Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence. - Wikidata - awgldk - TriplyDB

Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

http://www.wikidata.org/entity/Q35256323

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Wnt5a Signaling in Cancer Biological functions of macrophage-derived Wnt5a, and its roles in human diseases Synthetic lethality by targeting EZH2 methyltransferase activity in ARID1A-mutated cancers Wnt5A promotes an adaptive, senescent-like stress response, while continuing to drive invasion in melanoma cells.Wnt5a Signaling in Normal and Cancer Stem Cells WNT5A-ROR2 is induced by inflammatory mediators and is involved in the migration of human ovarian cancer cell line SKOV-3 IL-1β mediates MCP-1 induction by Wnt5a in gastric cancer cells.A high-content screening assay for small-molecule modulators of oncogene-induced senescence.BRAF mutation is associated with a specific cell type with features suggestive of senescence in ovarian serous borderline (atypical proliferative) tumors Ovarian cancer stem cells: are they real and why are they important?Expression profiling of primary and metastatic ovarian tumors reveals differences indicative of aggressive disease.Paracrine WNT5A Signaling Inhibits Expansion of Tumor-Initiating Cells.Using distance covariance for improved variable selection with application to learning genetic risk models.ALDH1A1 is a novel EZH2 target gene in epithelial ovarian cancer identified by genome-wide approaches Wnt7a is a novel inducer of β-catenin-independent tumor-suppressive cellular senescence in lung cancer SUZ12 promotes human epithelial ovarian cancer by suppressing apoptosis via silencing HRK.BCAT1 expression associates with ovarian cancer progression: possible implications in altered disease metabolism.Epigenetic repression of PDZ-LIM domain-containing protein 2 promotes ovarian cancer via NOS2-derived nitric oxide signaling.Role of the stem cell niche in the pathogenesis of epithelial ovarian cancers.Matrix metalloproteinase-10 regulates stemness of ovarian cancer stem-like cells by activation of canonical Wnt signaling and can be a target of chemotherapy-resistant ovarian cancer.Targeting ROR1 inhibits epithelial-mesenchymal transition and metastasis.Multifaceted roles of GSK-3 and Wnt/β-catenin in hematopoiesis and leukemogenesis: opportunities for therapeutic intervention.An analysis of polymorphisms within the Wnt signaling pathway in relation to ovarian cancer risk in a Polish population.Identification of ribonucleotide reductase M2 as a potential target for pro-senescence therapy in epithelial ovarian cancer The Ubiquitin-like with PHD and Ring Finger Domains 1 (UHRF1)/DNA Methyltransferase 1 (DNMT1) Axis Is a Primary Regulator of Cell Senescence.Wnt/β-catenin signaling in bone marrow niche.Response to comments regarding article, "Wnt5a inhibited human trophoblast cell line HTR8/SVneo invasion: implications for early placentation and preeclampsia".Epithelial cell senescence: an adaptive response to pre-carcinogenic stresses?Wnt5a Promotes Cortical Neuron Survival by Inhibiting Cell-Cycle Activation Reduced production and uptake of lactate are essential for the ability of WNT5A signaling to inhibit breast cancer cell migration and invasion.Daam1 activates RhoA to regulate Wnt5a‑induced glioblastoma cell invasion.ARID1A-mutated ovarian cancers depend on HDAC6 activity.CARM1-expressing ovarian cancer depends on the histone methyltransferase EZH2 activity.WNT5A and Its Receptors in the Bone-Cancer Dialogue.Cellular Senescence in the Treatment of Ovarian Cancer.Involvement of Wnt signaling in the injury of murine mesenchymal stem cells exposed to X-radiation.ID1-induced p16/IL6 axis activation contributes to the resistant of hepatocellular carcinoma cells to sorafenib

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Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

http://www.wikidata.org/entity/Q35256323

description

2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

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Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

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type

label

Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

@ast

Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

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prefLabel

Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

@ast

Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

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0008-5472.CAN-11-1341

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Cancer Research

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Wnt5a suppresses epithelial ovarian cancer by promoting cellular senescence.

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P2093

Andrew K Godwin

http://www.w3.org/2001/XMLSchema#string

Benjamin G Bitler

http://www.w3.org/2001/XMLSchema#string

Hong Wu

http://www.w3.org/2001/XMLSchema#string

Hua Li

http://www.w3.org/2001/XMLSchema#string

Jasmine P Nicodemus

http://www.w3.org/2001/XMLSchema#string

Michael J Birrer

http://www.w3.org/2001/XMLSchema#string

Paul Cairns

http://www.w3.org/2001/XMLSchema#string

Qi Cai

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Rugang Zhang

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Tianyu Li

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P2860

PML regulates p53 acetylation and premature senescence induced by oncogenic Ras

Wnt-5a inhibits the canonical Wnt pathway by promoting GSK-3-independent beta-catenin degradation

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Molecular dissection of formation of senescence-associated heterochromatin foci

Dapper, a Dishevelled-associated antagonist of beta-catenin and JNK signaling, is required for notochord formation

The role of PML in tumor suppression

Formation of MacroH2A-containing senescence-associated heterochromatin foci and senescence driven by ASF1a and HIRA

Purified Wnt5a protein activates or inhibits beta-catenin-TCF signaling depending on receptor context

Wnt signalling in stem cells and cancer

WNT and beta-catenin signalling: diseases and therapies

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6184-6194

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10.1158/0008-5472.CAN-11-1341

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19

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