NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking. - Wikidata - awgldk - TriplyDB

NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.

NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.

http://www.wikidata.org/entity/Q35224505

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Dynorphin Controls the Gain of an Amygdalar Anxiety Circuit Optogenetic approaches for dissecting neuromodulation and GPCR signaling in neural circuits Commonalities and Distinctions Among Mechanisms of Addiction to Alcohol and Other Drugs Acute engagement of Gq-mediated signaling in the bed nucleus of the stria terminalis induces anxiety-like behavior.Functional Heterogeneity in the Bed Nucleus of the Stria Terminalis.Mad men, women and steroid cocktails: a review of the impact of sex and other factors on anabolic androgenic steroids effects on affective behaviors Stress Modulation of Opposing Circuits in the Bed Nucleus of the Stria Terminalis.Effects of chronic ethanol exposure on neuronal function in the prefrontal cortex and extended amygdala.Npy deletion in an alcohol non-preferring rat model elicits differential effects on alcohol consumption and body weight.Effects of Intermittent Alcohol Exposure on Emotion and Cognition: A Potential Role for the Endogenous Cannabinoid System and Neuroinflammation.Defining the role of corticotropin releasing factor binding protein in alcohol consumption.Diversity of Reporter Expression Patterns in Transgenic Mouse Lines Targeting Corticotropin-Releasing Hormone-Expressing Neurons A comparative analysis of the physiological properties of neurons in the anterolateral bed nucleus of the stria terminalis in the Mus musculus, Rattus norvegicus, and Macaca mulatta.IL-1 receptor signaling in the basolateral amygdala modulates binge-like ethanol consumption in male C57BL/6J mice.A Transgenic Rat for Investigating the Anatomy and Function of Corticotrophin Releasing Factor Circuits DREADDs for Neuroscientists.Neuropeptide Y response to alcohol is altered in nucleus accumbens of mice selectively bred for drinking to intoxication DREADDS: Use and application in behavioral neuroscience Resolving Behavioral Output via Chemogenetic Designer Receptors Exclusively Activated by Designer Drugs Control of chronic excessive alcohol drinking by genetic manipulation of the Edinger-Westphal nucleus urocortin-1 neuropeptide system.Neuropeptide Y in Alcohol Addiction and Affective Disorders.Substance use modulates stress reactivity: Behavioral and physiological outcomes.The CRF family of neuropeptides and their receptors - mediators of the central stress response.The phosphodiesterase-4 inhibitor roflumilast decreases ethanol consumption in C57BL/6J mice.Glial and Neuroimmune Mechanisms as Critical Modulators of Drug Use and Abuse.Influence of stress associated with chronic alcohol exposure on drinking.Epigenetic mechanisms of alcoholism and stress-related disorders.Altered responsiveness of BNST and amygdala neurons in trauma-induced anxiety.Extended Amygdala to Ventral Tegmental Area Corticotropin-Releasing Factor Circuit Controls Binge Ethanol Intake.Overexpressing Corticotropin-Releasing Factor in the Primate Amygdala Increases Anxious Temperament and Alters Its Neural Circuit Dissection of the Drosophila neuropeptide F circuit using a high-throughput two-choice assay.Effects of chronic alcohol consumption on neuronal function in the non-human primate BNST.Neuropeptide Y Regulates Sleep by Modulating Noradrenergic Signaling.Corticotropin Releasing Factor in the Bed Nucleus of the Stria Terminalis in Socially Defeated and Non-stressed Mice with a History of Chronic Alcohol Intake.Circuit and synaptic mechanisms of repeated stress: Perspectives from differing contexts, duration, and development.Drosophila: An Emergent Model for Delineating Interactions between the Circadian Clock and Drugs of Abuse.Voluntary Binge-like Ethanol Consumption Site-specifically Increases c-Fos Immunoexpression in Male C57BL6/J Mice.Corticotropin-Releasing Factor (CRF) Neurocircuitry and Neuropharmacology in Alcohol Drinking.The Corticotropin Releasing Factor Receptor 1 in Alcohol Use Disorder: Still a Valid Drug Target?Distribution of corticotropin-releasing factor neurons in the mouse brain: a study using corticotropin-releasing factor-modified yellow fluorescent protein knock-in mouse.

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P2860

NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.

http://www.wikidata.org/entity/Q35224505

description

2015 nî lūn-bûn

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2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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name

NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.

@ast

NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.

@en

type

label

NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.

@ast

NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.

@en

prefLabel

NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.

@ast

NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.

@en

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Nature Neuroscience

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NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking

@en

P2093

Alexis M Kendra

http://www.w3.org/2001/XMLSchema#string

Bradford B Lowell

http://www.w3.org/2001/XMLSchema#string

Christopher M Mazzone

http://www.w3.org/2001/XMLSchema#string

David P Olson

http://www.w3.org/2001/XMLSchema#string

Emily G Lowery-Gionta

http://www.w3.org/2001/XMLSchema#string

Kathleen A Grant

http://www.w3.org/2001/XMLSchema#string

Kristen E Pleil

http://www.w3.org/2001/XMLSchema#string

Todd E Thiele

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P2860

A role for brain stress systems in addiction.

The NPY system in stress, anxiety and depression

Single-nucleotide polymorphisms in corticotropin releasing hormone receptor 1 gene (CRHR1) are associated with quantitative trait of event-related potential and alcohol dependence

Modulation of glutamatergic synaptic transmission in the bed nucleus of the stria terminalis.

Drinking typography established by scheduled induction predicts chronic heavy drinking in a monkey model of ethanol self-administration

Experience-dependent modification of a central amygdala fear circuit.

Cloned neuropeptide Y (NPY) Y1 and pancreatic polypeptide Y4 receptors expressed in Chinese hamster ovary cells show considerable agonist-driven internalization, in contrast to the NPY Y2 receptor.

Neuropeptide Y signaling in the central nucleus of amygdala regulates alcohol-drinking and anxiety-like behaviors of alcohol-preferring rats.

Corticotropin releasing factor-induced amygdala gamma-aminobutyric Acid release plays a key role in alcohol dependence.

"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake

P2888

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545-552

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10.1038/NN.3972

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P433

4

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18

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Jennifer A Rinker

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2015-03-09T00:00:00Z

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25751534

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4376619

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