Investigating hypoxic tumor physiology through gene expression patterns. - Wikidata - awgldk - TriplyDB

Investigating hypoxic tumor physiology through gene expression patterns.

Investigating hypoxic tumor physiology through gene expression patterns.

http://www.wikidata.org/entity/Q35208837

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Inactivation of the CYLD deubiquitinase by HPV E6 mediates hypoxia-induced NF-kappaB activation Hypoxia-induced lysyl oxidase is a critical mediator of bone marrow cell recruitment to form the premetastatic niche Gene expression programs in response to hypoxia: cell type specificity and prognostic significance in human cancers Hypoxic stress suppresses RNA polymerase III recruitment and tRNA gene transcription in cardiomyocytes.Targeting Tumor Metabolism for Cancer Treatment: Is Pyruvate Dehydrogenase Kinases (PDKs) a Viable Anticancer Target?HypoxamiRs and cancer: from biology to targeted therapy Targeting microenvironment in cancer therapeutics The lysyl oxidase inhibitor, beta-aminopropionitrile, diminishes the metastatic colonization potential of circulating breast cancer cells The l1-l2 regularization framework unmasks the hypoxia signature hidden in the transcriptome of a set of heterogeneous neuroblastoma cell lines.Hypoxia and TGF-β1 induced PLOD2 expression improve the migration and invasion of cervical cancer cells by promoting epithelial-to-mesenchymal transition (EMT) and focal adhesion formation In vivo profiling of hypoxic gene expression in gliomas using the hypoxia marker EF5 and laser-capture microdissection.Hypoxia modulates EWS-FLI1 transcriptional signature and enhances the malignant properties of Ewing's sarcoma cells in vitro.Serum signature of hypoxia-regulated factors is associated with progression after induction therapy in head and neck squamous cell cancer.Global gene expression profiling in three tumor cell lines subjected to experimental cycling and chronic hypoxia.COPing with hypoxia The emerging role of the COP9 signalosome in cancer.Report from the Radiation Therapy Committee of the Southwest Oncology Group (SWOG): Research Objectives Workshop 2008.Hypoxia induces adhesion molecules on cancer cells: A missing link between Warburg effect and induction of selectin-ligand carbohydrates.Complement anaphylatoxins as immune regulators in cancer.Ionizing radiation induces tumor cell lysyl oxidase secretion.The p53 tumor suppressor network is a key responder to microenvironmental components of chronic inflammatory stress.Extracellular matrix genes as hypoxia-inducible targets.HO-3867, a safe STAT3 inhibitor, is selectively cytotoxic to ovarian cancer.Functional erythropoietin autocrine loop in melanoma Aldose reductase inhibition prevents hypoxia-induced increase in hypoxia-inducible factor-1alpha (HIF-1alpha) and vascular endothelial growth factor (VEGF) by regulating 26 S proteasome-mediated protein degradation in human colon cancer cells Regional hypoxia in glioblastoma multiforme quantified with [18F]fluoromisonidazole positron emission tomography before radiotherapy: correlation with time to progression and survival Gene expression and hypoxia in breast cancer.Characterization of a Gene Expression Signature in Normal Rat Prostate Tissue Induced by the Presence of a Tumor Elsewhere in the Organ.In vivo topoisomerase I inhibition attenuates the expression of hypoxia-inducible factor 1α target genes and decreases tumor angiogenesis.Cancer cell: using inflammation to invade the host.High Lysyl Oxidase (LOX) in the Non-Malignant Prostate Epithelium Predicts a Poor Outcome in Prostate Cancer Patient Managed by Watchful Waiting.Metabolic targeting of hypoxia and HIF1 in solid tumors can enhance cytotoxic chemotherapy.Hypoxia-induced autophagy mediates cisplatin resistance in lung cancer cells.Hypoxia-inducible miR-182 enhances HIF1α signaling via targeting PHD2 and FIH1 in prostate cancer.miR-210: fine-tuning the hypoxic response.The role of ascorbate in the modulation of HIF-1alpha protein and HIF-dependent transcription by chromium(VI) and nickel(II)Fibroblasts--a key host cell type in tumor initiation, progression, and metastasis.Induction of gastrin expression in gastrointestinal cells by hypoxia or cobalt is independent of hypoxia-inducible factor (HIF).In Vivo Interrogation of the Hypoxic Transcriptome of Solid Tumors: Optimizing Hypoxic Probe Labeling with Laser Capture Microdissection for Isolation of High-Quality RNA for Deep Sequencing Analysis.Lysyl oxidase-like-1 enhances lung metastasis when lactate accumulation and monocarboxylate transporter expression are involved

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Investigating hypoxic tumor physiology through gene expression patterns.

http://www.wikidata.org/entity/Q35208837

description

2003 nî lūn-bûn

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2003 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2003 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

Investigating hypoxic tumor physiology through gene expression patterns.

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Investigating hypoxic tumor physiology through gene expression patterns.

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type

label

Investigating hypoxic tumor physiology through gene expression patterns.

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Investigating hypoxic tumor physiology through gene expression patterns.

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prefLabel

Investigating hypoxic tumor physiology through gene expression patterns.

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Investigating hypoxic tumor physiology through gene expression patterns.

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Investigating hypoxic tumor physiology through gene expression patterns.

@en

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Amato J Giaccia

http://www.w3.org/2001/XMLSchema#string

Karen M Hudson

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Lucrezia A Fontana

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Nicholas C Denko

http://www.w3.org/2001/XMLSchema#string

Patrick D Sutphin

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Soumya Raychaudhuri

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P2860

General involvement of hypoxia-inducible factor 1 in transcriptional response to hypoxia

Tumor hypoxia: definitions and current clinical, biologic, and molecular aspects

Hypoxia actively represses transcription by inducing negative cofactor 2 (Dr1/DrAP1) and blocking preinitiation complex assembly

Dimerization, DNA binding, and transactivation properties of hypoxia-inducible factor 1

Nitric oxide regulates the expression of vasoconstrictors and growth factors by vascular endothelium under both normoxia and hypoxia.

Functional versatility in the CRP-FNR superfamily of transcription factors: FNR and FLP.

Endothelial response to hypoxia: physiologic adaptation and pathologic dysfunction.

Tissue factor transcription driven by Egr-1 is a critical mechanism of murine pulmonary fibrin deposition in hypoxia.

Hypoxia stimulates insulin-like growth factor binding protein 1 (IGFBP-1) gene expression in HepG2 cells: a possible model for IGFBP-1 expression in fetal hypoxia.

Hypoxia-inducible factor-1 modulates gene expression in solid tumors and influences both angiogenesis and tumor growth.

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5907-5914

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scholarly article

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10.1038/SJ.ONC.1206703

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37

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22

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2003-09-01T00:00:00Z

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10590646

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12947397

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