Direct binding of myelin basic protein and synthetic copolymer 1 to class II major histocompatibility complex molecules on living antigen-presenting cells--specificity and promiscuity. - Wikidata - awgldk - TriplyDB

Direct binding of myelin basic protein and synthetic copolymer 1 to class II major histocompatibility complex molecules on living antigen-presenting cells--specificity and promiscuity.

Direct binding of myelin basic protein and synthetic copolymer 1 to class II major histocompatibility complex molecules on living antigen-presenting cells--specificity and promiscuity.

http://www.wikidata.org/entity/Q35310089

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Glatiramer acetate reduces the risk for experimental cerebral malaria: a pilot study Molecular analysis of thymopentin binding to HLA-DR molecules Cytokine-Defined B Cell Responses as Therapeutic Targets in Multiple Sclerosis Optimizing therapeutics in the management of patients with multiple sclerosis: a review of drug efficacy, dosing, and mechanisms of action Mechanism of action of glatiramer acetate in multiple sclerosis and its potential for the development of new applications Glatiramer acetate in the treatment of multiple sclerosis: emerging concepts regarding its mechanism of action Macrophage-specific chemokines induced via innate immunity by amino acid copolymers and their role in EAE Glatiramer acetate (Copaxone) induces degenerate, Th2-polarized immune responses in patients with multiple sclerosis.T-cell based immunotherapy in experimental autoimmune encephalomyelitis and multiple sclerosis.Induction of CD4+CD25+ regulatory T cells by copolymer-I through activation of transcription factor Foxp3 The concept of specific immune treatment against autoimmune diseases.Inhibition of experimental autoimmune uveitis by amino acid copolymers Glatiramer acetate triggers PI3Kδ/Akt and MEK/ERK pathways to induce IL-1 receptor antagonist in human monocytes.Glatiramer acetate attenuates pro-inflammatory T cell responses but does not directly protect neurons from inflammatory cell death Memory B cells from a subset of treatment-naïve relapsing-remitting multiple sclerosis patients elicit CD4(+) T-cell proliferation and IFN-γ production in response to myelin basic protein and myelin oligodendrocyte glycoprotein.Glatiramer acetate (Copaxone) therapy induces CD8(+) T cell responses in patients with multiple sclerosis Novel synthetic amino acid copolymers that inhibit autoantigen-specific T cell responses and suppress experimental autoimmune encephalomyelitis.Copolymer 1 acts against the immunodominant epitope 82-100 of myelin basic protein by T cell receptor antagonism in addition to major histocompatibility complex blocking The effect of glatiramer acetate therapy on functional properties of B cells from patients with relapsing-remitting multiple sclerosis.Multiple sclerosis: comparison of copolymer-1- reactive T cell lines from treated and untreated subjects reveals cytokine shift from T helper 1 to T helper 2 cells Seeking balance: Potentiation and inhibition of multiple sclerosis autoimmune responses by IL-6 and IL-10.Therapeutic vaccines in autoimmunity.Immunomodulation of experimental autoimmune encephalomyelitis by oral administration of copolymer 1.Synthetic amino acid copolymers that bind to HLA-DR proteins and inhibit type II collagen-reactive T cell clones.Copolymer 1 induces T cells of the T helper type 2 that crossreact with myelin basic protein and suppress experimental autoimmune encephalomyelitis The role of dendritic cells in the generation of CD4(+) CD25(HI) Foxp3(+) T cells induced by amino acid copolymers Mechanism of action of glatiramer acetate in treatment of multiple sclerosis.Design of peptide immunotherapies for MHC Class-II-associated autoimmune disorders.Glatiramer acetate treatment effects on gene expression in monocytes of multiple sclerosis patients.Amelioration of proteolipid protein 139-151-induced encephalomyelitis in SJL mice by modified amino acid copolymers and their mechanisms Modified amino acid copolymers suppress myelin basic protein 85-99-induced encephalomyelitis in humanized mice through different effects on T cells.A synthetic random basic copolymer with promiscuous binding to class II major histocompatibility complex molecules inhibits T-cell proliferative responses to major and minor histocompatibility antigens in vitro and confers the capacity to prevent mu Glatiramer acetate and the glatiramoid class of immunomodulator drugs in multiple sclerosis: an update.Immunomodulatory drug treatment in multiple sclerosis.Antibody-independent B cell effector functions in relapsing remitting multiple sclerosis: clues to increased inflammatory and reduced regulatory B cell capacity.Glatiramer acetate for treatment of relapsing-remitting multiple sclerosis.New findings and old controversies in the research of multiple sclerosis and its model experimental autoimmune encephalomyelitis.The pharmacokinetics of glatiramer acetate for multiple sclerosis treatment.A naturally processed HLA-DR-bound peptide from the IL-9 receptor alpha of HTLV-1-transformed T cells serves as a T helper epitope.Clinical utility of glatiramer acetate in the management of relapse frequency in multiple sclerosis.

P2860

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P2860

Direct binding of myelin basic protein and synthetic copolymer 1 to class II major histocompatibility complex molecules on living antigen-presenting cells--specificity and promiscuity.

http://www.wikidata.org/entity/Q35310089

description

1994 nî lūn-bûn

@nan

1994 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

1994 թվականի մայիսին հրատարակված գիտական հոդված

@hy

1994年の論文

@ja

1994年論文

@yue

1994年論文

@zh-hant

1994年論文

@zh-hk

1994年論文

@zh-mo

1994年論文

@zh-tw

1994年论文

@wuu

name

Direct binding of myelin basic ...... --specificity and promiscuity.

@ast

Direct binding of myelin basic ...... --specificity and promiscuity.

@en

type

label

Direct binding of myelin basic ...... --specificity and promiscuity.

@ast

Direct binding of myelin basic ...... --specificity and promiscuity.

@en

prefLabel

Direct binding of myelin basic ...... --specificity and promiscuity.

@ast

Direct binding of myelin basic ...... --specificity and promiscuity.

@en

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P2860

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PNAS.91.11.4872

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Direct binding of myelin basic ...... s--specificity and promiscuity

@en

P2093

C Brautbar

http://www.w3.org/2001/XMLSchema#string

D Teitelbaum

http://www.w3.org/2001/XMLSchema#string

E Gurevich

http://www.w3.org/2001/XMLSchema#string

M Fridkis-Hareli

http://www.w3.org/2001/XMLSchema#string

M Sela

http://www.w3.org/2001/XMLSchema#string

O J Kwon

http://www.w3.org/2001/XMLSchema#string

R Arnon

http://www.w3.org/2001/XMLSchema#string

T Brenner

http://www.w3.org/2001/XMLSchema#string

P2860

Structure of the human class I histocompatibility antigen, HLA-A2

Identification of a motif for HLA-DR1 binding peptides using M13 display libraries

Specific inhibition of the T-cell response to myelin basic protein by the synthetic copolymer Cop 1

Binding of myelin basic protein peptides to human histocompatibility leukocyte antigen class II molecules and their recognition by T cells from multiple sclerosis patients

Three-dimensional structure of the human class II histocompatibility antigen HLA-DR1.

Ia-antigen-T-cell interactions for a thymus-independent antigen composed of D amino acids

Specificity and promiscuity among naturally processed peptides bound to HLA-DR alleles.

T-cell recognition of myelin basic protein.

Synthetic copolymer 1 inhibits human T-cell lines specific for myelin basic protein.

Interactions between immunogenic peptides and MHC proteins.

P304

4872-4876

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P31

scholarly article

P356

10.1073/PNAS.91.11.4872

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P407

P433

11

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P478

91

http://www.w3.org/2001/XMLSchema#string

P50

P577

1994-05-01T00:00:00Z

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P5875

15684288

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P698

7515181

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P932

43891

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