Malignant Drosophila tumors interrupt insulin signaling to induce cachexia-like wasting.
about
Using Drosophila to discover mechanisms underlying type 2 diabetesThe interplay between intestinal bacteria and host metabolism in health and disease: lessons from Drosophila melanogasterUnderstanding cachexia as a cancer metabolism syndromeMechanisms of metabolic dysfunction in cancer-associated cachexiaControl of Germline Stem Cell Lineages by Diet and Physiology.Systemic organ wasting induced by localized expression of the secreted insulin/IGF antagonist ImpL2.The Regulation of Muscle Structure and Metabolism by Mio/dChREBP in Drosophila.Nutrition-dependent control of insect development by insulin-like peptides.Accumulation of differentiating intestinal stem cell progenies drives tumorigenesisPancreatic cancer cell-derived IGFBP-3 contributes to muscle wastingSTAT3 in the systemic inflammation of cancer cachexia.An in vivo genetic screen in Drosophila identifies the orthologue of human cancer/testis gene SPO11 among a network of targets to inhibit lethal(3)malignant brain tumour growthThe Systemic Control of Growth.Insulin/IGF signaling in Drosophila and other insects: factors that regulate production, release and post-release action of the insulin-like peptides.PP6 Disruption Synergizes with Oncogenic Ras to Promote JNK-Dependent Tumor Growth and Invasion.Interorgan Communication Pathways in Physiology: Focus on Drosophila.Microenvironmental autophagy promotes tumour growth.The transcriptional response to tumorigenic polarity loss in Drosophila.A Drosophila model of insulin resistance associated with the human TRIB3 Q/R polymorphism.Feedback amplification loop drives malignant growth in epithelial tissues.The Drosophila Imaginal Disc Tumor Model: Visualization and Quantification of Gene Expression and Tumor Invasiveness Using Genetic Mosaics.Breaking Down Neighbors to Fuel Tumorigenesis.Modelling Cooperative Tumorigenesis in Drosophila.Drosophila as a Model to Study the Link between Metabolism and Cancer.A novel zebrafish intestinal tumor model reveals a role for cyp7a1-dependent tumor-liver crosstalk in tumor's adverse effects on host.Cancer metabolism: A waste of insulin interference.Studying tumor growth in Drosophila using the tissue allograft method.Functions of autophagy in the tumor microenvironment and cancer metastasis.A hormonal cue promotes timely follicle cell migration by modulating transcription profiles.Drosophila Larval Brain Neoplasms Present Tumour-Type Dependent Genome Instability.Drosophila as a Model System to Study Nonautonomous Mechanisms Affecting Tumour Growth and Cell Death.
P2860
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P2860
Malignant Drosophila tumors interrupt insulin signaling to induce cachexia-like wasting.
description
2015 nî lūn-bûn
@nan
2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Malignant Drosophila tumors interrupt insulin signaling to induce cachexia-like wasting.
@ast
Malignant Drosophila tumors interrupt insulin signaling to induce cachexia-like wasting.
@en
type
label
Malignant Drosophila tumors interrupt insulin signaling to induce cachexia-like wasting.
@ast
Malignant Drosophila tumors interrupt insulin signaling to induce cachexia-like wasting.
@en
prefLabel
Malignant Drosophila tumors interrupt insulin signaling to induce cachexia-like wasting.
@ast
Malignant Drosophila tumors interrupt insulin signaling to induce cachexia-like wasting.
@en
P2860
P1433
P1476
Malignant Drosophila tumors interrupt insulin signaling to induce cachexia-like wasting.
@en
P2093
Alejandra Figueroa-Clarevega
David Bilder
P2860
P356
10.1016/J.DEVCEL.2015.03.001
P407
P577
2015-04-01T00:00:00Z