Increased O-linked beta-N-acetylglucosamine levels on proteins improves survival, reduces inflammation and organ damage 24 hours after trauma-hemorrhage in rats - Wikidata - awgldk - TriplyDB

Increased O-linked beta-N-acetylglucosamine levels on proteins improves survival, reduces inflammation and organ damage 24 hours after trauma-hemorrhage in rats

Increased O-linked beta-N-acetylglucosamine levels on proteins improves survival, reduces inflammation and organ damage 24 hours after trauma-hemorrhage in rats

http://www.wikidata.org/entity/Q35282231

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Functional O-GlcNAc modifications: implications in molecular regulation and pathophysiology Cardiovascular Effects of Shock and Trauma in Experimental Models. A Review Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling O-GlcNAc signaling in the cardiovascular system.Inhibition of O-GlcNAcase in perfused rat hearts by NAG-thiazolines at the time of reperfusion is cardioprotective in an O-GlcNAc-dependent manner.Increasing O-GlcNAcylation level on organ culture of soleus modulates the calcium activation parameters of muscle fibers.Heparin use in a rat hemorrhagic shock model induces biologic activity in mesenteric lymph separate from shock Aging influences cardiac mitochondrial gene expression and cardiovascular function following hemorrhage injury.The roles of O-linked β-N-acetylglucosamine in cardiovascular physiology and disease.Protein O-GlcNAcylation: a new signaling paradigm for the cardiovascular system.Dynamic O-GlcNAcylation and its roles in the cellular stress response and homeostasis.Glycosylation of the nuclear pore.Fatty acid synthase inhibits the O-GlcNAcase during oxidative stress.O-GlcNAc protein modification in C2C12 myoblasts exposed to oxidative stress indicates parallels with endogenous antioxidant defense.Protein O-GlcNAcylation is a novel cytoprotective signal in cardiac stem cells.O-linked beta-N-acetylglucosamine (O-GlcNAc) regulates stress-induced heat shock protein expression in a GSK-3beta-dependent manner.Protein O-GlcNAcylation: A critical regulator of the cellular response to stress.Dynamic O-linked N-acetylglucosamine modification of proteins affects stress responses and survival of mesothelial cells exposed to peritoneal dialysis fluids.Antrodia cinnamomea Oligosaccharides Suppress Lipopolysaccharide-Induced Inflammation through Promoting O-GlcNAcylation and Repressing p38/Akt Phosphorylation.The Role of Stress-Induced O-GlcNAc Protein Modification in the Regulation of Membrane Transport.Protein O-GlcNAc Modification Increases in White Blood Cells After a Single Bout of Physical Exercise.

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P2860

Increased O-linked beta-N-acetylglucosamine levels on proteins improves survival, reduces inflammation and organ damage 24 hours after trauma-hemorrhage in rats

http://www.wikidata.org/entity/Q35282231

description

2010 nî lūn-bûn

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2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2010 թվականի փետրվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Increased O-linked beta-N-acet ...... fter trauma-hemorrhage in rats

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Increased O-linked beta-N-acet ...... fter trauma-hemorrhage in rats

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Increased O-linked beta-N-acet ...... fter trauma-hemorrhage in rats

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Increased O-linked beta-N-acet ...... fter trauma-hemorrhage in rats

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Critical Care Medicine

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Increased O-linked beta-N-acet ...... fter trauma-hemorrhage in rats

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Charlye A Brocks

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John C Chatham

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Laszlo G Nöt

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Laszlo Vámhidy

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Richard B Marchase

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P2860

Glucosamine inhibits angiotensin II-induced cytoplasmic Ca2+ elevation in neonatal cardiomyocytes via protein-associated O-linked N-acetylglucosamine

Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAc

Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling

Improved resuscitation minimizes respiratory dysfunction and blunts interleukin-6 and nuclear factor-kappa B activation after traumatic hemorrhage.

Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2

Glucosamine cardioprotection in perfused rat hearts associated with increased O-linked N-acetylglucosamine protein modification and altered p38 activation.

O-Glycosylation of nuclear and cytosolic proteins. Dynamic interplay between O-GlcNAc and O-phosphate.

Altered glycan-dependent signaling induces insulin resistance and hyperleptinemia

Pathophysiology of polytrauma.

Alterations in fuel metabolism in critical illness: hyperglycaemia.

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