Collecting duct-specific gene inactivation of alphaENaC in the mouse kidney does not impair sodium and potassium balance. - Wikidata - awgldk - TriplyDB

Collecting duct-specific gene inactivation of alphaENaC in the mouse kidney does not impair sodium and potassium balance.

Collecting duct-specific gene inactivation of alphaENaC in the mouse kidney does not impair sodium and potassium balance.

http://www.wikidata.org/entity/Q35242835

about

Ubiquitylation and control of renal Na+ balance and blood pressure The role of transforming growth factor β1 in the regulation of blood pressure Arachidonic acid monooxygenase: Genetic and biochemical approaches to physiological/pathophysiological relevance Collecting duct-specific knockout of endothelin-1 causes hypertension and sodium retention Long-term aldosterone treatment induces decreased apical but increased basolateral expression of AQP2 in CCD of rat kidney Benzamil sensitive ion channels contribute to volume regulation in canine chondrocytes Deletion of hensin/DMBT1 blocks conversion of beta- to alpha-intercalated cells and induces distal renal tubular acidosis.Genetics of salt-sensitive hypertension.Mice heterozygous for beta-ENaC deletion have defective potassium excretion.Proteomic profiling of nuclei from native renal inner medullary collecting duct cells using LC-MS/MS Sodium and potassium balance depends on αENaC expression in connecting tubule alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.Colon-specific deletion of epithelial sodium channel causes sodium loss and aldosterone resistance The Na+-dependent chloride-bicarbonate exchanger SLC4A8 mediates an electroneutral Na+ reabsorption process in the renal cortical collecting ducts of mice Regulation of ENaC-Mediated Sodium Reabsorption by Peroxisome Proliferator-Activated Receptors KCNJ10 determines the expression of the apical Na-Cl cotransporter (NCC) in the early distal convoluted tubule (DCT1).Cyp2c44 epoxygenase in the collecting duct is essential for the high K+ intake-induced antihypertensive effect.Regulation of the epithelial sodium channel (ENaC) by membrane trafficking Renal tubular NEDD4-2 deficiency causes NCC-mediated salt-dependent hypertension Severe urinary concentrating defect in renal collecting duct-selective AQP2 conditional-knockout mice.A nonconventional look at ionic fluxes in the skin: lessons from genetically modified mice.Epithelial Sodium Channel-Mediated Sodium Transport Is Not Dependent on the Membrane-Bound Serine Protease CAP2/Tmprss4.Reducing αENaC expression in the kidney connecting tubule induces pseudohypoaldosteronism type 1 symptoms during K+ loading.Sodium and potassium handling by the aldosterone-sensitive distal nephron: the pivotal role of the distal and connecting tubule.ENaC inhibition stimulates HCl secretion in the mouse cortical collecting duct. I. Stilbene-sensitive Cl- secretion.Generation and Characterisation of a Pax8-CreERT2 Transgenic Line and a Slc22a6-CreERT2 Knock-In Line for Inducible and Specific Genetic Manipulation of Renal Tubular Epithelial Cells.Links between dietary salt intake, renal salt handling, blood pressure, and cardiovascular diseases.A new look at electrolyte transport in the distal tubule ENaC inhibition stimulates Cl- secretion in the mouse cortical collecting duct through an NKCC1-dependent mechanism Hypertension, kidney, and transgenics: a fresh perspective.Regulation of transport in the connecting tubule and cortical collecting duct BK channels in the kidney: role in K(+) secretion and localization of molecular components.Preferential assembly of epithelial sodium channel (ENaC) subunits in Xenopus oocytes: role of furin-mediated endogenous proteolysis mTORC2 critically regulates renal potassium handling.Mouse models and the urinary concentrating mechanism in the new millennium.Systems-level analysis of cell-specific AQP2 gene expression in renal collecting duct Thiazolidinedione-induced fluid retention is independent of collecting duct alphaENaC activity.Role of BKbeta1 in Na+ reabsorption by cortical collecting ducts of Na+-deprived mice Transgenic mice and their impact on kidney research.Inactivation of Notch signaling in the renal collecting duct causes nephrogenic diabetes insipidus in mice.

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P2860

Collecting duct-specific gene inactivation of alphaENaC in the mouse kidney does not impair sodium and potassium balance.

http://www.wikidata.org/entity/Q35242835

description

2003 nî lūn-bûn

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2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի օգոստոսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

Collecting duct-specific gene ...... sodium and potassium balance.

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Collecting duct-specific gene ...... sodium and potassium balance.

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type

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Collecting duct-specific gene ...... sodium and potassium balance.

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Collecting duct-specific gene ...... sodium and potassium balance.

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Collecting duct-specific gene ...... sodium and potassium balance.

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Collecting duct-specific gene ...... sodium and potassium balance.

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Journal of Clinical Investigation

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Collecting duct-specific gene ...... r sodium and potassium balance

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P2093

Andrew McMahon

http://www.w3.org/2001/XMLSchema#string

Bernard C Rossier

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Daniel Sauter

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Edith Hummler

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Gustavo Frindt

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Johannes Loffing

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Lawrence G Palmer

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Nicole Fowler-Jaeger

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Tom Carroll

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P2860

Sonic hedgehog regulates proliferation and differentiation of mesenchymal cells in the mouse metanephric kidney

Cell surface expression of the epithelial Na channel and a mutant causing Liddle syndrome: a quantitative approach

Cre reporter strains produced by targeted insertion of EYFP and ECFP into the ROSA26 locus

Epithelial sodium channel and the control of sodium balance: interaction between genetic and environmental factors

SCNN1, an epithelial cell sodium channel gene in the conserved linkage group on mouse chromosome 6 and human chromosome 12

Liddle's syndrome: heritable human hypertension caused by mutations in the beta subunit of the epithelial sodium channel

Amiloride-sensitive epithelial Na+ channel is made of three homologous subunits

Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1

Cell-specific expression of epithelial sodium channel alpha, beta, and gamma subunits in aldosterone-responsive epithelia from the rat: localization by in situ hybridization and immunocytochemistry

Differential activities of the RET tyrosine kinase receptor isoforms during mammalian embryogenesis

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