Anthony Rosenzweig

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Antke Messmer-Kratzsch

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Eric N Olson

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Jason Mansoori

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Stephen W Luckey

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Tyson Smyth

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P2860

Increased Ca2+-sensitivity of the contractile apparatus in end-stage human heart failure results from altered phosphorylation of contractile proteins

Hypertrophic cardiomyopathy: distribution of disease genes, spectrum of mutations, and implications for a molecular diagnosis strategy

Familial hypertrophic cardiomyopathy: from mutations to functional defects

Protooncogene induction and reprogramming of cardiac gene expression produced by pressure overload

Cardiac troponin I mutants. Phosphorylation by protein kinases C and A and regulation of Ca(2+)-stimulated MgATPase of reconstituted actomyosin S-1

Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCepsilon phosphorylation

A non-equilibrium isoelectric focusing method to determine states of phosphorylation of cardiac troponin I: identification of Ser-23 and Ser-24 as significant sites of phosphorylation by protein kinase C

A truncated cardiac troponin T molecule in transgenic mice suggests multiple cellular mechanisms for familial hypertrophic cardiomyopathy

Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo

Intracellular localization and functional effects of P21-activated kinase-1 (Pak1) in cardiac myocytes

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739-747

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scholarly article

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10.1016/J.YJMCC.2009.02.010

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Leslie Leinwand

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2009-02-21T00:00:00Z

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