Retinoic acid is essential for Th1 cell lineage stability and prevents transition to a Th17 cell program. - Wikidata - awgldk - TriplyDB

Retinoic acid is essential for Th1 cell lineage stability and prevents transition to a Th17 cell program.

Retinoic acid is essential for Th1 cell lineage stability and prevents transition to a Th17 cell program.

http://www.wikidata.org/entity/Q35213499

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Retinoic Acid as a Modulator of T Cell Immunity Breast Milk and Solid Food Shaping Intestinal Immunity Seeing through the dark: New insights into the immune regulatory functions of vitamin A Differentiated all-trans retinoic acid response of naive CD4+CD25- cells isolated from rats with collagen-induced arthritis and healthy ones under in vitro conditions.Contraction of intestinal effector T cells by retinoic acid-induced purinergic receptor P2X7 Pathogenesis of acute graft-versus-host disease: from intestinal microbiota alterations to donor T cell activation.Treatment with retinoid X receptor agonist IRX4204 ameliorates experimental autoimmune encephalomyelitis.Radiographic damage in hands and wrists of patients with juvenile idiopathic arthritis after 29 years of disease duration.Influence of nutrient-derived metabolites on lymphocyte immunity.Differentiation and function of group 3 innate lymphoid cells, from embryo to adult.Early life factors that affect allergy development.Ubiquitin Ligases and Deubiquitinating Enzymes in CD4+ T Cell Effector Fate Choice and Function.Parasites, nutrition, immune responses and biology of metabolic tissues.Recent advances in gut immunology.Immunomodulation of autoimmune arthritis by pro-inflammatory cytokines.Retinoic acid-primed human dendritic cells inhibit Th9 cells and induce Th1/Th17 cell differentiation.Beta 1-integrin ligation and TLR ligation enhance GM-CSF-induced ALDH1A2 expression in dendritic cells, but differentially regulate their anti-inflammatory properties.Low plasma vitamin A concentration is associated with tuberculosis in Moroccan population: a preliminary case control study.Impact of Vitamin A and Carotenoids on the Risk of Tuberculosis Progression.The transcriptional repressor HIC1 regulates intestinal immune homeostasis.Reverse plasticity: TGF-β and IL-6 induce Th1-to-Th17-cell transdifferentiation in the gut.IRF8-dependent molecular complexes control the Th9 transcriptional program.VISTA Deficiency Accelerates the Development of Fatal Murine Lupus Nephritis.Intestinal epithelial cell-specific RARα depletion results in aberrant epithelial cell homeostasis and underdeveloped immune system.Mechanism underlying the suppressor activity of retinoic acid on IL4-induced IgE synthesis and its physiological implication.T-bet over-expression regulates aryl hydrocarbon receptor-mediated T helper type 17 differentiation through an interferon (IFN)γ-independent pathway.BJ-2266 ameliorates experimental autoimmune encephalomyelitis through down-regulation of the JAK/STAT signaling pathway.The transdifferentiation of regulatory T and Th17 cells in autoimmune/inflammatory diseases and its potential implications in pregnancy complications.Topical treatment of all-trans retinoic acid inhibits murine melanoma partly by promoting CD8+ T-cell immunity.Mechanistic Differences in Neuropathic Pain Modalities Revealed by Correlating Behavior with Global Expression Profiling.Elevated Levels of T-helper 17-associated Cytokines in Diabetes Type I Patients: Indicators for Following the Course of Disease.CD45 regulates GM-CSF, retinoic acid and T-cell homing in intestinal inflammation.Oral tolerance is inefficient in neonatal mice due to a physiological vitamin A deficiency.Control of Innate and Adaptive Lymphocytes by the RAR-Retinoic Acid Axis.Retinoic acid elicits a coordinated expression of gut homing markers on T lymphocytes of Zambian men receiving oral Vivotif, but not Rotarix, Dukoral or OPVERO vaccines.Impact of Retinoic Acid on Immune Cells and Inflammatory Diseases Retinoic Acid, Leaky Gut, and Autoimmune Diseases

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Retinoic acid is essential for Th1 cell lineage stability and prevents transition to a Th17 cell program.

http://www.wikidata.org/entity/Q35213499

description

2015 nî lūn-bûn

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2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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Retinoic acid is essential for ...... sition to a Th17 cell program.

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J.IMMUNI.2015.02.003

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Retinoic acid is essential for ...... sition to a Th17 cell program.

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Aurelien Sarde

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Carla Ortiz

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Chrysothemis C Brown

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Daniel Mucida

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Daria Esterhazy

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Emanuele de Rinaldis

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Ines Osma-Garcia

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Mariya London

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Matthew Arno

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Randolph J Noelle

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P2860

Transcription factor IRF8 directs a silencing programme for TH17 cell differentiation

Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci

The AP-1 transcription factor Batf controls T(H)17 differentiation

A CBP integrator complex mediates transcriptional activation and AP-1 inhibition by nuclear receptors

A validated regulatory network for Th17 cell specification

A unique chromatin signature uncovers early developmental enhancers in humans

IL-6 programs T(H)-17 cell differentiation by promoting sequential engagement of the IL-21 and IL-23 pathways

Phenotypic and functional features of human Th17 cells

Reciprocal TH17 and regulatory T cell differentiation mediated by retinoic acid

Retinoic acid and histone deacetylases regulate epigenetic changes in embryonic stem cells

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499-511

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10.1016/J.IMMUNI.2015.02.003

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