Intra-renal slow cell-cycle cells contribute to the restoration of kidney tubules injured by ischemia/reperfusion. - Wikidata - awgldk - TriplyDB

Intra-renal slow cell-cycle cells contribute to the restoration of kidney tubules injured by ischemia/reperfusion.

Intra-renal slow cell-cycle cells contribute to the restoration of kidney tubules injured by ischemia/reperfusion.

http://www.wikidata.org/entity/Q35374741

about

Distinct populations of label-retaining cells in the adult kidney are defined temporally and exhibit divergent regional distributions.Cisplatin induces primary necrosis through poly(ADP-ribose) polymerase 1 activation in kidney proximal tubular cells Renal telocytes contribute to the repair of ischemically injured renal tubules Donor ABCB1 variant associates with increased risk for kidney allograft failure.The role of long-term label-retaining cells in the regeneration of adult mouse kidney after ischemia/reperfusion injury.Stem cells and their role in renal ischaemia reperfusion injury.Different modes of renal proximal tubule regeneration in health and disease.Reduction of oxidative stress during recovery accelerates normalization of primary cilia length that is altered after ischemic injury in murine kidneys.Hydrogen sulfide accelerates the recovery of kidney tubules after renal ischemia/reperfusion injury.Recruitment and subsequent proliferation of bone marrow-derived cells in the postischemic kidney are important to the progression of fibrosis.

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Intra-renal slow cell-cycle cells contribute to the restoration of kidney tubules injured by ischemia/reperfusion.

http://www.wikidata.org/entity/Q35374741

description

2011 nî lūn-bûn

@nan

2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Intra-renal slow cell-cycle ce ...... jured by ischemia/reperfusion.

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Intra-renal slow cell-cycle ce ...... jured by ischemia/reperfusion.

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type

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Intra-renal slow cell-cycle ce ...... jured by ischemia/reperfusion.

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Intra-renal slow cell-cycle ce ...... jured by ischemia/reperfusion.

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Intra-renal slow cell-cycle ce ...... jured by ischemia/reperfusion.

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P356

ACB.2011.44.3.186

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Anatomy & cell biology

P1476

Intra-renal slow cell-cycle ce ...... jured by ischemia/reperfusion.

@en

P2093

Jee In Kim

http://www.w3.org/2001/XMLSchema#string

Jinu Kim

http://www.w3.org/2001/XMLSchema#string

Kwon Moo Park

http://www.w3.org/2001/XMLSchema#string

Yeon Kyung Na

http://www.w3.org/2001/XMLSchema#string

P2860

The renal papilla is a niche for adult kidney stem cells

Intrarenal cells, not bone marrow-derived cells, are the major source for regeneration in postischemic kidney.

Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells.

Localization of proliferating cell nuclear antigen, vimentin, c-Fos, and clusterin in the postischemic kidney. Evidence for a heterogenous genetic response among nephron segments, and a large pool of mitotically active and dedifferentiated cells.

Stem cell approaches for the treatment of renal failure.

Intrinsic epithelial cells repair the kidney after injury.

Bone marrow stem cells contribute to repair of the ischemically injured renal tubule

Dedifferentiation and proliferation of surviving epithelial cells in acute renal failure.

Epidermal stem cells: properties, markers, and location.

Adult stem cell-like tubular cells reside in the corticomedullary junction of the kidney.

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186-193

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scholarly article

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10.5115/ACB.2011.44.3.186

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3

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44

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22025970

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3195822

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