Hyperphosphorylation of RyRs underlies triggered activity in transgenic rabbit model of LQT2 syndrome.
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Calcium transients closely reflect prolonged action potentials in iPSC models of inherited cardiac arrhythmia.Beyond the Electrocardiogram: Mutations in Cardiac Ion Channel Genes Underlie Nonarrhythmic Phenotypes.Role of sarcoplasmic reticulum calcium in development of secondary calcium rise and early afterdepolarizations in long QT syndrome rabbit model.Calcium-voltage coupling in the genesis of early and delayed afterdepolarizations in cardiac myocytesRegulation of sarcoplasmic reticulum Ca2+ release by serine-threonine phosphatases in the heart.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Potassium currents in the heart: functional roles in repolarization, arrhythmia and therapeutics.Improved Prediction of Drug-Induced Torsades de Pointes Through Simulations of Dynamics and Machine Learning Algorithms.Spontaneous initiation of premature ventricular complexes and arrhythmias in type 2 long QT syndrome.Global Optimization of Ventricular Myocyte Model to Multi-Variable Objective Improves Predictions of Drug-Induced Torsades de Pointes.A model of cardiac ryanodine receptor gating predicts experimental Ca2+-dynamics and Ca2+-triggered arrhythmia in the long QT syndrome.Mechanisms linking T-wave alternans to spontaneous initiation of ventricular arrhythmias in rabbit models of long QT syndrome.Identification of a targeted and testable antiarrhythmic therapy for long-QT syndrome type 2 using a patient-specific cellular model.The role of RyR2 oxidation in the blunted frequency-dependent facilitation of Ca2+ transient amplitude in rabbit failing myocytes.Deficiency of NOD1 Improves the β-Adrenergic Modulation of Ca2+ Handling in a Mouse Model of Heart Failure.Cardiac Arrhythmias as Manifestations of Nanopathies: An Emerging ViewA Universal Scaling Relation for Defining Power Spectral Bands in Mammalian Heart Rate Variability AnalysisCalcium-Mediated Oscillation in Membrane Potentials and Atrial-Triggered Activity in Atrial Cells of Casq2 Mutation Mice
P2860
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P2860
Hyperphosphorylation of RyRs underlies triggered activity in transgenic rabbit model of LQT2 syndrome.
description
2014 nî lūn-bûn
@nan
2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Hyperphosphorylation of RyRs u ...... rabbit model of LQT2 syndrome.
@ast
Hyperphosphorylation of RyRs u ...... rabbit model of LQT2 syndrome.
@en
type
label
Hyperphosphorylation of RyRs u ...... rabbit model of LQT2 syndrome.
@ast
Hyperphosphorylation of RyRs u ...... rabbit model of LQT2 syndrome.
@en
prefLabel
Hyperphosphorylation of RyRs u ...... rabbit model of LQT2 syndrome.
@ast
Hyperphosphorylation of RyRs u ...... rabbit model of LQT2 syndrome.
@en
P2093
P2860
P1433
P1476
Hyperphosphorylation of RyRs u ...... rabbit model of LQT2 syndrome.
@en
P2093
Alain Karma
Bum-Rak Choi
Colin M Rees
Dmitry Terentyev
Gideon Koren
Hitesh K Jindal
Jean Daley
Kamana Bist
Katja E Odening
Leroy L Cooper
P2860
P304
P356
10.1161/CIRCRESAHA.115.305146
P577
2014-09-23T00:00:00Z