Enhanced antiviral T cell function in the absence of B7-H1 is insufficient to prevent persistence but exacerbates axonal bystander damage during viral encephalomyelitis.
about
The importance of mouse models to define immunovirologic determinants of progressive multifocal leukoencephalopathyImmune Surveillance of the CNS following Infection and InjuryShifting hierarchies of interleukin-10-producing T cell populations in the central nervous system during acute and persistent viral encephalomyelitis.CXCR3-dependent plasma blast migration to the central nervous system during viral encephalomyelitis.CD4 T cells promote CD8 T cell immunity at the priming and effector site during viral encephalitisMyd88 Initiates Early Innate Immune Responses and Promotes CD4 T Cells during Coronavirus Encephalomyelitis.Granzyme B-inhibitor serpina3n induces neuroprotection in vitro and in vivo.Programmed death-1 controls T cell survival by regulating oxidative metabolism.Self-reactive CD4(+) T cells activated during viral-induced demyelination do not prevent clinical recoveryProgrammed death 1 protects from fatal circulatory failure during systemic virus infection of mice.Enhanced CD8 T-cell anti-viral function and clinical disease in B7-H1-deficient mice requires CD4 T cells during encephalomyelitis.Interleukin-10 is a critical regulator of white matter lesion containment following viral induced demyelinationAstrocyte-derived CXCL10 drives accumulation of antibody-secreting cells in the central nervous system during viral encephalomyelitis.CXCL13 promotes isotype-switched B cell accumulation to the central nervous system during viral encephalomyelitis.IL-21 optimizes T cell and humoral responses in the central nervous system during viral encephalitis.MMP-independent role of TIMP-1 at the blood brain barrier during viral encephalomyelitis.Role of CD25(+) CD4(+) T cells in acute and persistent coronavirus infection of the central nervous system.Intrathecal humoral immunity to encephalitic RNA viruses.Maintenance of PD-1 on brain-resident memory CD8 T cells is antigen independent.Protective Humoral Immunity in the CNS Requires Peripheral CD19-Dependent Germinal Center Formation Following Coronavirus Encephalomyelitis.Niches for the Long-Term Maintenance of Tissue-Resident Memory T Cells.
P2860
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P2860
Enhanced antiviral T cell function in the absence of B7-H1 is insufficient to prevent persistence but exacerbates axonal bystander damage during viral encephalomyelitis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Enhanced antiviral T cell func ...... uring viral encephalomyelitis.
@ast
Enhanced antiviral T cell func ...... uring viral encephalomyelitis.
@en
type
label
Enhanced antiviral T cell func ...... uring viral encephalomyelitis.
@ast
Enhanced antiviral T cell func ...... uring viral encephalomyelitis.
@en
prefLabel
Enhanced antiviral T cell func ...... uring viral encephalomyelitis.
@ast
Enhanced antiviral T cell func ...... uring viral encephalomyelitis.
@en
P2093
P2860
P356
P1476
Enhanced antiviral T cell func ...... during viral encephalomyelitis
@en
P2093
David R Hinton
Roscoe Atkinson
Stephen A Stohlman
Timothy W Phares
P2860
P304
P356
10.4049/JIMMUNOL.1001984
P407
P577
2010-09-27T00:00:00Z