AD synapses contain abundant Aβ monomer and multiple soluble oligomers, including a 56-kDa assembly
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Tau Oligomers: The Toxic Player at Synapses in Alzheimer's DiseaseCerebrospinal Fluid Particles in Alzheimer Disease and Parkinson DiseaseIVIg protects the 3xTg-AD mouse model of Alzheimer’s disease from memory deficit and Aβ pathologySynaptic protein α1-takusan mitigates amyloid-β-induced synaptic loss via interaction with tau and postsynaptic density-95 at postsynaptic sitesEnvironmental enrichment strengthens corticocortical interactions and reduces amyloid-β oligomers in aged mice.Memantine prevents memory consolidation failure induced by soluble beta amyloid in rats.Oligomeric Aβ-induced synaptic dysfunction in Alzheimer's disease.Pre-synaptic C-terminal truncated tau is released from cortical synapses in Alzheimer's diseaseIsolation of synaptic terminals from Alzheimer's disease cortexPreferential accumulation of amyloid-beta in presynaptic glutamatergic terminals (VGluT1 and VGluT2) in Alzheimer's disease cortex.Apolipoprotein E level and cholesterol are associated with reduced synaptic amyloid beta in Alzheimer's disease and apoE TR mouse cortexExtensive p-tau pathology and SDS-stable p-tau oligomers in Alzheimer's cortical synapses.Synaptic Amyloid-β Oligomers Precede p-Tau and Differentiate High Pathology Control Cases.Levels of soluble apolipoprotein E/amyloid-β (Aβ) complex are reduced and oligomeric Aβ increased with APOE4 and Alzheimer disease in a transgenic mouse model and human samplesNew ELISAs with high specificity for soluble oligomers of amyloid β-protein detect natural Aβ oligomers in human brain but not CSF.Loss of endophilin-B1 exacerbates Alzheimer's disease pathologyA novel recombinant 6Aβ15-THc-C chimeric vaccine (rCV02) mitigates Alzheimer's disease-like pathology, cognitive decline and synaptic loss in aged 3 × Tg-AD mice.Alzheimer's disease, β-amyloid, glutamate, NMDA receptors and memantine--searching for the connectionsSoluble apoE/Aβ complex: mechanism and therapeutic target for APOE4-induced AD risk.Oligomeropathies and pathogenesis of Alzheimer and Parkinson's diseases.Abnormal accumulation of autophagic vesicles correlates with axonal and synaptic pathology in young Alzheimer's mice hippocampus.Inflammation Combined with Ischemia Produces Myelin Injury and Plaque-Like Aggregates of Myelin, Amyloid-β and AβPP in Adult Rat Brain.How many amyloid-β peptides can a neuron bind before it dies?Membrane-bound tetramer and trimer Aβ oligomeric species correlate with toxicity towards cultured neurons.Human Striatal Dopaminergic and Regional Serotonergic Synaptic Degeneration with Lewy Body Disease and Inheritance of APOE ε4.
P2860
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P2860
AD synapses contain abundant Aβ monomer and multiple soluble oligomers, including a 56-kDa assembly
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
AD synapses contain abundant A ...... s, including a 56-kDa assembly
@ast
AD synapses contain abundant A ...... s, including a 56-kDa assembly
@en
type
label
AD synapses contain abundant A ...... s, including a 56-kDa assembly
@ast
AD synapses contain abundant A ...... s, including a 56-kDa assembly
@en
prefLabel
AD synapses contain abundant A ...... s, including a 56-kDa assembly
@ast
AD synapses contain abundant A ...... s, including a 56-kDa assembly
@en
P2093
P2860
P50
P1476
AD synapses contain abundant A ...... s, including a 56-kDa assembly
@en
P2093
Carol A Miller
Gregory M Cole
Karen Hoppens Gylys
Kristen M Henkins
Sophie Sokolow
P2860
P304
P356
10.1016/J.NEUROBIOLAGING.2011.05.011
P577
2011-07-07T00:00:00Z