Oral administration of Ginsenoside Rg1 prevents cardiac toxicity induced by doxorubicin in mice through anti-apoptosis.

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Oral administration of Ginsenoside Rg1 prevents cardiac toxicity induced by doxorubicin in mice through anti-apoptosis.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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Oral administration of Ginseno ...... n mice through anti-apoptosis.

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Oral administration of Ginseno ...... n mice through anti-apoptosis.

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Oral administration of Ginseno ...... n mice through anti-apoptosis.

@en

Oral administration of Ginseno ...... n mice through anti-apoptosis.

@nl

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Oral administration of Ginseno ...... n mice through anti-apoptosis.

@en

Oral administration of Ginseno ...... n mice through anti-apoptosis.

@nl

P1476

Oral administration of Ginseno ...... n mice through anti-apoptosis.

@en

P2093

Chen Zhu

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Haiman Mu

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Hua Liu

http://www.w3.org/2001/XMLSchema#string

Jianhua Huang

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Jiayi Zhang

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Yi Wang

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Yue Lu

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P2860

Identification of the molecular basis of doxorubicin-induced cardiotoxicity.

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Estrogen-like activity of ginsenoside Rg1 derived from Panax notoginseng.

Protecting against anthracycline-induced myocardial damage: a review of the most promising strategies.

Doxorubicin pathways: pharmacodynamics and adverse effects.

Pretreatment with angiotensin-converting enzyme inhibitor improves doxorubicin-induced cardiomyopathy via preservation of mitochondrial function.

Cannabidiol Protects against Doxorubicin-Induced Cardiomyopathy by Modulating Mitochondrial Function and Biogenesis.

Combined Salvianolic Acid B and Ginsenoside Rg1 Exerts Cardioprotection against Ischemia/Reperfusion Injury in Rats.

Molecular determinants of the physiological adaptation to stress in the cardiomyocyte: a focus on AKT.

Curcumin Downregulates Phosphate Carrier and Protects against Doxorubicin Induced Cardiomyocyte Apoptosis.

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83792-83801

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scholarly article

P356

10.18632/ONCOTARGET.19698

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P407

English

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P478

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P577

2017-07-31T00:00:00Z

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29137383

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P932

5663555

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