A secreted Salmonella protein induces a proinflammatory response in epithelial cells, which promotes neutrophil migration.
about
Salmonella effector proteins and host-cell responsesRegulation of bacterial virulence by Csr (Rsm) systemsThe Dynamic Interactions between Salmonella and the Microbiota, within the Challenging Niche of the Gastrointestinal TractPERP, a host tetraspanning membrane protein, is required for Salmonella-induced inflammation.Examination of Salmonella gene expression in an infected mammalian host using the green fluorescent protein and two-colour flow cytometry.Three-dimensional tissue assemblies: novel models for the study of Salmonella enterica serovar Typhimurium pathogenesis.Mass spectrometry-based quantitative proteomic analysis of Salmonella enterica serovar Enteritidis protein expression upon exposure to hydrogen peroxideThe SdiA-regulated gene srgE encodes a type III secreted effector.In vitro coculture assay to assess pathogen induced neutrophil trans-epithelial migration.Murein lipoprotein is a critical outer membrane component involved in Salmonella enterica serovar typhimurium systemic infectionSalmonella pathogenesis and processing of secreted effectors by caspase-3Enteroaggregative Escherichia coli promotes transepithelial migration of neutrophils through a conserved 12-lipoxygenase pathway.A Salmonella nanoparticle mimic overcomes multidrug resistance in tumours.Shiga toxin translocation across intestinal epithelial cells is enhanced by neutrophil transmigration.The chemokine CCL2 is required for control of murine gastric Salmonella enterica infectionThe oxido-reductase enzyme glutathione peroxidase 4 (GPX4) governs Salmonella Typhimurium-induced neutrophil transepithelial migrationBreaching the mucosal barrier by stealth: an emerging pathogenic mechanism for enteroadherent bacterial pathogens.Role of cross talk in regulating the dynamic expression of the flagellar Salmonella pathogenicity island 1 and type 1 fimbrial genesCharacterization of Salmonella type III secretion hyper-activity which results in biofilm-like cell aggregation.Pathways leading from BarA/SirA to motility and virulence gene expression in Salmonella.Microbes and microbial toxins: paradigms for microbial-mucosal interactions. VII. Enteropathogenic Escherichia coli: physiological alterations from an extracellular position.Involvement of SipA in modulating actin dynamics during Salmonella invasion into cultured epithelial cells.RtsA coordinately regulates DsbA and the Salmonella pathogenicity island 1 type III secretion system.Bacterial secreted effectors and caspase-3 interactionsHilE interacts with HilD and negatively regulates hilA transcription and expression of the Salmonella enterica serovar Typhimurium invasive phenotype.Pretreatment of mice with streptomycin provides a Salmonella enterica serovar Typhimurium colitis model that allows analysis of both pathogen and hostCXCR2 ligands and G-CSF mediate PKCalpha-induced intraepidermal inflammation.Molecular pathogenesis of Salmonella enterica serotype typhimurium-induced diarrhea.Pathogenesis of Salmonella-induced enteritis.Intestinal epithelial responses to enteric pathogens: effects on the tight junction barrier, ion transport, and inflammation.Functions and effectors of the Salmonella pathogenicity island 2 type III secretion system.Role of the Salmonella pathogenicity island 1 effector proteins SipA, SopB, SopE, and SopE2 in Salmonella enterica subspecies 1 serovar Typhimurium colitis in streptomycin-pretreated mice.The ERM protein, ezrin, regulates neutrophil transmigration by modulating the apical localization of MRP2 in response to the SipA effector protein during Salmonella Typhimurium infection.A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.Leukocyte-epithelial interactions and mucosal homeostasis.Salmonella effectors: important players modulating host cell function during infection.Induction of interleukin-8 in T84 cells by Vibrio cholerae.Salmonella infections: immune and non-immune protection with vaccines.Distinct isoforms of phospholipase A2 mediate the ability of Salmonella enterica serotype typhimurium and Shigella flexneri to induce the transepithelial migration of neutrophils.The actin-polymerizing activity of SipA is not essential for Salmonella enterica serovar Typhimurium-induced mucosal inflammation
P2860
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P2860
A secreted Salmonella protein induces a proinflammatory response in epithelial cells, which promotes neutrophil migration.
description
2000 nî lūn-bûn
@nan
2000 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
A secreted Salmonella protein ...... promotes neutrophil migration.
@ast
A secreted Salmonella protein ...... promotes neutrophil migration.
@en
type
label
A secreted Salmonella protein ...... promotes neutrophil migration.
@ast
A secreted Salmonella protein ...... promotes neutrophil migration.
@en
prefLabel
A secreted Salmonella protein ...... promotes neutrophil migration.
@ast
A secreted Salmonella protein ...... promotes neutrophil migration.
@en
P2093
P2860
P356
P1476
A secreted Salmonella protein ...... promotes neutrophil migration.
@en
P2093
P2860
P304
12283-12288
P356
10.1073/PNAS.97.22.12283
P407
P577
2000-10-01T00:00:00Z