MyD88 and Trif signaling play distinct roles in cardiac dysfunction and mortality during endotoxin shock and polymicrobial sepsis.
about
Cardiotoxicity during invasive pneumococcal diseaseMyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory responseToll-like receptor 4 is essential to preserving cardiac function and survival in low-grade polymicrobial sepsis.Role of cardiac- and myeloid-MyD88 signaling in endotoxin shock: a study with tissue-specific deletion models.Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetesToll-like receptors and myocardial inflammation.Attenuation of Cardiac Dysfunction in Polymicrobial Sepsis by MicroRNA-146a Is Mediated via Targeting of IRAK1 and TRAF6 Expression.Cardiac RNA induces inflammatory responses in cardiomyocytes and immune cells via Toll-like receptor 7 signalingOverexpression of myeloid differentiation protein 88 in mice induces mild cardiac dysfunction, but no deficit in heart morphologyB and T lymphocyte attenuator inhibits LPS-induced endotoxic shock by suppressing Toll-like receptor 4 signaling in innate immune cells.In vivo TLR9 inhibition attenuates CpG-induced myocardial dysfunction.The role of myeloid differentiation factor 88 on mitochondrial dysfunction of peritoneal leukocytes during polymicrobial sepsisComplement factor B is the downstream effector of TLRs and plays an important role in a mouse model of severe sepsis.PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsisIntravenous anesthetic propofol binds to 5-lipoxygenase and attenuates leukotriene B4 production.Pleiotropic regulations of neutrophil receptors response to sepsis.Enhanced Glycolytic Metabolism Contributes to Cardiac Dysfunction in Polymicrobial Sepsis.MicroRNA-125b Prevents Cardiac Dysfunction in Polymicrobial Sepsis by Targeting TRAF6-Mediated Nuclear Factor κB Activation and p53-Mediated Apoptotic Signaling.From the Cover: Prolonged Exposure to Volatile Anesthetic Isoflurane Worsens the Outcome of Polymicrobial Abdominal Sepsis.α2A-adrenergic blockade attenuates septic cardiomyopathy by increasing cardiac norepinephrine concentration and inhibiting cardiac endothelial activation.
P2860
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P2860
MyD88 and Trif signaling play distinct roles in cardiac dysfunction and mortality during endotoxin shock and polymicrobial sepsis.
description
2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
MyD88 and Trif signaling play ...... hock and polymicrobial sepsis.
@ast
MyD88 and Trif signaling play ...... hock and polymicrobial sepsis.
@en
type
label
MyD88 and Trif signaling play ...... hock and polymicrobial sepsis.
@ast
MyD88 and Trif signaling play ...... hock and polymicrobial sepsis.
@en
prefLabel
MyD88 and Trif signaling play ...... hock and polymicrobial sepsis.
@ast
MyD88 and Trif signaling play ...... hock and polymicrobial sepsis.
@en
P2093
P2860
P1433
P1476
MyD88 and Trif signaling play ...... shock and polymicrobial sepsis
@en
P2093
P2860
P304
P356
10.1097/ALN.0B013E31822A22F7
P407
P50
P577
2011-09-01T00:00:00Z