about
Platelet-activating factor: receptors and signal transductionWNT1-inducible signaling pathway protein 1 contributes to ventilator-induced lung injuryVisnagin protects against doxorubicin-induced cardiomyopathy through modulation of mitochondrial malate dehydrogenase.Synergistic inflammation is induced by blood degradation products with microbial Toll-like receptor agonists and is blocked by hemopexin.Toll-like receptor 4 is essential to preserving cardiac function and survival in low-grade polymicrobial sepsis.Role of cardiac- and myeloid-MyD88 signaling in endotoxin shock: a study with tissue-specific deletion models.Importance of Toll-like receptor 2 in mitochondrial dysfunction during polymicrobial sepsisToll-like receptor 4 signaling confers cardiac protection against ischemic injury via inducible nitric oxide synthase- and soluble guanylate cyclase-dependent mechanisms.Validation of noninvasive measurements of cardiac output in mice using echocardiography.MyD88 and Trif signaling play distinct roles in cardiac dysfunction and mortality during endotoxin shock and polymicrobial sepsis.Differential effects of etomidate and its pyrrole analogue carboetomidate on the adrenocortical and cytokine responses to endotoxemia.Functional brown adipose tissue limits cardiomyocyte injury and adverse remodeling in catecholamine-induced cardiomyopathy.Inhibitory effect of porcine surfactant on the respiratory burst oxidase in human neutrophils. Attenuation of p47phox and p67phox membrane translocation as the mechanism.Evidence for platelet-activating factor as a late-phase mediator of chronic pancreatitis in the ratCardiac RNA induces inflammatory responses in cardiomyocytes and immune cells via Toll-like receptor 7 signalingComplement Factor B Production in Renal Tubular Cells and Its Role in Sodium Transporter Expression During Polymicrobial Sepsis.Toll-like receptor 4-myeloid differentiation factor 88 signaling contributes to ventilator-induced lung injury in miceToll-like receptor signaling: a critical modulator of cell survival and ischemic injury in the heartThe role of myeloid differentiation factor 88 on mitochondrial dysfunction of peritoneal leukocytes during polymicrobial sepsisComplement factor B is the downstream effector of TLRs and plays an important role in a mouse model of severe sepsis.Toll-like receptor 2 plays a critical role in cardiac dysfunction during polymicrobial sepsis.Theranostic Nucleic Acid Binding Nanoprobe Exerts Anti-inflammatory and Cytoprotective Effects in Ischemic Injury.Extracellular MicroRNAs Induce Potent Innate Immune Responses via TLR7/MyD88-Dependent Mechanisms.Fas-associated death-domain protein inhibits TNF-alpha mediated NF-kappaB activation in cardiomyocytes.Reduced Expression of SARM in Mouse Spleen during Polymicrobial Sepsis.Splenic RNA and MicroRNA Mimics Promote Complement Factor B Production and Alternative Pathway Activation via Innate Immune Signaling.Imaging Lymphoid Cell Death In Vivo During Polymicrobial Sepsis.Cyclopropyl-methoxycarbonyl Metomidate: Studies in a Lipopolysaccharide Inflammatory Model of Sepsis.18F-FDG kinetics parameters depend on the mechanism of injury in early experimental acute respiratory distress syndrome.Platelet-activating factor-stimulated protein tyrosine phosphorylation and eicosanoid synthesis in rat Kupffer cells. Evidence for calcium-dependent and protein kinase C-dependent and -independent pathways.Strategic advantages of insulin-like growth factor-I expression for cardioprotection.Inhibition of the human neutrophil respiratory burst by native and synthetic surfactant.Lipopeptide Pam3Cys4 Synergizes N-Formyl-Met-Leu-Phe (Fmlp)-Induced Calcium Transients in Mouse Neutrophils.Importance of the Complement Alternative Pathway in Serum Chemotactic Activity During Sepsis.MyD88 and NOS2 are essential for toll-like receptor 4-mediated survival effect in cardiomyocytes.Circulating Plasma Extracellular Vesicles from Septic Mice Induce Inflammation via MicroRNA- and TLR7-Dependent MechanismsRole of platelet-activating factor in hepatic responses after bile duct ligation in ratsRegulation of platelet-activating factor receptor and PAF receptor-mediated arachidonic acid release by protein kinase C activation in rat Kupffer cellsIdentification of receptors for platelet-activating factor in rat Kupffer cellsRegulation of platelet-activating factor receptors in rat Kupffer cells
P50
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P50
description
researcher ORCID ID = 0000-0002-2505-1360
@en
name
Wei Chao
@ast
Wei Chao
@en
Wei Chao
@es
Wei Chao
@nl
type
label
Wei Chao
@ast
Wei Chao
@en
Wei Chao
@es
Wei Chao
@nl
prefLabel
Wei Chao
@ast
Wei Chao
@en
Wei Chao
@es
Wei Chao
@nl
P31
P496
0000-0002-2505-1360