Pretreatment with angiotensin-converting enzyme inhibitor improves doxorubicin-induced cardiomyopathy via preservation of mitochondrial function.
about
The tell-tale heart: molecular and cellular responses to childhood anthracycline exposureVisnagin protects against doxorubicin-induced cardiomyopathy through modulation of mitochondrial malate dehydrogenase.Diazoxide protects against doxorubicin-induced cardiotoxicity in the rat.Reversible blockade of complex I or inhibition of PKCβ reduces activation and mitochondria translocation of p66Shc to preserve cardiac function after ischemiaThe role of renin angiotensin system antagonists in the prevention of doxorubicin and trastuzumab induced cardiotoxicityPotent Paracrine Effects of human induced Pluripotent Stem Cell-derived Mesenchymal Stem Cells Attenuate Doxorubicin-induced CardiomyopathyMetabolic and anthropometric changes in early breast cancer patients receiving adjuvant therapy.Cancer Therapy-Related Cardiac Dysfunction and Heart Failure: Part 2: Prevention, Treatment, Guidelines, and Future Directions.Grape-Derived Polyphenols Prevent Doxorubicin-Induced Blunted EDH-Mediated Relaxations in the Rat Mesenteric Artery: Role of ROS and Angiotensin IIImpaired cardiac and skeletal muscle bioenergetics in children, adolescents, and young adults with Barth syndrome.Brain-derived neurotrophic factor attenuates doxorubicin-induced cardiac dysfunction through activating Akt signalling in rats.Importance of metabolic changes induced by chemotherapy on prognosis of early-stage breast cancer patients: a review of potential mechanisms.Use of angiotensin-converting enzyme inhibitors in the prophylaxis of anthracycline or trastuzumab-related cardiac dysfunction: preclinical and clinical considerations.Cardio-oncology: cardiovascular complications of cancer therapy.All-trans-retinoic acid ameliorates doxorubicin-induced cardiotoxicity: in vivo potential involvement of oxidative stress, inflammation, and apoptosis via caspase-3 and p53 down-expression.The restoration of kidney mitochondria function by inhibition of angiotensin-II production in rats with acute adriamycin-induced nephrotoxicity.Oral administration of Ginsenoside Rg1 prevents cardiac toxicity induced by doxorubicin in mice through anti-apoptosis.Diverging effects of enalapril or eplerenone in primary prevention against doxorubicin-induced cardiotoxicity.
P2860
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P2860
Pretreatment with angiotensin-converting enzyme inhibitor improves doxorubicin-induced cardiomyopathy via preservation of mitochondrial function.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Pretreatment with angiotensin- ...... ion of mitochondrial function.
@ast
Pretreatment with angiotensin- ...... ion of mitochondrial function.
@en
type
label
Pretreatment with angiotensin- ...... ion of mitochondrial function.
@ast
Pretreatment with angiotensin- ...... ion of mitochondrial function.
@en
prefLabel
Pretreatment with angiotensin- ...... ion of mitochondrial function.
@ast
Pretreatment with angiotensin- ...... ion of mitochondrial function.
@en
P2093
P2860
P1476
Pretreatment with angiotensin- ...... ion of mitochondrial function.
@en
P2093
Andrew J Connolly
Andrew Stephen Lee
Asimina Hiona
Jayan Nagendran
Xiaoyan Xie
P2860
P304
396-403.e3
P356
10.1016/J.JTCVS.2010.07.097
P407
P577
2010-11-20T00:00:00Z