Thioredoxin reductase-2 is essential for keeping low levels of H(2)O(2) emission from isolated heart mitochondria. - Wikidata - awgldk - TriplyDB

Thioredoxin reductase-2 is essential for keeping low levels of H(2)O(2) emission from isolated heart mitochondria.

Thioredoxin reductase-2 is essential for keeping low levels of H(2)O(2) emission from isolated heart mitochondria.

http://www.wikidata.org/entity/Q35312726

about

Cardiac stem cells: biology and clinical applications Abnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balance Regulation of cell survival and death by pyridine nucleotides Mitochondrial health, the epigenome and healthspan Protective mechanisms of mitochondria and heart function in diabetes Mitochondrial dynamics in diabetic cardiomyopathy Stromal response to prostate cancer: nanotechnology-based detection of thioredoxin-interacting protein partners distinguishes prostate cancer associated stroma from that of benign prostatic hyperplasia The characterization of the Caenorhabditis elegans mitochondrial thioredoxin system uncovers an unexpected protective role of thioredoxin reductase 2 in β-amyloid peptide toxicity.The Emerging Role of Thioredoxin-Interacting Protein in Myocardial Ischemia/Reperfusion Injury Acetaminophen reactive intermediates target hepatic thioredoxin reductase Mitochondrial respiration and ROS emission during β-oxidation in the heart: An experimental-computational study.Complex oscillatory redox dynamics with signaling potential at the edge between normal and pathological mitochondrial function.Glutaredoxin 2 reduces both thioredoxin 2 and thioredoxin 1 and protects cells from apoptosis induced by auranofin and 4-hydroxynonenal.A deficiency of apoptosis inducing factor (AIF) in Harlequin mouse heart mitochondria paradoxically reduces ROS generation during ischemia-reperfusion Mitochondrial and cellular mechanisms for managing lipid excess.Reactive oxygen species and redox compartmentalization Hypertrophic cardiomyopathy: a heart in need of an energy bar?Regenerative medicine for the heart: perspectives on stem-cell therapy.Thioredoxin reductase deficiency potentiates oxidative stress, mitochondrial dysfunction and cell death in dopaminergic cells Restoring redox balance enhances contractility in heart trabeculae from type 2 diabetic rats exposed to high glucose.Can endurance exercise preconditioning prevention disuse muscle atrophy?Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.Knockout of mitochondrial thioredoxin reductase stabilizes prolyl hydroxylase 2 and inhibits tumor growth and tumor-derived angiogenesis.Distinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate.Increased reactive oxygen species production during reductive stress: The roles of mitochondrial glutathione and thioredoxin reductases.Differentiating between apparent and actual rates of H2O2 metabolism by isolated rat muscle mitochondria to test a simple model of mitochondria as regulators of H2O2 concentration.Metabolic alterations induce oxidative stress in diabetic and failing hearts: different pathways, same outcome.Down-regulation of flavin reductase and alcohol dehydrogenase-1 (ADH1) in metronidazole-resistant isolates of Trichomonas vaginalis Dietary inorganic nitrate alleviates doxorubicin cardiotoxicity: mechanisms and implications.Glutathione/thioredoxin systems modulate mitochondrial H2O2 emission: an experimental-computational study.miR-17-3p Exacerbates Oxidative Damage in Human Retinal Pigment Epithelial Cells.Redox mechanisms of cardiomyocyte mitochondrial protection.ErbB2 overexpression upregulates antioxidant enzymes, reduces basal levels of reactive oxygen species, and protects against doxorubicin cardiotoxicity.GSH or palmitate preserves mitochondrial energetic/redox balance, preventing mechanical dysfunction in metabolically challenged myocytes/hearts from type 2 diabetic mice Integrating mitochondrial energetics, redox and ROS metabolic networks: a two-compartment model Beetroot juice reduces infarct size and improves cardiac function following ischemia-reperfusion injury: Possible involvement of endogenous H2S Angiotensin II-induced production of mitochondrial reactive oxygen species: potential mechanisms and relevance for cardiovascular disease.Mitochondrial reactive oxygen species: which ROS signals cardioprotection?Oxidative stress and neurodegenerative disorders An integrated mitochondrial ROS production and scavenging model: implications for heart failure.

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P2860

Thioredoxin reductase-2 is essential for keeping low levels of H(2)O(2) emission from isolated heart mitochondria.

http://www.wikidata.org/entity/Q35312726

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Thioredoxin reductase-2 is ess ...... m isolated heart mitochondria.

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Thioredoxin reductase-2 is ess ...... m isolated heart mitochondria.

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type

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Thioredoxin reductase-2 is ess ...... m isolated heart mitochondria.

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Thioredoxin reductase-2 is ess ...... m isolated heart mitochondria.

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prefLabel

Thioredoxin reductase-2 is ess ...... m isolated heart mitochondria.

@ast

Thioredoxin reductase-2 is ess ...... m isolated heart mitochondria.

@en

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JBC.M111.284612

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Journal of Biological Chemistry

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Thioredoxin reductase-2 is ess ...... m isolated heart mitochondria.

@en

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Brian A Stanley

http://www.w3.org/2001/XMLSchema#string

David Lloyd

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Iain McDonald

http://www.w3.org/2001/XMLSchema#string

Miguel A Aon

http://www.w3.org/2001/XMLSchema#string

Nazareno Paolocci

http://www.w3.org/2001/XMLSchema#string

Sa Shi

http://www.w3.org/2001/XMLSchema#string

Vidhya Sivakumaran

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Walter H Watson

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P2860

Overexpressed human mitochondrial thioredoxin confers resistance to oxidant-induced apoptosis in human osteosarcoma cells

Essential role for mitochondrial thioredoxin reductase in hematopoiesis, heart development, and heart function

How mitochondria produce reactive oxygen species

Inhibition of Schistosoma mansoni thioredoxin-glutathione reductase by auranofin: structural and kinetic aspects

Peroxiredoxin, a novel family of peroxidases

Effect of auranofin on the mitochondrial generation of hydrogen peroxide. Role of thioredoxin reductase

Mitochondrial respiratory chain and thioredoxin reductase regulate intermembrane Cu,Zn-superoxide dismutase activity: implications for mitochondrial energy metabolism and apoptosis

Molecular cloning and characterization of a mitochondrial selenocysteine-containing thioredoxin reductase from rat liver

Catalase takes part in rat liver mitochondria oxidative stress defense

High protonic potential actuates a mechanism of production of reactive oxygen species in mitochondria

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33669-33677

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38

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286

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2011-08-05T00:00:00Z

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21832082

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3190932

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