DNA damage response factors from diverse pathways, including DNA crosslink repair, mediate alternative end joining.
about
Update of the human and mouse Fanconi anemia genesDNA polymerase θ (POLQ), double-strand break repair, and cancerUse of poly ADP-ribose polymerase [PARP] inhibitors in cancer cells bearing DDR defects: the rationale for their inclusion in the clinicContribution of classical end-joining to PTEN inactivation in p53-mediated glioblastoma formation and drug-resistant survival.WRN regulates pathway choice between classical and alternative non-homologous end joiningMelatonin, a Full Service Anti-Cancer Agent: Inhibition of Initiation, Progression and Metastasis.Microhomology-mediated end joining induces hypermutagenesis at breakpoint junctions.Efficient Recreation of t(11;22) EWSR1-FLI1+ in Human Stem Cells Using CRISPR/Cas9.Cellular Repair of DNA-DNA Cross-Links Induced by 1,2,3,4-Diepoxybutane.Drosophila DNA polymerase theta utilizes both helicase-like and polymerase domains during microhomology-mediated end joining and interstrand crosslink repairNbs1 ChIP-Seq Identifies Off-Target DNA Double-Strand Breaks Induced by AID in Activated Splenic B Cells.CRL4(Wdr70) regulates H2B monoubiquitination and facilitates Exo1-dependent resection.A role for human homologous recombination factors in suppressing microhomology-mediated end joiningA Selective Small Molecule DNA2 Inhibitor for Sensitization of Human Cancer Cells to ChemotherapyMechanistic Modelling and Bayesian Inference Elucidates the Variable Dynamics of Double-Strand Break Repair.Apoptosis Induction and Gene Expression Profile Alterations of Cutaneous T-Cell Lymphoma Cells following Their Exposure to Bortezomib and MethotrexateRepair Pathway Choices and Consequences at the Double-Strand BreakRisky business: Microhomology-mediated end joining.Tetratricopeptide repeat factor XAB2 mediates the end resection step of homologous recombinationFANCD2 Maintains Fork Stability in BRCA1/2-Deficient Tumors and Promotes Alternative End-Joining DNA Repair.Contribution of canonical nonhomologous end joining to chromosomal rearrangements is enhanced by ATM kinase deficiency.Maintenance of genome stability by Fanconi anemia proteins.hPso4/hPrp19: a critical component of DNA repair and DNA damage checkpoint complexes.Control of alternative end joining by the chromatin remodeler p400 ATPase.BERing the burden of damage: Pathway crosstalk and posttranslational modification of base excision repair proteins regulate DNA damage management.Tracking metastatic breast cancer: the future of biology in biosensors.Regulation of Single-Strand Annealing and its Role in Genome Maintenance.Melatonin: A pleiotropic molecule that modulates DNA damage response and repair pathways.Mechanisms of DNA damage, repair, and mutagenesis.Inhibition of DNA2 nuclease as a therapeutic strategy targeting replication stress in cancer cells.Genomic portfolio of Merkel cell carcinoma as determined by comprehensive genomic profiling: implications for targeted therapeutics.FANCJ helicase controls the balance between short- and long-tract gene conversions between sister chromatids.Gene expression and mutation-guided synthetic lethality eradicates proliferating and quiescent leukemia cells.Differences in the recruitment of DNA repair proteins at subtelomeric and interstitial I-SceI endonuclease-induced DNA double-strand breaks.PRPF8 is important for BRCA1-mediated homologous recombination.Main steps in DNA double-strand break repair: an introduction to homologous recombination and related processes.Super-resolution imaging identifies PARP1 and the Ku complex acting as DNA double-strand break sensors.How cells ensure correct repair of DNA double-strand breaks.Overexpression of the base excision repair NTHL1 glycosylase causes genomic instability and early cellular hallmarks of cancer.Repeat-mediated deletions can be induced by a chromosomal break far from a repeat, but multiple pathways suppress such rearrangements
P2860
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P2860
DNA damage response factors from diverse pathways, including DNA crosslink repair, mediate alternative end joining.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
DNA damage response factors fr ...... diate alternative end joining.
@ast
DNA damage response factors fr ...... diate alternative end joining.
@en
type
label
DNA damage response factors fr ...... diate alternative end joining.
@ast
DNA damage response factors fr ...... diate alternative end joining.
@en
prefLabel
DNA damage response factors fr ...... diate alternative end joining.
@ast
DNA damage response factors fr ...... diate alternative end joining.
@en
P2093
P2860
P1433
P1476
DNA damage response factors fr ...... diate alternative end joining.
@en
P2093
Diana A Yanez
Jeremy M Stark
Sean M Howard
P2860
P304
P356
10.1371/JOURNAL.PGEN.1004943
P577
2015-01-28T00:00:00Z